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Stem cell reviews and reports2025; 21(8); 2654-2666; doi: 10.1007/s12015-025-10959-9

Mitohormesis and Regeneration: Natural Compounds Chlorogenic Acid (CGA) and Isovanillic Acid 3-O-sulfate (IVAS) Boost Muscle Cell Recovery in the Equine Athlete Model.

Abstract: Skeletal muscle satellite cells ( SCs), essential for muscle regeneration, are a valuable model for studying exercise-induced stress relevant to human athletes. This study examined the effects of two natural compounds-chlorogenic acid (CGA) and isovanillic acid 3-O-sulfate (IVAS)-increasingly recognized as components of modern, nature-based recovery strategies. Their combination (Hybrid) was also tested on equine model of skeletal muscle satellite cells (ESCs) exposed to heat shock (40 °C, 1 h), mimicking exercise stress. Cells were treated with CGA (0.005%), IVAS (0.0005%), or both for 24 h post-stress. Cell viability (MTS), mitochondrial membrane potential, apoptosis (Annexin V/7-AAD), nitric oxide (NO) production, and gene expression (RT-qPCR) were assessed. CGA significantly improved viability under both normothermia and heat stress (216-227%, p < 0.05), while IVAS was effective only without stress. Only the Hybrid group maintained elongated morphology post-heat shock. CGA increased NO levels (p < 0.05), with no effect from IVAS or Hybrid. Antioxidant gene expression remained unchanged, but proinflammatory cytokines IL-6 and IL-1β were upregulated in the Hybrid group (2.74- and 5.64-fold, p < 0.01), suggesting a controlled, adaptive immune response. Early apoptosis rose in CGA and Hybrid groups (~ 34%, p < 0.05), but total cell death was lowest in the Hybrid group (6.26%). BCL2 was downregulated (p < 0.05), while BAX increased only in the Hybrid group (8.14-fold, p < 0.01). Mitochondrial genes MFN2, TFAM, and PUSL1 were significantly upregulated in the Hybrid group; MIRO1 expression increased in all treated groups. CGA and IVAS synergistically promote mitochondrial stability and ESC survival via mitochondrial activation and inflammation regulation-supporting the growing trend of using natural compounds in muscle recovery strategies.
Publication Date: 2025-09-03 PubMed ID: 40900286PubMed Central: PMC12504121DOI: 10.1007/s12015-025-10959-9Google Scholar: Lookup
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  • Journal Article

Summary

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Overview

  • This study investigates how two natural compounds, chlorogenic acid (CGA) and isovanillic acid 3-O-sulfate (IVAS), individually and combined, affect muscle satellite cells from horses under heat stress, simulating exercise-induced damage.
  • The research focuses on cell recovery, mitochondrial function, apoptosis, and inflammatory responses to understand their potential for enhancing muscle regeneration and recovery in athletes.

Background and Purpose

  • Skeletal muscle satellite cells (SCs) are crucial for muscle repair and regeneration, making them an important model for exercise-related stress studies.
  • Exercise induces cellular stress, including heat shock, which challenges muscle cell viability and function.
  • Natural compounds such as CGA and IVAS have gained attention for their possible roles in natural recovery and regeneration strategies.
  • The purpose was to test these compounds’ effects on equine skeletal muscle satellite cells (ESCs), a relevant model for human muscle recovery due to physiological similarities.
  • The study specifically explored how CGA, IVAS, and their combination (Hybrid) influence cell viability, mitochondrial activity, apoptosis, nitric oxide production, and gene expression after heat-induced stress.

Methods

  • ESCs were exposed to heat shock at 40 °C for 1 hour to simulate exercise stress conditions.
  • Post-heat shock, cells were treated with CGA (0.005%), IVAS (0.0005%), or their combination for 24 hours.
  • Various assays and measurements were conducted:
    • Cell viability via MTS assay
    • Mitochondrial membrane potential assessment
    • Apoptosis detection through Annexin V/7-AAD staining
    • Nitric oxide (NO) production measurement
    • Gene expression profiling using RT-qPCR for antioxidant, inflammatory, apoptotic, and mitochondrial genes

Key Findings

  • Cell Viability:
    • CGA significantly enhanced cell viability under both normal and heat stress conditions (increase of ~216-227%).
    • IVAS improved viability only in cells not exposed to heat stress.
    • The combination (Hybrid) maintained cell viability beyond individual treatments.
  • Cell Morphology:
    • Only the Hybrid treatment preserved elongated morphology of ESCs after heat shock, indicating better structural preservation.
  • Nitric Oxide Production:
    • CGA increased NO levels, which are involved in cellular signaling and muscle repair.
    • No significant NO change was seen with IVAS or Hybrid treatments.
  • Gene Expression:
    • Antioxidant gene expression did not significantly change, suggesting these compounds do not primarily act through antioxidant pathways.
    • Proinflammatory cytokines IL-6 and IL-1β were significantly upregulated in the Hybrid group (2.74- and 5.64-fold increases), suggesting an adaptive immune response rather than harmful inflammation.
    • Apoptotic genes showed differential regulation:
      • BCL2 (anti-apoptotic) was downregulated.
      • BAX (pro-apoptotic) was strongly upregulated only in the Hybrid group (8.14-fold).
    • Mitochondrial-related genes MFN2, TFAM, and PUSL1 were significantly increased in the Hybrid group, indicating enhanced mitochondrial function and biogenesis.
    • MIRO1, involved in mitochondrial transport, was upregulated in all treated groups.
  • Apoptosis and Cell Death:
    • Early apoptosis increased in CGA and Hybrid groups (~34%), implying activation of controlled cell turnover processes.
    • The Hybrid group showed the lowest total cell death (6.26%), reflecting a protective effect possibly through mitochondrial and inflammatory regulation.

Interpretation and Implications

  • The study suggests that CGA and IVAS, especially when combined, synergistically enhance the recovery and survival of muscle satellite cells after stress.
  • The combination supports mitochondrial stability and function, crucial for energy production and cell survival during recovery.
  • The controlled upregulation of proinflammatory cytokines may represent a beneficial, adaptive immune response that facilitates regeneration rather than damaging inflammation.
  • Activation of apoptotic pathways alongside increased viability points to a fine-tuned balance between removing damaged cells and promoting muscle repair.
  • These findings support the use of natural bioactive compounds in muscle recovery therapies and interventions, potentially applicable to human athletes based on the equine model.

Conclusion

  • Natural compounds CGA and IVAS promote muscle cell recovery after exercise-like stress by enhancing mitochondrial function and regulating inflammatory and apoptotic responses.
  • Their combined use (Hybrid) provides superior benefits, maintaining cell viability and morphology, and activating adaptive mechanisms involved in muscle regeneration.
  • This research underlines the potential of nature-based compounds as effective elements in muscle recovery strategies for athletes.

Cite This Article

APA
Witkowska-Piłaszewicz O, Nowicka-Kazmierczak M, Pietrzak P, Marycz K. (2025). Mitohormesis and Regeneration: Natural Compounds Chlorogenic Acid (CGA) and Isovanillic Acid 3-O-sulfate (IVAS) Boost Muscle Cell Recovery in the Equine Athlete Model. Stem Cell Rev Rep, 21(8), 2654-2666. https://doi.org/10.1007/s12015-025-10959-9

Publication

ISSN: 2629-3277
NlmUniqueID: 101752767
Country: United States
Language: English
Volume: 21
Issue: 8
Pages: 2654-2666

Researcher Affiliations

Witkowska-Piłaszewicz, Olga
  • Department of Large Animals Diseases and Clinic, Institute of Veterinary Medicine, Warsaw University of Life Sciences, Nowoursynowska 166, Warsaw, 02- 787, Poland. olga_witkowska_pilaszewicz@sggw.edu.pl.
Nowicka-Kazmierczak, Małgorzata
  • International Institute of Translational Medicine (MIMT), ul. Jesionowa 11, Malin Wisznia Mała, 55-114, Poland.
Pietrzak, Patrycja
  • International Institute of Translational Medicine (MIMT), ul. Jesionowa 11, Malin Wisznia Mała, 55-114, Poland.
Marycz, Krzysztof
  • International Institute of Translational Medicine (MIMT), ul. Jesionowa 11, Malin Wisznia Mała, 55-114, Poland. krzysztofmarycz@gmail.com.
  • Veterinary Institute for Regenerative Cures, School of Veterinary Medicine, University of California-Davis, Davis, CA, USA. krzysztofmarycz@gmail.com.

MeSH Terms

  • Animals
  • Horses
  • Chlorogenic Acid / pharmacology
  • Apoptosis / drug effects
  • Membrane Potential, Mitochondrial / drug effects
  • Regeneration / drug effects
  • Cell Survival / drug effects
  • Mitochondria / drug effects
  • Mitochondria / metabolism
  • Muscle, Skeletal / drug effects
  • Nitric Oxide / metabolism

Grant Funding

  • INNOGLOBO/2/myo(miR)egen/10/2022 / Narodowe Centrum Badań i Rozwoju

Conflict of Interest Statement

Declarations. Ethics Approval: Muscles sampling procedures were conducted post mortem. Following the European Directive 2010/63/EU and Polish regulations on animal experimentation, ethical approval was not required and are therefore exempt from the directive. Consent to Participate: n/a. Consent for Publication: n/a. Competing Interests: The authors declare no competing interests. Conflict of interest: The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

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