Equine laminitis: congenital, hemidesmosomal plectin deficiency in a Quarter Horse foal.
Abstract: No abstract available
Publication Date: 2004-05-19 PubMed ID: 15147142DOI: 10.2746/0425164044877206Google Scholar: Lookup
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- Case Reports
- Journal Article
- Research Support
- Non-U.S. Gov't
Cite This Article
APA
French KR, Pollitt CC.
(2004).
Equine laminitis: congenital, hemidesmosomal plectin deficiency in a Quarter Horse foal.
Equine Vet J, 36(3), 299-303.
https://doi.org/10.2746/0425164044877206 Publication
Researcher Affiliations
- Australian Equine Laminitis Research Unit, School of Veterinary Science, Faculty of Natural Resources, Agriculture and Veterinary Science, The University of Queensland, Brisbane, Queensland 4072, Australia.
MeSH Terms
- Animals
- Animals, Newborn
- Fatal Outcome
- Foot Diseases / congenital
- Foot Diseases / pathology
- Foot Diseases / veterinary
- Hemidesmosomes / chemistry
- Hemidesmosomes / pathology
- Hemidesmosomes / ultrastructure
- Hoof and Claw / pathology
- Horse Diseases / congenital
- Horse Diseases / pathology
- Horses
- Immunohistochemistry / veterinary
- Inflammation / congenital
- Inflammation / pathology
- Inflammation / veterinary
- Intermediate Filament Proteins / deficiency
- Male
- Microscopy, Electron / veterinary
- Microscopy, Fluorescence / veterinary
- Plectin
- Skin / pathology
Citations
This article has been cited 2 times.- Wang L, Pawlak EA, Johnson PJ, Belknap JK, Eades S, Stack S, Cousin H, Black SJ. Impact of laminitis on the canonical Wnt signaling pathway in basal epithelial cells of the equine digital laminae.. PLoS One 2013;8(2):e56025.
- Wang L, Pawlak E, Johnson PJ, Belknap JK, Alfandari D, Black SJ. Effects of cleavage by a disintegrin and metalloproteinase with thrombospondin motifs-4 on gene expression and protein content of versican and aggrecan in the digital laminae of horses with starch gruel-induced laminitis.. Am J Vet Res 2012 Jul;73(7):1047-56.
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