Analyze Diet

Topic:Clinical Symptoms

Clinical symptoms in horses encompass a range of observable signs that may indicate underlying health issues, such as disease, injury, or physiological stress. These symptoms can vary widely depending on the condition and may include changes in behavior, appetite, respiratory patterns, or physical appearance. Common clinical symptoms in horses include lameness, colic, coughing, nasal discharge, and changes in body temperature or heart rate. Identifying and interpreting these symptoms is an essential aspect of equine veterinary practice, as they provide critical information for diagnosis and management of health conditions. This page compiles peer-reviewed research studies and scholarly articles that explore the identification, assessment, and implications of clinical symptoms in equine health management.
Multilobular osteoma (chondroma rodens) in a horse.
Journal of the American Veterinary Medical Association    February 1, 1983   Volume 182, Issue 3 289-291 
Richardson DW, Acland HM.No abstract available
Diaphyseal angular deformities in three foals.
Journal of the American Veterinary Medical Association    February 1, 1983   Volume 182, Issue 3 272-279 
White KK.Angular limb deformities in 3 foals were found to originate in the diaphyseal region of the 3rd metacarpal (2) and metatarsal (1) bones. In each case, treatment consisted of wedge ostectomy followed by compression plating. Two foals survived for useful performance; the 3rd was euthanatized because of ischemia of the operated limb. The condition appeared to resemble a similar syndrome in man involving tibial curvature.
Cantharidin toxicosis in horses.
Journal of the American Veterinary Medical Association    February 1, 1983   Volume 182, Issue 3 283-284 
Beasley VR, Wolf GA, Fischer DC, Ray AC, Edwards WC.No abstract available
Uroperitoneum in the foal.
Journal of the American Veterinary Medical Association    February 1, 1983   Volume 182, Issue 3 267-271 
Richardson DW, Kohn CW.The case records of 22 foals with uroperitoneum were reviewed. The most common cause was a defect in the urinary bladder. A sex predilection for males was apparent. Clinical signs were tachycardia, tachypnea, and abdominal distention. The history pertaining to micturition was variable. Hyponatremia, hypochloremia, and hyperkalemia were marked. A reliable means of diagnosis was considered to be the demonstration of a wide disparity in serum and peritoneal fluid creatinine values. Surgical repair was successful in 11 of 18 cases, although surgical and anesthetic complications were common. Some o...
Complications of abdominocentesis in the horse.
Journal of the American Veterinary Medical Association    February 1, 1983   Volume 182, Issue 3 232-234 
Tulleners EP.No abstract available
Renal papillary necrosis in horses.
Journal of the American Veterinary Medical Association    February 1, 1983   Volume 182, Issue 3 263-266 
Gunson DE.No abstract available
Cantharidin toxicosis in horses.
Journal of the American Veterinary Medical Association    February 1, 1983   Volume 182, Issue 3 283-284 
Beasley VR, Wolf GA, Fischer DC, Ray AC, Edwards WC.No abstract available
Selected skeletal dysplasias: craniomandibular osteopathy, multiple cartilaginous exostoses, and hypertrophic osteodystrophy.
The Veterinary clinics of North America. Small animal practice    February 1, 1983   Volume 13, Issue 1 55-70 doi: 10.1016/s0195-5616(83)50004-1
Alexander JW.No abstract available
Pathologic changes in 3-methylindole-induced equine bronchiolitis.
The American journal of pathology    February 1, 1983   Volume 110, Issue 2 209-218 
Turk MA, Breeze RG, Gallina AM.The pathologic features of bronchiolitis were studied in horses and ponies from 30 minutes to 27 days after an oral dose of 3-methylindole (3MI). From 30 minutes to 3 hours, lesions were limited to nonciliated bronchiolar epithelial (Clara) cells, which lost apical caps and cytoplasmic granules and had dilated smooth endoplasmic reticulum (SER). At 12 hours, necrotic Clara cells were exfoliated; degeneration and necrosis were evident, in bronchiolar ciliated cells. Rare epithelial cells with hyperplastic SER appeared on the denuded basal lamina at 24 hours. Inflammatory cells, epithelia, fibro...
Equine dentistry.
British dental journal    January 22, 1983   Volume 154, Issue 2 51 doi: 10.1038/sj.bdj.4804989
Knott NJ, Cull RS.No abstract available
Treatment of navicular disease.
The Veterinary record    January 22, 1983   Volume 112, Issue 4 90 doi: 10.1136/vr.112.4.90
Colles CM.No abstract available
Metaldehyde poisoning in horses.
The Veterinary record    January 15, 1983   Volume 112, Issue 3 64-65 doi: 10.1136/vr.112.3.64
Sutherland C.No abstract available
[A pony with “false” colic].
Tijdschrift voor diergeneeskunde    January 15, 1983   Volume 108, Issue 2 67-69 
van Wuijckhuise-Sjouke LA.Report on the case of a pony with colic submitted a to the department of internal medicine. However, the animal was found to be affected with dilatation of the oesophagus which had given rise to acute pleurisy resulting from rupture of the wall of the oesophagus. The diagnosis was established after death.
[Gasterophilosis (gastric myiasis) in the horse].
Tierarztliche Praxis    January 1, 1983   Volume 11, Issue 2 201-207 
Frahm J.No abstract available
Invagination of the caecal apex in a foal.
Equine veterinary journal    January 1, 1983   Volume 15, Issue 1 62-63 doi: 10.1111/j.2042-3306.1983.tb01710.x
Semrad SD, Moore JN.No abstract available
Relapsing idiopathic thrombocytopenia in a horse.
Equine veterinary journal    January 1, 1983   Volume 15, Issue 1 73-75 doi: 10.1111/j.2042-3306.1983.tb01715.x
Morris DD, Whitlock RH.TEIROMBOCYTOPENIA can be caused by failure of production or increased destruction of platelets, resulting in a bleeding diathesis, characterised by mucosal petechiac and haemorrhage into various organs. In humans, some drugs (cg, phenylbutazone and chloramphenicol) cause idiosyncratic marrow suppression and subsequent thrombocytopenia. Bone marrow suppression is also well documented in humans and small animals receiving cytotoxic drugs (Calabresi 1979; MacEwen 1980). Decreased megakaryocytic proliferation is rarely recognised in large animals. The most prevalent causes of thrombocytopenia are ...
[Effect of loading on respiration mechanics in horses with healthy and with diseased lungs].
Berliner und Munchener tierarztliche Wochenschrift    January 1, 1983   Volume 96, Issue 1 1-4 
Müller P, Deegen E, Fister D.No abstract available
Current concepts of infectious polyarthritis in foals.
Equine veterinary journal    January 1, 1983   Volume 15, Issue 1 5-9 doi: 10.1111/j.2042-3306.1983.tb01686.x
Firth EC.No abstract available
Effects of transportation, surgery, and antibiotic therapy in ponies infected with Salmonella.
American journal of veterinary research    January 1, 1983   Volume 44, Issue 1 46-50 
Owen RA, Fullerton J, Barnum DA.Seventeen ponies were infected with Salmonella typhimurium and then 15 were variously stressed by transportation and/or surgery and 9 were given oxytetracycline. Indications of Salmonella reactivation occurred in all the stressed ponies. Diarrhea due to a reactivation of the Salmonella infection did not develop until greater than 3 days after stress, although maximal shedding of organisms occurred within 24 hours. A neutropenia generally occurred within 24 hours after stress and lasted about 5 days. A rectal temperature greater than 39 C usually did not occur. An increase in serologic titer wa...
Cutaneous onchocerciasis in the horse: five cases in southwestern british columbia.
The Canadian veterinary journal = La revue veterinaire canadienne    January 1, 1983   Volume 24, Issue 1 3-5 
Lees MJ, Kleider N, Tuddenham TJ.Five horses were presented because of a dermatitis of the forehead. Unlike previous reports, ventral midline dermatitis was not the major problem, and was present in only two of five cases. All five horses responded to levamisole therapy at a daily dosage of 5.5 g for one week. Owners were cautioned that repeat therapy may be necessary.
[Histopathology of chronic obstructive bronchitis in clinically sick horses].
Tierarztliche Praxis    January 1, 1983   Volume 11, Issue 2 213-221 
Schoon HA, Deegen E.No abstract available
[Contact eczema due to metal ion allergy in a drill show horse].
Tierarztliche Praxis    January 1, 1983   Volume 11, Issue 3 339-344 
Fabry H.No abstract available
Neuroepithelial tumor of the optic nerve in a horse.
The Cornell veterinarian    January 1, 1983   Volume 73, Issue 1 30-40 
Bistner S, Campbell RJ, Shaw D, Leininger JR, Ghobrial HK.A nine-year-old horse was presented with severe exophthalmos of the right eye and a large mass in the vitreal cavity. The affected globe was enucleated and two months following surgery the horse was euthanized because of spread of the tumor into the calvarium producing compression of the brain stem and neurologic signs. Examination of tumor tissue by light and electron microscopic examination showed a mixed neoplasm of primitive neuroepithelium.
Blood fructose levels in the newborn foal.
Equine veterinary journal    January 1, 1983   Volume 15, Issue 1 60-61 doi: 10.1111/j.2042-3306.1983.tb01708.x
Rogers PA, Fahey GC, Albert WW.No abstract available
Ischaemic necrosis of the jejunum of a horse caused by a penetrating foreign body.
Equine veterinary journal    January 1, 1983   Volume 15, Issue 1 66-68 doi: 10.1111/j.2042-3306.1983.tb01712.x
Davies JV.No abstract available
Clinical aspects of lymphosarcoma in the horse: a clinical report of 16 cases.
Equine veterinary journal    January 1, 1983   Volume 15, Issue 1 49-53 doi: 10.1111/j.2042-3306.1983.tb01702.x
van den Hoven R, Franken P.This paper describes the clinical and laboratory findings in 16 horses suffering from lymphosarcoma and the autopsy results in 13 of these cases which were examined at the Large Animal Medicine Clinic, State University of Utrecht, from 1969 to 1981. The cases were classified into four groups, designated multicentric, alimentary, thymic and cutaneous forms, according to the situation of the tumours.
Pemphigus and pemphigoid in dogs, cats, and horses.
Annals of the New York Academy of Sciences    January 1, 1983   Volume 420 353-360 doi: 10.1111/j.1749-6632.1983.tb22223.x
Scott DW, Manning TO, Smith CA, Lewis RM.No abstract available
Navicular disease in the horse. The subchondral bone pressure.
Nordisk veterinaermedicin    January 1, 1983   Volume 35, Issue 1 31-37 
Svalastoga E, Smith M.The subchondral bone pressure in the navicular bone of horses with navicular disease is compared with corresponding pressures in normal horses. An increased intraosseous pressure and a lengthened pressure drop time was demonstrated in patients with navicular disease, which indicates the existence of a venous stasis. Compared with similar investigations in humans with arthrosis it is concluded that navicular disease is a condition resembling arthrosis. The classical clinical symptoms are explained as an expression of resting pains. Treatments are suggested which theoretically can render the pat...
[Experimental reproduction of lactic acidosis in the pony].
Annales de recherches veterinaires. Annals of veterinary research    January 1, 1983   Volume 14, Issue 3 195-206 
Wolter R, Durix A, Letourneau JC, Carcelen M, Henry N.One pony has been subjected to the intravenous injections of L-lactic acid. Two other ponies have been trained to intracaecal administration of L-lactic acid or sucrose. The obtained results show that: Intravenous injection of lactic acid increases the concentration of histamin and lactic acid, decreases the level of magnesium and reduces the pressure of carbon dioxide in the blood (the control animals and the treated animals) without the clinical symptoms of lactic acidosis. Intracaecal administration of lactic acid induces a high liberation of histamin in the caecum (the control animals and ...
What is your diagnosis? Sesamoid bone fractures in a horse.
Journal of the American Veterinary Medical Association    December 15, 1982   Volume 181, Issue 12 1543-1544 
Hathcock JT.No abstract available