Analyze Diet

Topic:Molybdenum

Molybdenum is a trace mineral found in the environment, including soil, water, and plants, and is an essential element in the diets of horses. It is involved in various enzymatic processes, acting as a cofactor for enzymes that facilitate the metabolism of sulfur-containing amino acids and the detoxification of certain compounds. The concentration of molybdenum in a horse's diet can influence its absorption and utilization of other minerals, such as copper, due to competitive interactions. Excessive levels of molybdenum can lead to imbalances and potential health issues, such as copper deficiency, which can affect growth and development. This page compiles peer-reviewed research studies and scholarly articles that explore the role, dietary requirements, and effects of molybdenum on equine health and nutrition.
Reference intervals for trace mineral and heavy metal concentrations in horse livers in the Netherlands. van der Merwe D, van den Wollenberg L, van Hees-Valkenborg J, de Haan T, van der Drift S.We determined reference intervals (RIs) for concentrations of trace minerals and toxic elements based on liver samples from 122 apparently healthy horses at 2 slaughter facilities in the Netherlands. Samples were collected during the spring and fall of 2021, and the sex and age of the horses were registered upon sampling. Concentrations of arsenic, cadmium, chromium, cobalt, copper, iron, lead, manganese, molybdenum, nickel, selenium, vanadium, and zinc were measured in liver samples using inductively coupled plasma-mass spectrometry (ICP-MS) after nitric acid digestion. RIs were calculated us...
Copper deficiency in cattle, sheep and horses caused by excess molybdenum from fly ash: a case report.
Veterinary and human toxicology    February 1, 1995   Volume 37, Issue 1 63-65 
Ladefoged O, Stürup S.A case of copper deficiency or molybdenum toxicosis in cattle, sheep and horses after heavy pollution of a pasture with fly ash is described. If the pastures had not been grazed by cattle and sheep as well as the horses, it would have been difficult to identify the reason for the intoxication in the horses. It is argued that molybdenum intoxication, although seldom seen in non-ruminants, was the cause of the deaths of the horses. It is suggested that the bioavailability of molybdenum in fly ash is high and therefore can cause equine intoxication.
Dietary molybdenum as a putative copper antagonist in the horse.
Equine veterinary journal    January 1, 1987   Volume 19, Issue 1 50-54 doi: 10.1111/j.2042-3306.1987.tb02581.x
Strickland K, Smith F, Woods M, Mason J.Four horses were stabled and fed a diet of hay ad libitum, and 2 kg oats per animal per day, for a month. The basic diet was then supplemented with molybdenum, at a rate of 20 mg/kg dry matter for 4.5 months. For one month of this period the diet was supplemented also with sulphur at a rate of 1.2 g/kg dry matter. Analyses of jugular blood samples, obtained at intervals varying between two and 20 days, showed no evidence of a decline in total plasma copper or of an increased proportion of trichloroacetic acid (TCA) insoluble copper in plasma over this period. In separate studies, two other hor...
Influence of dietary molybdenum on copper metabolism in ponies.
The Journal of nutrition    January 1, 1981   Volume 111, Issue 1 96-106 doi: 10.1093/jn/111.1.96
Cymbaluk NF, Schryver HF, Hintz HF, Smith DF, Lowe JE.The effects of molybdenum ingestion on copper metabolism were investigated in bile duct-cannulated ponies fed diets containing 1.01, 27.4 and 107.3 ppm molybdenum. Copper metabolism was assessed by examination of 64Cu and stable copper excretion in feces, bile and urine. The addition of molybdenum to the diet decreased copper absorption and retention as a consequence of increased excretion of dietary copper in feces and increased excretion of absorbed copper in bile. Only 5% or less of the absorbed copper was excreted in urine regardless of dietary treatment. Biliary molybdenum excretion was o...
Molybdenum content of equine liver tissue.
Nature    October 25, 1958   Volume 182, Issue 4643 1175-1176 doi: 10.1038/1821175b0
MOORE PA.No abstract available