Omneity® Pellets
All-In-One Vitamin & Mineral Pellet
Topic:Asymptomatic Carriers
Asymptomatic carriers are horses that harbor infectious agents without displaying clinical signs of disease. These carriers can shed pathogens into the environment, potentially transmitting infections to other animals. Understanding the role of asymptomatic carriers is important for managing disease spread within equine populations. Research in this area focuses on identifying specific pathogens that can be carried asymptomatically, the duration and conditions of carriage, and the implications for herd health management. This page aggregates peer-reviewed research studies and scholarly articles that explore the identification, epidemiology, and impact of asymptomatic carriers in equine health.
Renal coccidiosis of the horse associated with Klossiella equi. Klossiella equi in the kidney of a horse is described. Gametocytic and sporogonic stages were observed in the epithelium of the collecting tubules in association with schizonts in the glomeruli and proximal convoluted tubules. The condition was asymptomatic and caused only minor renal damage and inflammation.
Klebsiella and Enterobacter organisms isolated from horses. An account is given of K. pneumoniae capsule types occurring in horses, with particular reference to strains originating from the genital tract in the mare and the external genitalia of the stallion. A survey of the prevalence of K. pneumoniae and E. aerogenes strains in the preputial flora of healthy stallions is described. The majority of horses were found to be carriers of these organisms. The cultural characteristics of these preputial strains are described and compared with those of K. pneumoniae strains associated with epidemic metritis in mares. The epidemiological significance of certa...
Coital exanthema in stallions. Equine coital exanthema can be produced experimentally in stallions by inoculation with an equine herpesvirus (strain 65/61) and be transmitted during coitus with an infected mare. Serological responses to this infection include the production of complement-fixing and serum-neutralizing antibodies which reach maximum levels 14 to 21 days after infection. Complement-fixing antibodies decline rapidly and are usually not detectable by 60 days after infection, whereas serum-neutralizing antibody activity is maintained for at least 1 year. This disparity provides a useful method for the diagnosis o...
Proliferative Optic Neuropathy in Horses. An asymptomatic, ophthalmoscopically visible proliferation affected the optic disc and nerve of two aged horses. The lesion consisted of an accumulation of foamy cells, histologically akin to fat cells, which contained an unidentified lipid-like material. The affected area and its environs were permeated by tortuous, thickened blood vessels with heavy deposits of collagen in their walls. The neuropathy is considered to be a storage disease, and although the product stored is unidentified, the lesion is similar to that of human xanthelasma. The neuropathy seems distinct from the exudative optic...
