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Topic:Paralysis

Paralysis in horses refers to the loss of voluntary muscle function, which can result from a variety of neurological or musculoskeletal conditions. It can affect different parts of the horse's body and may be partial or complete. Causes of paralysis in horses include traumatic injuries, neurological disorders, infections, and toxicities. Common conditions associated with equine paralysis include equine protozoal myeloencephalitis (EPM), cervical vertebral stenotic myelopathy (CVSM), and botulism. Diagnosis and management of paralysis in horses require comprehensive veterinary evaluation, including neurological examinations and diagnostic imaging. This page compiles peer-reviewed research studies and scholarly articles that explore the causes, diagnostic approaches, and treatment options for paralysis in horses.
Borna disease in horses.
The Veterinary clinics of North America. Equine practice    February 24, 2001   Volume 16, Issue 3 579-xi doi: 10.1016/s0749-0739(17)30097-4
Richt JA, Grabner A, Herzog S.Borna disease is a sporadically occurring, progressive viral polioencephalomyelitis that primarily affects horses and sheep. The etiological agent, Borna disease virus (BDV), is an enveloped, single-stranded RNA virus that has been classified in the new virus family Bornaviridae within the order Mononegavirales. Serological evidence of BDV infection has been found in an increasing number of countries throughout the world. After an incubation period lasting a few weeks to several months, BDV infection can cause locomotor and sensory dysfunction followed by paralysis and death. Borna disease is ...
An in vitro comparison of cordopexy, cordopexy and laryngoplasty, and laryngoplasty for treatment of equine laryngeal hemiplegia.
Veterinary surgery : VS    August 5, 2000   Volume 29, Issue 4 326-334 doi: 10.1053/jvet.2000.5599
Jansson N, Ducharme NG, Hackett RP, Mohammed HO.To examine the effect of cordopexy, laryngoplasty, and cordopexy combined with a modified laryngoplasty on airway mechanics. Methods: Experimental airway mechanics were determined by subjecting equine cadaveric larynges to airflows similar to inspiratory airflow of exercising horses. Methods: Twenty equine larynges. Methods: Using cadaveric larynges, we developed and tested a new technique of arytenoid cartilage abduction. All larynges had the right arytenoid cartilage abducted to mimic the degree of arytenoid abduction that occurs at maximal exertion in live horses. Three surgical techniques ...
Ataxia and paresis with equine herpesvirus type 1 infection in a herd of riding school horses.
Journal of veterinary internal medicine    April 20, 2000   Volume 14, Issue 2 197-201 doi: 10.1892/0891-6640(2000)014<0197:aapweh>2.3.co;2
Friday PA, Scarratt WK, Elvinger F, Timoney PJ, Bonda A.An outbreak of neurologic disease associated with serologic evidence of equine herpesvirus type 1 (EHV-1) infection occurred in a herd of 46 riding school horses. Ataxia and paresis were observed in 14 geldings and 5 barren mares. Eight affected horses had distal limb edema, 1 horse had a head tilt, and 3 others had urinary incontinence. Other clinical signs included fever, depression, and inappetance in 30 horses. Seven horses with neurologic signs were treated with acyclovir. Serum neutralizing antibody titers against EHV-1 increased 4-fold between acute and convalescent samples or exceeded ...
Detection of Borna disease virus in a pregnant mare and her fetus.
Veterinary microbiology    March 23, 2000   Volume 72, Issue 3-4 207-216 doi: 10.1016/s0378-1135(99)00206-0
Hagiwara K, Kamitani W, Takamura S, Taniyama H, Nakaya T, Tanaka H, Kirisawa R, Iwai H, Ikuta K.A pregnant mare showing pyrexia, reduced appetite, ataxia and paresis was euthanized and examined for the presence of Borna disease virus (BDV). Her brain, showing multiple neuronal degeneration and necrosis with hemorrhage, and the histologically normal brain of the fetus were both positive for BDV RNA. The BDV nucleotide sequences were identical in the mare and fetus in the second open reading frame (ORF). This is the first report of the possible vertical transmission of BDV in a horse.
Shivering in a thoroughbred mare.
The Canadian veterinary journal = La revue veterinaire canadienne    March 21, 2000   Volume 41, Issue 2 128-129 
Davies PC.An 11-year-old mare presented with neuromuscular deficits and what resembled shivering in the left hind limb. On necropsy, there was no evidence of denervation atrophy of the left hind gastrocnemius muscle. The spinal cord had a small, right-sided lesion at C3-C4 and C4-C5. Tests for equine herpesvirus-1 and Sarcocystis spp. were negative.
Clinical and neuropathological features of West Nile virus equine encephalomyelitis in Italy.
Equine veterinary journal    February 8, 2000   Volume 32, Issue 1 31-35 doi: 10.2746/042516400777612080
Cantile C, Di Guardo G, Eleni C, Arispici M.West Nile (WN) virus infection is a mosquito-borne flavivirosis endemic in Africa and Asia. Clinical disease is usually rare and mild and only in a few cases the infection causes encephalomyelitis in horses, fever and meningoencephalitis in man. We report here the clinical and pathological findings in an epidemic of the disease involving 14 horses from Tuscany, Italy. All cases were observed from August to October 1998. Affected horses showed ataxia, weakness paresis of the hindlimbs and, in 6 cases, there was paraparesis progressing to tetraplegia and recumbency within 2 to 9 days. Eight anim...
Spinal cord compression secondary to hemangiosarcoma in a saddlebred stallion.
The Canadian veterinary journal = La revue veterinaire canadienne    January 26, 2000   Volume 40, Issue 12 886-887 
Berry S.Hemangiosarcoma in the spinal canal was diagnosed in a 25-year-old stallion showing progressive and symmetrical 4-limb ataxia, proprioceptive deficits, and weakness. On necropsy, an extradural mass consisting of spindle-shaped cells and numerous free erythrocytes was found at the level of C7-T1. Immunohistochemical staining confirmed a neoplasm of endothelial origin.
[Rhinopneumonia or mycotoxin intoxication? Neurologic phenomena in horses from a riding school].
Tijdschrift voor diergeneeskunde    December 22, 1999   Volume 124, Issue 22 679-681 
Sloet van Oldruitenborgh-Oosterbaan MM, Schipper FC, Goehring LS, Gremmels JF.In the course of several days most of the 40 riding-school horses turned out in paddocks developed ataxia of variable severity. Five of these horses showed severe ataxia and tremors, became paralyzed and were euthanized. Eleven privately-owned horses which were stabled on the same premises showed no clinical signs. The most likely diagnosis seemed to be the 'neurological form of EHV1', although the signs were not entirely typical. A few weeks later a second outbreak occurred among the riding-school horses and one of the privately-owned horses also showed signs of ataxia. In the meantime it had...
Hyperkalaemic periodic paralysis in horses: a review.
Equine veterinary journal    October 3, 1999   Volume 31, Issue 5 362-367 doi: 10.1111/j.2042-3306.1999.tb03834.x
Meyer TS, Fedde MR, Cox JH, Erickson HH.No abstract available
Clinicopathological features of equine primary hepatic disease: a review of 50 cases.
The Veterinary record    August 31, 1999   Volume 145, Issue 5 134-139 doi: 10.1136/vr.145.5.134
McGorum BC, Murphy D, Love S, Milne EM.The clinicopathological features of 50 cases of equine hepatic disease were reviewed. There was a wide range of clinical signs and at least 50 per cent of the animals exhibited either dull demeanour, anorexia, abdominal pain, cerebral dysfunction and/or weight loss. Life-threatening complications of hepatic failure recorded were: gastric impaction in 10 cases, bilateral laryngeal paralysis in seven cases and coagulopathy in five cases. All the cases had high activities of gamma-glutamyl transferase (GGT) and most had high activities of glutamate dehydrogenase (GLDH) and high concentrations of ...
[HYPP: hyperkalemic periodic paralysis in the horse].
Tijdschrift voor diergeneeskunde    April 3, 1999   Volume 124, Issue 6 176-181 
Sloet van Oldruitenborgh-Oosterbaan MM.Hyperkalaemic periodic paralysis(HYPP) is characterized by intermittent episodes of muscular tremor, weakness, and collapse, and is probably caused by abnormal electrolyte transport in the muscle cell membrane. During an episode of HYPP, most animals are severely hyperkalaemic. HYPP is a hereditary disease and occurs only in American Quarter horses or crossbreds. Because these horses are now being imported into the Netherlands, HYPP should be included in the differential diagnosis of horses showing signs of muscle tremor, paresis, or paralysis. The present article reviews the literature on HYP...
Paranasal sinus cyst as a cause of stridor in a standardbred colt.
The Canadian veterinary journal = La revue veterinaire canadienne    January 27, 1999   Volume 40, Issue 1 49-51 
Gilroy BJ, Lofstedt J, Pack L, McBurney SR.Sinus cysts are epithelium-lined, fluid-filled cavities that can occur in the paranasal sinuses of horses. Extensive damage to the permanent tooth buds was a significant feature in this case. The sequellae of these abnormalities, although not apparent at the time of presentation, remain an important consideration for prognosis.
Herpesvirus myeloencephalopathy in horses: 11 cases (1982-1996).
Journal of the American Veterinary Medical Association    September 10, 1998   Volume 213, Issue 5 671-675 
Donaldson MT, Sweeney CR.To determine results of CSF analysis in horses with equid herpesvirus myeloencephalopathy (EHM) and to determine whether results of CSF analysis were associated with outcome. Methods: Retrospective study. Methods: 11 horses. Methods: Medical records of all horses admitted to the veterinary teaching hospital between February 1982 and March 1996 in which EHM was diagnosed were reviewed. Results: 7 horses were < or = 4 years old; 8 were admitted during January, February, or March. Six horses were febrile prior to admission, but none was febrile on the day of admission. Five horses had been sta...
‘Stray voltage’ and sudden collapse in horses and cattle.
The Veterinary record    April 29, 1998   Volume 142, Issue 10 256 
Williams JB.No abstract available
Videoendoscopic assessment of dynamic laryngeal function during exercise in horses with grade-III left laryngeal hemiparesis at rest: 26 cases (1992-1995).
Journal of the American Veterinary Medical Association    February 21, 1998   Volume 212, Issue 3 399-403 
Hammer EJ, Tulleners EP, Parente EJ, Martin BB.To evaluate laryngeal function by means of videoendoscopy during high-speed treadmill exercise in racehorses with grade-III left laryngeal hemiparesis at rest and to determine outcome of treatment. Methods: Retrospective study. Methods: 26 racehorses. Methods: Videoendoscopy of the larynx was performed while horses were at rest and exercising on a treadmill. Horses were classified as having grade-IIIA, -IIIB, or -IIIC laryngeal hemiparesis on the basis of the degree of arytenoid cartilage abduction maintained during exercise. Postoperative racing performance was determined by evaluating race r...
Comparison of alveolar ventilation, oxygenation, pressure support, and respiratory system resistance in response to noninvasive versus conventional mechanical ventilation in foals.
American journal of veterinary research    December 24, 1997   Volume 58, Issue 12 1463-1467 
Hoffman AM, Kupcinskas RL, Paradis MR.To compare the efficacy of positive pressure ventilation applied through a mask versus an endotracheal tube, using anesthetized/paralyzed foals as a model for foals with hypoventilation. Methods: Six 1-month-old foals. Methods: A crossover design was used to compare the physiologic response of foals to 2 ventilatory techniques, noninvasive mask mechanical ventilation (NIMV) versus endotracheal mechanical ventilation (ETMV), during a single period of anesthesia and paralysis. Arterial pH, PaO2, PaCO2, oxygen saturation, end-tidal CO2 tension, airway pressures, total respiratory system resistanc...
[Salinomycin poisoning in a Polish stud horse].
Tierarztliche Praxis. Ausgabe G, Grosstiere/Nutztiere    August 1, 1997   Volume 25, Issue 5 438-441 
Nicpon J, Czerw P, Harps O, Deegen E.24 cases of salinomycin poisoning in horses occurring recently in Silesia are discussed. All of these horses, used for riding-purposes, were fed with concentrate containing 61 mg/kg salinomycin as faulty prepared by the manufacturer. Each horse received approximately two to three kilograms of this forage. All horses developed severe clinical signs of intoxication. Despite therapy eight horses died within three to six days. Ten others became recumbent and had to be euthanased. Only six horses survived. Clinical and laboratory examinations were performed and are discussed. Laboratory examination...
Effects of airway obstruction on transmural pulmonary artery pressure in exercising horses.
American journal of veterinary research    August 1, 1997   Volume 58, Issue 8 897-903 
Jackson JA, Ducharme NG, Hackett RP, Rehder RS, Ainsworth DM, Shannon KJ, Erickson BK, Erb HN, Jansson N, Soderholm LV, Thorson LM.To determine whether laryngeal hemiplegia would increase transmural pulmonary artery pressure (TPAP). Methods: 6 horses. Methods: Horses were studied under 5 conditions: control conditions, after induction of left laryngeal hemiplegia, during obstruction of the left nostril, after placement of an instrumented tracheostomy, and after placement of an open tracheostomy. Horses were evaluated after being given saline solution and after being given furosemide. Methods: Horses were exercised on a high speed treadmill, using a maximum speed of 13 m/s. During each exercise, airway pressures, airflow, ...
Postanesthetic hemorrhagic myelopathy or myelomalacia.
The Veterinary clinics of North America. Equine practice    April 1, 1997   Volume 13, Issue 1 73-77 doi: 10.1016/s0749-0739(17)30256-0
Trim CM.Hemorrhagic myelopathy or myelomalacia is an uncommon cause of failure to stand after general anesthesia. Affected horses are usually young and have been anesthetized for relatively short times in dorsal recumbency. Clinical signs involve the hind limbs and include loss of deep pain perception. Etiology of this condition is not known. Differentiation from other forms of neuropathy and myopathy is important to the prognosis.
Botulism.
The Veterinary clinics of North America. Equine practice    April 1, 1997   Volume 13, Issue 1 107-128 doi: 10.1016/s0749-0739(17)30259-6
Whitlock RH, Buckley C.Equine botulism is being recognized with increasing frequency by veterinarians throughout North America. Muscular weakness and dysphagia that progress during a period of 1 to 4 days, in the absence of laboratory derangements that indicate the presence of systemic disease, are suggestive of botulism. A tentative diagnosis usually is based on the presence of the following findings on physical examination: delayed pupillary light response, mydriasis, ptosis, generalized weakness, decreased tail tone, and slow prehension of feed. Definitive diagnosis requires detection of botulinum toxin in plasma...
Acute paraplegia in a thoroughbred racehorse after general anaesthesia.
Australian veterinary journal    March 1, 1997   Volume 75, Issue 3 178-179 doi: 10.1111/j.1751-0813.1997.tb10060.x
Raidal SR, Raidal SL, Richards RB, Cosgriff AV, Rose KJ.No abstract available
Fracture of the 7th cervical and 1st thoracic vertebrae presenting as radial nerve paralysis in a horse.
The Canadian veterinary journal = La revue veterinaire canadienne    February 1, 1997   Volume 38, Issue 2 112 
Lopez MJ, Nordberg C, Trostle S.No abstract available
Immunohistochemical demonstration of equine herpesvirus-1 antigen in neurons and astrocytes of horses with acute paralysis.
Veterinary pathology    January 1, 1997   Volume 34, Issue 1 52-54 doi: 10.1177/030098589703400110
Schultheiss PC, Collins JK, Hotaling SF.Equine herpesvirus-1 (EHV-1) infection in a few widely scattered neurons and astrocytes plus endothelial cells in brain and spinal cord of two horses with naturally occurring paralytic disease was demonstrated by use of an immunoperoxidase technique. These horses were euthanatized less than 48 hours after the onset of clinical signs. No staining for EHV-1 was demonstrated in brain or spinal cord of three horses that had a longer duration of clinical disease or in two uninfected horses.
[Immunopathology of Borna disease in the horse: clinical, virological and neuropathologic findings].
Tierarztliche Praxis    December 1, 1996   Volume 24, Issue 6 567-576 
Bilzer T, Grabner A, Stitz L.Tissues from nine horses and one donkey suffering from natural Borna disease were investigated. Clinically, all animals demonstrated progressive reduced mentation and aggravating gait disturbances. During the clinical course anorexia and progressive loss of proprioception were observed. Cranial nerve failure was accompanied by signs of pharyngeal paralysis, sialorrhea, bruxism, and by blindness. Virologically, infectious virus was detected in the brain of all animals investigated but was not found regularly in all areas of the brain. However, in all cases, infectivity was found in the thalamus...
The equine periodic paralysis Na+ channel mutation alters molecular transitions between the open and inactivated states.
The Journal of physiology    December 1, 1996   Volume 497 ( Pt 2), Issue Pt 2 349-364 doi: 10.1113/jphysiol.1996.sp021773
Hanna WJ, Tsushima RG, Sah R, McCutcheon LJ, Marban E, Backx PH.1. The Na+ channel mutation associated with equine hyperkalaemic periodic paralysis (HPP) affects a highly conserved phenylalanine residue in an unexplored region of the alpha-subunit. This mutation was introduced into the rat skeletal muscle Na+ channel gene at the corresponding location (i.e. F1412L) for functional expression and characterization in Xenopus oocytes. 2. In comparison with wild-type (WT) channels, equine HPP channels showed clear evidence for disruption of inactivation: increased time-to-peak current, slowed rates of whole-cell current decay, significant increases in sustained...
Efficacy of prosthetic laryngoplasty with and without bilateral ventriculocordectomy as treatments for laryngeal hemiplegia in horses.
American journal of veterinary research    November 1, 1996   Volume 57, Issue 11 1668-1673 
Tetens J, Derksen FJ, Stick JA, Lloyd JW, Robinson NE.To evaluate the efficacy of prosthetic laryngoplasty with and without bilateral ventriculocordectomy for treatment of experimentally induced left laryngeal hemiplegia (LLH). Methods: 15 adult Standardbreds. Methods: Horses were allotted to 3 equal groups. Sham operation (group 1), prosthetic laryngoplasty (group 2), or prosthetic laryngoplasty with bilateral ventriculocordectomy (group 3) was performed after induction of LLH. Upper airway function testing was performed prior to left recurrent laryngeal neurectomy (LRLN), 14 days after LRLN, and 60 and 180 days after surgical treatment. Measure...
Negative pressure pulmonary edema as a post-anesthetic complication associated with upper airway obstruction in a horse.
Veterinary surgery : VS    November 1, 1996   Volume 25, Issue 6 519-523 doi: 10.1111/j.1532-950x.1996.tb01453.x
Tute AS, Wilkins PA, Gleed RD, Credille KM, Murphy DJ, Ducharme NG.An 8-year-old Trakehner mare developed fulminant pulmonary edema following suspected upper airway obstruction 50 minutes into an otherwise unremarkable anesthetic recovery after surgery for left cricoarytenoideus dorsalis muscle reinnervation and ventriculocordectomy. Establishing a patent airway by orotracheal reintubation and cardiopulmonary resuscitation attempts were unsuccessful. Gross, histological, and electron microscopic postmortem examination showed severe hemorrhagic pulmonary edema. Laryngeal swelling or hemorrhage were not evident, suggesting laryngospasm or functional airway coll...
Laryngeal and pharyngeal dysfunction in horses homozygous for hyperkalemic periodic paralysis.
Journal of the American Veterinary Medical Association    August 15, 1996   Volume 209, Issue 4 798-803 
Carr EA, Spier SJ, Kortz GD, Hoffman EP.Evaluate histories, clinical signs, and laboratory data of 69 horses homozygous by DNA testing for hyperkalemic periodic paralysis (HPP). Methods: Cohort study. Methods: 69 of 189 horses testing homozygous for HPP between October 1992 and November 1994. Methods: Questionnaires addressing signalment, training regimes, medical history, and current status of affected horses were sent to owners, trainers, or attending veterinarians. Data from completed questionnaires were tabulated and evaluated, using descriptive statistics. Results: Sixty-nine (37%) of 189 questionnaires were completed and retur...
Clinical presentation of experimentally induced rabies in horses.
Zentralblatt fur Veterinarmedizin. Reihe B. Journal of veterinary medicine. Series B    July 1, 1996   Volume 43, Issue 5 277-285 doi: 10.1111/j.1439-0450.1996.tb00315.x
Hudson LC, Weinstock D, Jordan T, Bold-Fletcher NO.Twelve naive and nine test-vaccinated horses which developed clinical signs of rabies as a result of the required protocol of a vaccine trial were prospectively observed. Nineteen of the 21 cases were confirmed positive for rabies infection of the brain by fluorescent antibody test. The two horses with negative results had ganglioneuritis of the trigeminal ganglion or lymphocytic perivascular cuffing in the brain stem in addition to clinical signs. Average incubation period was 12.3 days and average morbidity was 5.5 days. Naive animals had significantly shorter incubation and morbidity period...
Potentiation of acetylcholine release from tracheal parasympathetic nerves by cAMP.
The American journal of physiology    April 1, 1996   Volume 270, Issue 4 Pt 1 L541-L546 doi: 10.1152/ajplung.1996.270.4.L541
Zhang XY, Robinson NE, Zhu FX.We tested the hypothesis that increasing intracellular levels of adenosine 3', 5'-cyclic monophosphate (cAMP) increases acetylcholine (ACh) release from airway parasympathetic nerves. Muscle strips from equine trachea were preincubated for 60 min with 10(-7)M atropine, 10(-6)M neostigmine, and 10(-5) M guanethidine. The ACh release was evoked by electrical field stimulation (EFS, 20 V, 0.5 ms, 0.5 Hz) and measured by high-performance liquid chromatography with electrochemical detection. Agents known to increase cAMP, i.e., forskolin (10(-6) - 10(-4) M), 8-bromoadenosine 3', 5'-cyclic monophosp...