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Topic:Clinical Signs

Clinical signs in horses refer to observable physical or behavioral manifestations that may indicate the presence of disease or injury. These signs can include a range of symptoms such as changes in appetite, alterations in gait, abnormal respiratory patterns, or variations in body temperature. Recognizing clinical signs is an integral part of equine veterinary practice, as they provide initial insights into the health status of the animal. Veterinarians rely on these signs to formulate differential diagnoses and guide further diagnostic testing. This page gathers peer-reviewed research studies and scholarly articles that explore the identification, interpretation, and diagnostic value of clinical signs in equine health management.
Acquired inguinal hernia in the horse: a review of 27 cases.
Journal of the American Veterinary Medical Association    February 1, 1982   Volume 180, Issue 3 317-320 
Schneider RK, Milne DW, Kohn CW.Twenty-seven cases of acquired inguinal hernia in the horse were reviewed. The history, clinical signs, treatment, and complications were summarized. Mortality was 25.9%. Only 13 or the 27 cases were diagnosed prior to admission. With early diagnosis and treatment, mortality should approach zero. In 24 of these cases, the hernia was strangulated. In each case, the signs were colic, a firm slightly enlarged testicle in the scrotum, and small intestine displacement through the inguinal ring, as determined via rectal palpation. In was concluded that palpation of the scrotum and inguinal rings sho...
[Diagnosis of osteochondrosis dissecans in the horse].
Berliner und Munchener tierarztliche Wochenschrift    January 15, 1982   Volume 95, Issue 2 26-30 
Hofmann R, Schönbauer M.No abstract available
[The medical killing of animals].
Tierarztliche Praxis    January 1, 1982   Volume 10, Issue 4 553 
Müller R.No abstract available
Equine laryngeal hemiplegia. Part III. Treatment by laryngoplasty.
New Zealand veterinary journal    January 1, 1982   Volume 30, Issue 1-2 1-5 doi: 10.1080/00480169.1982.34857
Goulden BE, Anderson LG.During the years 1971-1979, 127 horses with left laryngeal hemiplegia were studied. The physical characteristics and clinical signs observed in this study were recorded in Part I and Part II of ths series of papers. Of these 127 horses, 81 were treated by the laryngoplasty procedure. Complications of surgery are described and the effects of the operation on respiratory noise and performance are evaluated. In 54.8% of horses the chronic respiratory noise observed during exercise was apparently diminished or eliminated post-operatively. The performance of 44% of horses was apparently improved af...
Chemical-induced lung injury in domestic animals.
Advances in veterinary science and comparative medicine    January 1, 1982   Volume 26 201-231 
Breeze RG, Carlson JR.No abstract available
Clinical and serological observations on horses with suspected leptospirosis.
Australian veterinary journal    November 1, 1981   Volume 57, Issue 11 528-529 doi: 10.1111/j.1751-0813.1981.tb05798.x
Swan RA, Williams ES, Taylor EG.No abstract available
Equine laryngeal hemiplegia part II: Some clinical observations.
New Zealand veterinary journal    November 1, 1981   Volume 29, Issue 11 194-198 doi: 10.1080/00480169.1981.34846
Goulden BE, Anderson LJ.Some clinical features of laryngeal hemiplegia in 127 horses are described. Possible aetiologic factors were found in only 11% of affected animals. The onset of clinical signs was either sudden or insidious. The majority of cases were presented because of an abnormal respiratory noise made at exercise. Other clinical signs, particularly those usually attributed to laryngopalatal dislocation, were observed in a substantial proportion of affected animals. In all cases surveyed the left arytenoid was affected, although in 3 animals a bilateral laryngeal dysfunction was noted. Thirty of 65 animals...
Chronic respiratory disease in a horse infected with Dictyocaulus arnfieldi.
Journal of the American Veterinary Medical Association    October 15, 1981   Volume 179, Issue 8 820-822 
George LW, Tanner ML, Roberson EL, Burke TM.A 6-year-old Thoroughbred gelding was examined because of respiratory disease that developed after it was placed on pasture occupied by a donkey. Clinical signs in the gelding included a harsh, dry paroxysmal cough and increased expiratory effort. Eosinophils were seen in smears of mucus aspirated from the trachea and the bronchi. Immature, 5th-stage Dictyocaulus arnfieldi was identified in the tracheal mucus. The cough and other clinical signs were not diminished by corticosteroid therapy or by the administration of bronchodilators. Treatment with 10-fold therapeutic dosages of thiabendazole ...
Studies of an outbreak of Corynebacterium equi pneumonia in foals.
Equine veterinary journal    October 1, 1981   Volume 13, Issue 4 223-228 doi: 10.1111/j.2042-3306.1981.tb03500.x
Smith BP, Robinson RC.Five out of 6 foals between 2 and 4 months old, on a ranch in northern California, developed pneumonia within a 3 week period in June and July 1978. Corynebacterium equi was recovered from each of the 5 foals by transtracheal aspiration. Clinical signs were variable but included increased respiratory rate, fever, cough, nasal discharge, harsh airway sounds over middle sized airways and wheezing over small airways. Cyanosis was present in the most severely affected foal. Radiographic findings included diffusely increased interstitial and peribronchial densities, areas of consolidation and, in 3...
Prophylactic effects of sodium cromoglycate on chronic obstructive pulmonary disease in the horse.
Equine veterinary journal    October 1, 1981   Volume 13, Issue 4 243-246 doi: 10.1111/j.2042-3306.1981.tb03507.x
Thomson JR, McPherson EA.When stabled in a controlled environment, horses effected with chronic obstructive pulmonary disease (COPD) became clinically asymptomatic in 4 to 32 days (mean [+/-sd] 9.1 +/- 4.9 days), the time being influenced most by the severity of the disease judged on maximum intrathoracic pressure change (Max delta Ppl) and the age of the animal. Sodium cromoglycate, a drug widely used for prophylaxis of allergic respiratory disease in man, was administered by inhalation to 56 COPD-affected horses. The results showed that a linear response existed between the number of successive days treatment with t...
Phenylbutazone toxicosis in equidae: a biochemical and pathophysiological study.
American journal of veterinary research    October 1, 1981   Volume 42, Issue 10 1754-1759 
Snow DH, Douglas TA, Thompson H, Parkins JJ, Holmes PH.Toxic effects of phenylbutazone (PBZ) in ponies and horses were studied, using a variety of biochemical, pathophysiologic, and pathologic methods. At dosage levels of 10 to 12 mg/kg of body weight/day for 8 to 10 days, ponies frequently developed clinical signs of toxicosis characterized by hypoproteinemia. Studies using 51CrCl3 demonstrated that PBZ caused a protein-losing gastroenteropathy. The plasma loss was usually associated with gastrointestinal ulceration, but sometimes occurred without obvious lesions in mildly affected animals. Similar studies (8.2 mg/kg/day for 13 days) in Thoroughb...
Clinical toxicosis and erythrocyte cholinesterase inhibition of trichlorfon combined with mebendazole in horses.
American journal of veterinary research    September 1, 1981   Volume 42, Issue 9 1645-1650 
Gingerich DA, Mia AS.Thirty adult horses were used to compare the toxicity and cholinesterase (ChE) inhibition of various dosages of a combination anthelmintic, trichlorfon + mebendazole. Single oral doses of up to 5 times the effective dosage (39.7 mg of trichlorfon and 8.8 mg of mebendazole/kg of body weight) did not result in deaths. Horses given a placebo and horses treated at the recommended dosage rate showed few or no side effects, whereas horses given higher dosages showed dosage-related increases in the severity of clinical signs of organophosphate toxicosis. Dosage-related inhibition of erythrocyte ChE a...
Equine schistosomus fetus.
The Veterinary record    August 8, 1981   Volume 109, Issue 6 125 doi: 10.1136/vr.109.6.125-c
Johnstone R.No abstract available
Loss cause trends, halter accidents, and observations of the 1980 AAEP meeting.
Modern veterinary practice    July 1, 1981   Volume 62, Issue 7 547-550 
Greenhall JE.No abstract available
Pathologic features of horses given avirulent equine arteritis virus intramuscularly.
American journal of veterinary research    July 1, 1981   Volume 42, Issue 7 1218-1220 
McCollum WH.Twenty horses that were seronegative for equine arteritis virus antibodies were inoculated IM with live equine arteritis virus vaccine. The inoculation did not cause clinical signs of disease. A mild, transient febrile reaction developed in 6 horses, 3 of which were in poor condition before inoculation. Six horses, 2 of which were in poor condition before inoculation, experienced mild lymphopenia. Necropsy revealed mild lesions in the lymph nodes of 6 horses (3 of which were in poor condition before inoculation). Maximum concentrations of virus were detected in the lymph nodes and were consist...
A comparative study of the toxicity of Fusarium verticillioides (= F. moniliforme) to horses, primates, pigs, sheep and rats.
The Onderstepoort journal of veterinary research    June 1, 1981   Volume 48, Issue 2 129-131 
Kriek NP, Kellerman TS, Marasas WF.An isolate of Fusarium verticillioides (MRC826) that induced experimental leukoencephalomalacia, also caused acute toxicity when fed to pigs and administered per rumen fistula to sheep. Pigs developed severe pulmonary oedema while sheep manifested severe nephrosis and hepatosis. A less toxic isolate (F. verticillioides MRC602), fed to baboons, resulted in acute congestive heart failure or hepatic cirrhosis, depending on the dose. Both isolates were toxic to rats and caused similar lesions, namely, hepatic cirrhosis and intraventricular cardiac thrombosis.
Equine colitis “X”, still an enigma?
The Canadian veterinary journal = La revue veterinaire canadienne    May 1, 1981   Volume 22, Issue 5 162-165 
Schiefer HB.Colitis "X" is a sporadic diarrheal disease of horses with clinical signs of dehydration, electrolyte imbalances and "shock"-like features. Macroscopic and microscopic findings include signs of disseminated intravascular coagulation, necrosis of colonic mucosa and presence of large numbers of bacteria in the devitalized parts of the intestine. Recently published work suggests that the causative agent may be Clostridium perfringens, Type A, but the bacteria are recoverable only in the preliminary stages of the disease. Excess protein and lack of cellulose content in the diet is thought to be th...
Drugs used to produce standing chemical restraint in horses.
The Veterinary clinics of North America. Large animal practice    May 1, 1981   Volume 3, Issue 1 17-44 doi: 10.1016/s0196-9846(17)30144-1
Muir WW.No abstract available
Equine protozoal myeloencephalitis: a report of two cases from Western Canada.
The Canadian veterinary journal = La revue veterinaire canadienne    May 1, 1981   Volume 22, Issue 5 140-144 
Clark EG, Townsend HG, McKenzie NT.Two cases of nonsuppurative myeloencephalitis are reported which clinically and pathologically resemble equine protozoal myeloencephalitis. Lesions in both horses were associated with Toxoplasma-like organisms visible in microscopic sections. Clinical signs and lesions in one case primarily involved the brain and in the other case principally involved the spinal cord and associated meninges. Positive identification of the organisms was not achieved; however, the etiological agent is unlikely to be a species of Toxoplasma. Recently published studies suggest a species of Sarcocystis is involved....
Osteochondrosis of the shoulder joint of the horse.
The Cornell veterinarian    April 1, 1981   Volume 71, Issue 2 149-163 
Nyack B, Morgan JP, Pool R, Meagher D.Osteochondrosis of the shoulder joint in the horse was diagnosed radiographically in 54 shoulders of 38 horses. Clinical signs were those of an intermittent lameness characterized as a swinging leg shoulder lameness with pain elicited by extension, flexion or abduction of the limb. Diagnosis of shoulder lameness was supported by blocking the shoulder joint with local anesthetics. Radiographic changes consisted of: (1) alteration in the contour of the humeral head and glenoid cavity, (2) periarticular osteophyte formation, (3) sclerosis of the subchondral bone, and (4) bone cyst formation. The ...
Field outbreaks of leukoencephalomalacia in horses consuming maize infected by Fusarium verticillioides (= F. moniliforme) in South Africa.
Journal of the South African Veterinary Association    March 1, 1981   Volume 52, Issue 1 21-24 
Pienaar JG, Kellerman TS, Marasas WF.Four outbreaks of leukoencephalomalacia in horses in widely separated areas in South Africa are reported. The clinical signs and pathological lesions observed in each outbreak are briefly described. Mouldy home-grown maize from which Fusarium verticillioides (Sacc.) Nirenberg (= F. moniliforme Sheldon) was isolated in every instance, was involved in the outbreaks. Clinical signs and pathological lesions were identical to those seen in experimentally produced cases of F. verticillioides poisoning in horses.
Toxic effects of lasalocid in horses.
American journal of veterinary research    March 1, 1981   Volume 42, Issue 3 456-461 
Hanson LJ, Eisenbeis HG, Givens SV.Lasalocid was given to horses in a series of sequentially increasing single oral doses ranging between 5 and 30 mg/kg of body weight, with an appropriate washout period between treatments. One of the 5 horses died after a dosage of 15 mg/kg, 1 of 3 horses died after 21 mg/kg, 1 of 3 horses died after 22 mg/kg, and 1 of 2 horses died after 26 mg/kg. The LD50 of lasalocid for horses was estimated to be 21.5 mg/kg. Monensin was given to horses in a similar manner at dosages of 1, 2, and 3 mg/kg of body weight. One of the 2 horses died after a dosage of 2 mg/kg and 1 horse died after a dosage of 3...
Idiopathic hypersegmentation of neutrophils in a horse.
Journal of the American Veterinary Medical Association    February 1, 1981   Volume 178, Issue 3 303-305 
Prasse KW, George LW, Whitlock RH.No abstract available
The value of zinc protoporphyrin in equine lead poisoning: a case report.
Veterinary and human toxicology    February 1, 1981   Volume 23, Issue 1 12-15 
Kowalczyk DF, Naylor JM, Gunson D.A diagnosis of lead poisoning in a foal was established based on clinical signs and the presence of an elevation in the concentration of blood lead (40 microgram/dl). Additionally, ZPP was determined in whole blood and found to be significantly elevated compared to 10 normal horses not exposed to lead (634 vs 73 microgram/dl). An elevation in ZPP in man has been found to be a sensitive biochemical indicator of lead toxicity even before overt clinical signs are present. This report suggests that ZPP may be a useful screening test for evaluating biological toxicity due to lead in horses and poss...
Brainstem auditory evoked response in the diagnosis of inner ear injury in the horse.
Journal of the American Veterinary Medical Association    February 1, 1981   Volume 178, Issue 3 282-286 
Marshall AE, Byars TD, Whitlock RH, George LW.Brainstem auditory evoked response (BAER) testing was done to evaluate inner ear/VIIIth cranial nerve (CN8) function in the horse. The BAER test consisted of stimulating the auditory system with clicks and recording far-field responses of the brainstem auditory components via cutaneous electrodes and a signal averaging system. The normal response was shown to be a series of waves occurring within the first 10 msec after the stimulus click. Functional loss of the auditory receptor organ (cochlea) or CN8 results in loss of the entire response on the side of the injury. Because of the anatomic re...
Runners’ hemoglobinuria–in men, dogs and horses.
The Pharos of Alpha Omega Alpha-Honor Medical Society. Alpha Omega Alpha    January 1, 1981   Volume 44, Issue 1 33 
Altschule MD.No abstract available
Dermatologic problems in horses.
Modern veterinary practice    January 1, 1981   Volume 62, Issue 1 75-76 
No abstract available
Contagious equine metritis: effect of vaccination on control of the disease.
American journal of veterinary research    January 1, 1981   Volume 42, Issue 1 45-48 
Sahu SP.Pony mares were vaccinated with killed contagious equine metritis (CEM) bacteria by IV, subcutaneous, and intrauterine (IU) routes (or a combination of these routes). The serum agglutinating antibody titer varied from 1:64 to 1:1,024 after vaccination. In pony mares challenge exposed with 96-hour-old culture of CEM bacteria given by IU route, there were clinical signs of CEM, but these signs were less severe in vaccinated mares than in nonvaccinated mares. The bacterium was isolated for the exudate and from uterine samples collected from the mares after challenge exposure. A low titer of IU an...
Effects of copper pretreatment upon toxicity of selenium in ponies.
American journal of veterinary research    December 1, 1980   Volume 41, Issue 12 1925-1928 
Stowe HD.Thirty-four adult ponies were used to determine the effects of single oral doses of copper (Cu) supplements (0, 20, and 40 mg of Cu/kg of body weight) on the toxicity of oral doses of selenium (Se) supplements (0, 2, 4, 6, and 8 mg of Se/kg of body weight) administered 24 hours after the copper was given. Signs of Se toxicosis-sweating, diarrhea, tachycardia, tachypnea, mild pyrexia, lethargy, and colic-developed in ponies given 6 and 8 mg of Se/kg of body weight without Cu pretreatment. Two of 4 ponies given 6 mg of Se/kg and both ponies given 8 mg of Se/kg without Cu pretreatment died within...
Diagnostic endocrinology of the horse.
The Veterinary clinics of North America. Large animal practice    November 1, 1980   Volume 2, Issue 2 253-265 doi: 10.1016/s0196-9846(17)30160-x
Stabenfeldt GH, Hughes JP.No abstract available
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