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Topic:Disease Outbreaks

Disease outbreaks in horses refer to the occurrence and spread of infectious diseases within equine populations. These outbreaks can be caused by various pathogens, including bacteria, viruses, and parasites, and can lead to significant health issues in affected horses. Common diseases that may result in outbreaks include equine influenza, equine herpesvirus, strangles, and equine infectious anemia. The transmission of these diseases can occur through direct contact, environmental exposure, or vectors such as insects. Disease outbreaks can have substantial impacts on horse health, welfare, and the equine industry as a whole. This page compiles peer-reviewed research studies and scholarly articles that explore the epidemiology, transmission dynamics, and management strategies associated with disease outbreaks in equine populations.
Serological titers of equine monocytic ehrlichiosis associated with gastro-intestinal disorders and serological follow-up on two endemic farms.
Veterinary microbiology    April 1, 1993   Volume 34, Issue 4 345-353 doi: 10.1016/0378-1135(93)90059-g
Farrar WP, Bech-Nielsen S, Gordon JC, Reed SM, Pretzman CI, Kohn CW.The purpose of this work was to study the association of positive serological titers to Ehrlichia risticii, the causative agent of equine monocytic ehrlichiosis (EME) with gastro-intestinal disorders in hospitalized horses referred to The Ohio State University College of Veterinary Medicine Teaching Hospital (OSU VMTH). In addition, serological titers for E. risticii were monitored in two horse populations with endemic EME for one season to monitor temporal changes in titers. A statistically significant difference was found between the proportion of the total hospitalized horse population pres...
Characterisation of equine influenza isolates from the 1987 epizootic in India by nucleotide sequencing of the HA1 gene.
Equine veterinary journal    March 1, 1993   Volume 25, Issue 2 99-102 doi: 10.1111/j.2042-3306.1993.tb02916.x
Gupta AK, Yadav MP, Uppal PK, Mumford JA, Binns MM.Two A/Equi-2 (H3N8) isolates were obtained during the 1987 Indian equine influenza epizootic. The sequence of the Ludhiana/87 HA1 gene revealed that this isolate was very similar to recent European and North American isolates of equine influenza. In contrast, the Bhiwani/87 HA1 gene was nearly identical to the Miami/63 prototype H3 sequence. These results support the antigenic analysis previously carried out on these isolates using monoclonal antibodies. However, the finding that Bhiwani/87 is so similar to Miami/63, coupled with the finding that equine H3N8 influenza viruses have previously b...
‘Frozen’ evolution of equine influenza viruses?
Equine veterinary journal    March 1, 1993   Volume 25, Issue 2 87 doi: 10.1111/j.2042-3306.1993.tb02912.x
Wood JM.No abstract available
Recurrent diarrhoea in aged ponies associated with larval cyathostomiasis.
Equine veterinary journal    March 1, 1993   Volume 25, Issue 2 161-163 doi: 10.1111/j.2042-3306.1993.tb02929.x
Mair TS.No abstract available
Report of the Thirteenth Meeting of the OIE Ad hoc Group on Non Tsetse-Transmitted Animal Trypanosomoses.
Revue scientifique et technique (International Office of Epizootics)    March 1, 1993   Volume 12, Issue 1 237-272 
Touratier L.There is increasing interest in many parts of the world in trypanosomoses other than those transmitted by tsetse flies, as shown by numerous research projects and field studies. The refinement of techniques for studying the behaviour of trypanosomes (techniques of molecular biology) in axenic culture or in the parasitised host has led to progress in diagnosis and immunology, and a rational approach to chemotherapy and chemoprophylaxis of these infections. Field trials of enzyme-linked immunosorbent assays in Africa, Asia and South America have shown that these tests may now be regarded as reli...
Equine influenza in Hong Kong.
The Veterinary record    February 6, 1993   Volume 132, Issue 6 144 doi: 10.1136/vr.132.6.144-a
Watkins KL, Shortridge KF, Powell DG.No abstract available
Antibody responses of Japanese horses to influenza viruses in the past few years.
The Journal of veterinary medical science    February 1, 1993   Volume 55, Issue 1 33-37 doi: 10.1292/jvms.55.33
Goto H, Yamamoto Y, Ohta C, Shirahata T, Higuchi T, Ohishi H.A total of 305 horse sera collected in the Hidaka district of Hokkaido in the years 1988-90 were tested for the presence of hemagglutination-inhibition (HI) antibodies to A/equine/Newmarket/1/77 (H7N7), A/equine/Tokyo/2/71 (H3N8) and A/equine/Kentucky/1/81 (H3N8, Kentucky) strains of equine influenza (EI) virus. Antibodies to the 3 strains were detected in hardly of the 45 sera from 2-years-old horses which were collected before vaccination. Many of the 51 horses, after vaccination with inactivated EI virus, had HI antibodies to the 3 strains in 37 to 88 per cent. However, the number of positi...
Rhodococcus equi infection in foals: a report of an outbreak on a thoroughbred stud in Zimbabwe.
The Veterinary record    January 23, 1993   Volume 132, Issue 4 79-85 doi: 10.1136/vr.132.4.79
Knottenbelt DC.Twenty-four foals were confirmed to be infected with Rhodococcus equi on a private stud in Zimbabwe over a two-year period. Six mares had foals which were affected in each of the two years. All the foals were febrile and early cases were detected by this pyrexia. Bronchopneumonia was only clinically detectable in advanced cases. In spite of energetic hygiene measures relating to pasture and housing management, the incidence was higher in the second year (23 per cent of foals born) than in the first (15 per cent of foals born). The mean age of the foals was significantly greater in the second y...
Current status of the diagnosis and control of African horse sickness.
Veterinary research    January 1, 1993   Volume 24, Issue 2 189-197 
Rodriguez M, Hooghuis H, Castaño M.African horse sickness (AHS) is an infectious, non-contagious, highly fatal viral disease of Equidae, transmitted by arthropod vectors of the genus Culicoides, and endemic in Africa south and east of the Sahara. The disease is caused by a virus of the Reoviridae family, genus Orbivirus, and 9 serotypes have been recognized. Recent outbreaks of AHS in the Iberian peninsula and Northern Africa emphasize the need for accurate diagnosis and rapid implementation of control measures. In this paper, the epizootiological factors, clinical signs and necropsy findings of AHS are discussed, and an update...
Did vaccinia virus come from a horse?
Equine veterinary journal    January 1, 1993   Volume 25, Issue 1 8-10 doi: 10.1111/j.2042-3306.1993.tb02892.x
Taylor CE.No abstract available
Epizootic of equine influenza in 1969 in Poland.
Archivum veterinarium Polonicum    January 1, 1993   Volume 33, Issue 3-4 139-145 
Kita J.Epidemiological observations on the course and spread of equine influenza in Poland during the 1969 epizootic were carried out. The dynamics of the spread of the disease in the country are shown graphically by voivodship. The disease incidence and mortality rates for the entire country are also shown. The highest incidence of disease and mortality rates were found to be in November 1969. A/equi-2/Warsaw/69 was identified as the causal virus. It caused the largest epizootic in the country since 1954.
African horse sickness: transmission and epidemiology.
Veterinary research    January 1, 1993   Volume 24, Issue 2 199-212 
Mellor PS.African horse sickness (AHS) virus causes a non-contagious, infectious, arthropod-borne disease of equines and occasionally of dogs. The virus is widely distributed across sub-Saharan African where it is transmitted between susceptible vertebrate hosts by the vectors. These are usually considered to be species of Culicoides biting midges but mosquitoes and/or ticks may also be involved to a greater or lesser extent. Periodically the virus makes excursions beyond its sub-Saharan enzootic zones but until recently does not appear to have been able to maintain itself outside these areas for more t...
WHO/OIE meeting: consultation on newly emerging strains of equine influenza. 18-19 May 1992, Animal Health Trust, Newmarket, Suffolk, UK.
Vaccine    January 1, 1993   Volume 11, Issue 11 1172-1175 doi: 10.1016/0264-410x(93)90092-c
Mumford J, Wood J.No abstract available
Eastern equine encephalitis virus in Ohio during 1991.
Journal of medical entomology    January 1, 1993   Volume 30, Issue 1 217-222 doi: 10.1093/jmedent/30.1.217
Nasci RS, Berry RL, Restifo RA, Parsons MA, Smith GC, Martin DA.During August and September of 1991, an epizootic of eastern equine encephalitis (EEE) virus in horses occurred in Wayne and Holmes countries, OH. This was the first recorded epizootic of EEE virus in the state. Twelve horses were confirmed positive for EEE virus through virus isolation or seroconversion, and seven additional horses with compatible symptoms were in close spatial and temporal proximity to the confirmed cases and were presumed to have died from EEE virus. The outbreak was centered around the Killbuck Wildlife Area, a 2,147-ha tract maintained by the state, half of which consists...
[Venezuelan equine encephalitis. Determination of antibodies in the human population of Municipio Mirand, Estado Zulia, Venezuela].
Investigacion clinica    January 1, 1993   Volume 34, Issue 3 135-141 
Ryder S, Núñez-Camargo J, Rangel P, Añez F.With the purpose of determining antibodies prevalence against Venezuelan Equine Encephalitis Virus in the population of Puertos de Altagracia and Sabaneta de Palmas of Miranda county, Zulia State, Venezuela, 199 subjects were studied: 57 from Puertos de Altagracia and 142 from Sabaneta de Palmas. They were classified in older (42.78%) and younger (57.2%) than 15 years. The blood specimens were processed for Hemagglutination Inhibition Test using EEV antigen Goajira strain at pH 6.5. We found that all 57 specimens from Puertos de Altagracia were negative, whereas of 142 specimens from Sabaneta ...
Ocular manifestations of systemic diseases.
The Veterinary clinics of North America. Equine practice    December 1, 1992   Volume 8, Issue 3 627-636 doi: 10.1016/s0749-0739(17)30445-5
Lavach JD.The diseases included in this article constitute a wide range of maladies that affect the horse. Certainly, the diseases that are known today to produce ocular lesions are just a few of what will be discovered if attending veterinarians always examine the eyes of patients with systemic diseases.
Molecular evidence for the origin of the widespread Venezuelan equine encephalitis epizootic of 1969 to 1972.
The Journal of general virology    December 1, 1992   Volume 73 ( Pt 12) 3301-3305 doi: 10.1099/0022-1317-73-12-3301
Kinney RM, Tsuchiya KR, Sneider JM, Trent DW.Venezuelan equine encephalitis (VEE) virus is a mosquito-borne pathogen that has caused encephalitis in equine species and humans during sporadic outbreaks in the western hemisphere. The last, and most widespread, VEE outbreak occurred in South America, Central America, Mexico and the U.S.A. (Texas) during 1969 to 1972. We have cloned and sequenced the genome of a virulent VEE subtype I-AB virus, strain 71-180, isolated in Texas in 1971. Thirty-four nucleotide differences were detected between the genome of 71-180 virus and that of the subtype I-AB Trinidad donkey (TRD) virus isolated during t...
Halicephalobus deletrix infection in a horse in Scotland.
The Veterinary record    November 21, 1992   Volume 131, Issue 21 495 doi: 10.1136/vr.131.21.495-a
Angus KW, Roberts L, Archibald DR, Fraser DG, Jackson F, Gibbons LM.No abstract available
Secondary bacterial infections following an outbreak of equine influenza.
The Veterinary record    November 7, 1992   Volume 131, Issue 19 441-442 doi: 10.1136/vr.131.19.441
Sarasola P, Taylor DJ, Love S, McKellar QA.No abstract available
Cytogenetic monitoring of farm animals under conditions of environmental pollution.
Mutation research    November 1, 1992   Volume 283, Issue 3 199-210 doi: 10.1016/0165-7992(92)90108-t
Rubes J, Borkovec L, Horínová Z, Urbanová J, Proroková I, Kulíková L.Cytogenetic examinations were carried out in 13 cattle farms, two herds of horses, one stag farm and 13 pig farms in areas with different levels of environmental contamination. The frequency of aberrant cells per 100 mitoses was 3.67 +/- 1.89 in pigs (n = 260) and 4.16 +/- 2.4 in herbivores (n = 497). This is a significant difference (p < 0.01). Ten times higher frequencies of chromatid exchanges were found in pigs. The examined herds were classified into three groups by the level of environmental contamination (satisfactory, impaired and severely impaired environment). Significant differen...
Phylogenetic analysis of alphaviruses in the Venezuelan equine encephalitis complex and identification of the source of epizootic viruses.
Virology    November 1, 1992   Volume 191, Issue 1 282-290 doi: 10.1016/0042-6822(92)90190-z
Weaver SC, Bellew LA, Rico-Hesse R.We studied the evolution of alphaviruses in the Venezuelan equine encephalitis (VEE) complex using phylogenetic analysis of RNA nucleotide sequences from limited portions of the nsP4, E1, and 3' untranslated genome regions of representative strains. The VEE complex constituted a monophyletic group of viruses (descended from a common ancestor); some serologic VEE varieties such as subtype III formed monophyletic groups while subtype I did not. Subtype II Everglades and variety ID enzootic viruses formed a monophyletic group which also included all epizootic variety IAB and IC VEE isolates. Ever...
African horse sickness in Spain.
Veterinary microbiology    November 1, 1992   Volume 33, Issue 1-4 129-142 doi: 10.1016/0378-1135(92)90041-q
Rodriguez M, Hooghuis H, Castaño M.The aetiology, pathogenesis and epizootiology of African horse sickness (AHS) are reviewed with special reference to recent outbreaks in the Iberian peninsula. AHS is a highly fatal insect-borne viral disease of Equidae. It is caused by an Orbivirus (family Reoviridae) and nine serotypes are recognised. Outbreaks occurred in central Spain in 1987 and in southern regions of the Iberian peninsula in 1988, 1989 and 1990. All were associated with serotype 4 of the virus, whereas other occurrences of AHS outside Africa have all been caused by serotype 9. The clinical picture in the outbreaks was ma...
Cross-reactivity of existing equine influenza vaccines with a new strain of equine influenza virus from China.
The Veterinary record    October 24, 1992   Volume 131, Issue 17 388-391 doi: 10.1136/vr.131.17.388
Chambers TM.A novel strain of equine influenza virus, influenza A/equine/Jilin (China)/1/89, has emerged which is genetically distinct from all earlier strains of equine influenza. It is therefore possible that the vaccines against equine influenza may be unable to protect horses against disease caused by this virus strain. In vitro serological assays established that there were low levels of immunological cross-reactivity between the new virus, the current vaccine strains and the strains of equine-2 influenza virus now in circulation.
Vectors of African horse sickness in the Cape Verde Islands.
The Veterinary record    July 18, 1992   Volume 131, Issue 3 56 doi: 10.1136/vr.131.3.56
Boorman J, van Harten A.No abstract available
Equine fatigue syndrome.
The Veterinary record    July 18, 1992   Volume 131, Issue 3 58-59 doi: 10.1136/vr.131.3.58-a
Ricketts SW, Young A, Mowbray JF, Yousef GE, Wood J.No abstract available
Pneumocystis carinii infection in foals in the UK.
The Veterinary record    July 4, 1992   Volume 131, Issue 1 19 doi: 10.1136/vr.131.1.19-a
Whitwell K.No abstract available
Role of the embryonic vesicle and progesterone in embryonic loss in mares.
Journal of reproduction and fertility    July 1, 1992   Volume 95, Issue 2 339-347 doi: 10.1530/jrf.0.0950339
Bergfelt DR, Woods JA, Ginther OJ.Characteristics of spontaneous embryonic loss in 21 mares were compared with those of 52 contemporary mares that maintained pregnancy. Embryonic losses were, in retrospect, grouped according to day of loss and length of the interovulatory interval, respectively, as follows: group 1, less than or equal to day 20 and less than or equal to 30 days (n = 10); group 2, less than or equal to day 20 and greater than 30 days (n = 3); and group 3, greater than day 20 and greater than 30 days (n = 8); ovulation was day 0. Mean diameter of the embryonic vesicle in group 1 was smaller (P less than 0.05) on...
Venezuelan equine encephalomyelitis and African horse sickness. Current status and review.
Annals of the New York Academy of Sciences    June 16, 1992   Volume 653 217-227 doi: 10.1111/j.1749-6632.1992.tb19650.x
Walton TE, Holbrook FR, Bolivar-Raya R, Ferrer-Romero J, Ortega MD.The arthropod-borne virus (arbovirus) diseases of livestock have worldwide impact. The prevention of an introduction of an exotic disease and the control of one subsequent to an introduction will require the attention, cooperation, and support of the livestock industry, regulatory agencies, and researchers. The most effective protection of our livestock industries is to prevent the introduction of an exotic disease agent. This implies complete restriction of animal imports and exports. However, "zero risk" is an unacceptable option in today's world of internationally integrated and interdepend...
[Three cases of virus isolation from horse fetuses diagnosed with equine arteritis virus (EAV) abortion from stud farms with different breeds].
Berliner und Munchener tierarztliche Wochenschrift    June 1, 1992   Volume 105, Issue 6 181-187 
Nowotny N, Bürki F.Three cases of abortions were diagnosed as caused by Equine Arteritis Virus (EAV) by isolation and typing of this virus from the respective fetuses. All 3 abortions were single cases, one occurring on a stud with Iceland Ponies, one with Warmbloods, one with Lipizzaner horses. On each stud horses of the respective breed were kept exclusively, therefore there existed no epidemiologic link. By means of seroneutralization tests performed on in contact horses it could be shown, that EAV had only been introduced recently into the stud with the Iceland Ponies. An extraneous mare stabled temporarily ...
Failure of passive transfer in foals.
Journal of veterinary internal medicine    May 1, 1992   Volume 6, Issue 3 197-198 doi: 10.1111/j.1939-1676.1992.tb00337.x
Baldwin JL.No abstract available
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