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Topic:Lameness

Lameness in horses is a condition characterized by an abnormal gait or stance due to pain or mechanical dysfunction affecting the limbs or spine. It is a common issue in equine practice and can result from a variety of causes, including injury, infection, or degenerative diseases. Lameness can affect a horse's performance and quality of life, making accurate diagnosis and management important for equine health. Diagnostic techniques often include physical examination, imaging, and gait analysis. This page aggregates peer-reviewed research studies and scholarly articles that explore the etiology, diagnostic methods, and management strategies for lameness in horses.
Correlation of dermal-epidermal laminar lesions of equine hoof with various disease conditions.
Veterinary pathology    November 1, 1980   Volume 17, Issue 6 656 doi: 10.1177/030098588001700601
Roberts ED, Ochoa R, Haynes PF.Dermal and epidermal laminar lesions were correlated with acute intestinal, primary hepatic, septicemic, chronic laminar, and acute laminar diseases. Horses with acute intestinal disease had edema in the secondary dermal laminae. Those with hepatic disease had increased keratinization of the secondary epidermal laminae. Septicemia caused increased keratin formation in the primary and secondary epidermal laminae. Chronic laminitis caused architectural changes of the epidermal laminae characterized by hyperplasia and keratin formation of the basal epidermal layer. Horses with acute laminitis had...
[Lameness in the horse (author’s transl)].
Tijdschrift voor diergeneeskunde    October 15, 1980   Volume 105, Issue 20 863-866 
Dik KJ.No abstract available
Bicipital bursitis in a mare.
The Veterinary record    October 4, 1980   Volume 107, Issue 14 330-331 doi: 10.1136/vr.107.14.330
Mason TA.A case of bicipital bursitis in a six-year-old mare is described. The horse was presented with acute lameness, pyrexia and hot swelling over the right shoulder region. Although antibiotic treatment relieved these signs, a firm cold swelling remained which had to be removed by surgical resection and drainage.
Tripartite navicular bone and navicular disease in a horse.
Journal of the American Veterinary Medical Association    October 1, 1980   Volume 177, Issue 7 644-646 
Feeney DA, Booth LC, Johnston GR.No abstract available
Disorders of the thoracolumbar spine of the horse–a survey of 443 cases.
Equine veterinary journal    October 1, 1980   Volume 12, Issue 4 197-210 doi: 10.1111/j.2042-3306.1980.tb03427.x
Jeffcott LB.The survey comprised 443 horses, referred to the clinical department of the Equine Research Station, with a history of a thoracolumbar (TL) complaint. A wide range of lesions were capable of producing back problems and more than one condition or site of injury was found in 75 animals (17 per cent). There were 103 horses (19.7 per cent) with no evidence of damage to the TL spine or its associated structures. In 66 of these cases, clinical signs were attributed to a variety of hindlimb lamenesses and, in 37, no specific diagnosis could be made. Soft tissue injury was diagnosed in 203 cases (38.8...
Repair of digital flexor tendon lacerations in the horse, using carbon fiber implants.
Journal of the American Veterinary Medical Association    September 1, 1980   Volume 177, Issue 5 427-435 
Valdez H, Clark RG, Hanselka DV.Flexible medical grade carbon fiber was surgically implanted in tenectomized or lacerated superficial and deep digital flexor tendons of 13 horses (7 clinical cases and 6 experimental), ranging in age from 15 months to 10 years and weighing 300 to 500 kg. The 6 experimental horses were euthanatized at 30-, 45-, 60-, and 90-day intervals for gross and histologic evaluation of the results. Three of the experimental horses served as their own controls. Of the 7 clinically affected horses, 3 were euthanatized because they developed laminitis or the wound failed to heal. The remaining 4 horses in t...
Drug-induced laminitis in a colt.
Modern veterinary practice    July 1, 1980   Volume 61, Issue 7 608-610 
Lose MP.No abstract available
Clinical approach to determine the contribution of the palmar and palmar metacarpal nerves to the innervation of the equine fetlock joint.
American journal of veterinary research    June 1, 1980   Volume 41, Issue 6 940-943 
Gray BW, Engel HN, Rumph PF, LaFaver J, Brown BG, McKibbin JS.To determine the sensory nerve supply of the metacarpophalangeal joint, lameness was induced in eight horses by injecting the joint with a glycerin suspension of glass micropheres. When the medial and lateral palmar nerves were anesthetized in 4 horses, there was noticeable improvement in the gait, but each horse remained lame. When the medial and lateral palmar metacarpal nerves were also anesthetized, 3 of the 4 horses became sound. To confirm the results of local anesthesia, neurectomies were performed on a second group of four horses. The lameness was alleviated only upon resection of both...
Induced juglone toxicosis in ponies and horses.
American journal of veterinary research    June 1, 1980   Volume 41, Issue 6 944-945 
True RG, Lowe JE.Juglone, a toxic compound found in all parts of plants of the walnut tree family Jugans, was evaluated as the possible toxin involved in black walnut shaving-associated laminitis in the horse. Large amounts (up to 1 g) of this chemical administered per os inconsistently caused mild signs of laminitis in ponies. Topical application of juglone to the digits of horses caused local skin irritation but did not cause laminitis. Intravenous administration of juglone caused acute pulmonary edema in some individuals previously exposed to the compound per os or IV.
Avulsion fractures of the origin of the suspensory ligament of the horse.
Journal of the American Veterinary Medical Association    May 15, 1980   Volume 176, Issue 10 Pt 1 1004-1010 
Bramlage LR, Gabel AA, Hackett RP.Five horses with avulsion fractures of the proximal origin of the suspensory ligament were examined for lameness. The horses in the series each had lameness of acute onset; four were severely lame, and one was moderately lame. The condition was difficult to diagnose because commonly used local anesthetic blocks did not result in improvement during the routine examination. Local infiltration of the area with local anesthetic returned the horses to soundness. After a period of rest, varying from a few weeks in the acute injury to 6 months in the chronic injuries, the horses were returned to raci...
Update on equine laminitis.
The Veterinary clinics of North America. Large animal practice    May 1, 1980   Volume 2, Issue 1 25-32 doi: 10.1016/s0196-9846(17)30173-8
Garner HE.No abstract available
Corrective shoeing.
The Veterinary clinics of North America. Large animal practice    May 1, 1980   Volume 2, Issue 1 3-24 doi: 10.1016/s0196-9846(17)30172-6
Moyer W.No abstract available
Lameness caused by inflammation in the distal hock.
The Veterinary clinics of North America. Large animal practice    May 1, 1980   Volume 2, Issue 1 101-124 doi: 10.1016/s0196-9846(17)30177-5
Gabel AA.Since horses ridden at a gallop have a high incidence of bone spavin, and harness horses (Standardbred pacers and trotters) have a different syndrome, they will be discussed separately. Osteoarthritis (bone spavin) apparently is caused by impact and compression on the distal tarsal bones, which occurs most forcefully at the gallop (canter). Shear stresses, which occur in the joint between these bones, are most severe at the trot or pace, and probably cause the usual cunean-tarsitis problem in Standardbreds. Obviously some of each force occurs at each gait.
Diseases of the carpus.
The Veterinary clinics of North America. Large animal practice    May 1, 1980   Volume 2, Issue 1 81-99 doi: 10.1016/s0196-9846(17)30176-3
Auer J.Degenerative joint disease is the most frequently diagnosed problem of the carpus. Horses afflicted with this disease should not be injected with corticosteroids, but should be rested for several months. Swimming exercise and intra-articular injections of hyaluronic acid during this time help to restore the integrity of the involved joints. Chip fractures of the various carpal bones and the distal radius are commonly diagnosed as the cause of the lameness. Not all horses with carpal fractures are candidates for surgery. It is important that the severity of the problem be realized and that the ...
Hock lameness associated with degeneration of the talocalcaneal articulation: report of two cases in horses.
Veterinary medicine, small animal clinician : VM, SAC    April 1, 1980   Volume 75, Issue 4 678-681 
White NA, Turner TA.No abstract available
Effect of hyaluronic acid in naturally occurring and experimentally induced osteoarthritis.
American journal of veterinary research    April 1, 1980   Volume 41, Issue 4 568-574 
Auer JA, Fackelman GE, Gingerich DA, Fetter AW.Effect of intraarticular injections of hyaluronic acid was evaluated in two groups of horses, using force plates to assess quantitatively the degree of lameness observed. In six horses, chip fractures were created surgically on the dorsomedial aspects of both radial carpal bones. After the horses were trained for 30 days, 40 mg of hyaluronic acid was injected intraarticularly into the limb showing the greater degree of lameness. Horses were worked for 2 more weeks and then were evaluated. A highly significant (P less than 0.01) increase in weight bearing on the treated limb was observed. Ten h...
[Lameness in the horse (author’s transl)].
Tijdschrift voor diergeneeskunde    February 1, 1980   Volume 105, Issue 3 90-95 
Dik KJ.No abstract available
[Complete traumatic exungulation with open fracture of both hoof bone branches in a horse].
Tierarztliche Praxis    January 1, 1980   Volume 8, Issue 4 481-488 
Stanek C, Brkic A.No abstract available
Diagnosis, relative incidence, and probable cause of cunean tendon bursitis-tarsitis of Standardbred horses.
Journal of the American Veterinary Medical Association    November 15, 1979   Volume 175, Issue 10 1079-1085 
Gabel AA.A prospective study of the diagnostic results on 25 previously untreated, slightly lame Standardbred horses showed that manipulative tests are of some help in diagnosis. The lameness of each horse was diminished or the horse went lame on the opposite limb after being given an injection of anesthetic in the cunean bursa, and lameness improved more when local anesthetic was injected in the distal intertarsal and tarsometatarsal articulations. In four horses, lesions of the distal articulation of the hock were evident on radiography.
Spontaneous fracture of the navicular bone in the horse.
Nordisk veterinaermedicin    October 1, 1979   Volume 31, Issue 10 429-435 
Arnbjerg J.After a short review of previous literature about fractures of the navicular bone in horses, the symptoms and the pathological-anatomical changes of the condition are described. The most important clinical symptom is acute severe lameness without significant swellings, but with pronounced pain reaction to rotation of the coffin joint. After rest the lameness is reduced considerably, but without treatment it can persist for several years. Adherences between the deep flexor tendon and the site of the fracture and eventual damage to the coffin joint are considered to be the cause of the persistin...
Spavin.
Modern veterinary practice    September 1, 1979   Volume 60, Issue 9 724-725 
Rooney JR.No abstract available
A possible case of osteodystrophia fibrosa cystica in a horse.
The Canadian veterinary journal = La revue veterinaire canadienne    September 1, 1979   Volume 20, Issue 9 242-243 
Menard L, Marcoux M, Halle G.A case of unilateral front leg lameness with bone changes in the pedal bone suggestive of osteodystrophia fibrosa cystica is reported in an eight year old crossbred gelding. The changes were diagnosed at postmortem examination. The known possible causes are briefly reviewed.
A preliminary report on the use of warfarin in the treatment of navicular disease.
Equine veterinary journal    July 1, 1979   Volume 11, Issue 3 187-190 doi: 10.1111/j.2042-3306.1979.tb01338.x
Colles CM.Twenty horses suffering from navicular disease were treated with warfarin given orally. The dosage was to effect, to give a 2 to 4 second prolongation of the one stage prothrombin time (OSPT). Dosage was initially at a rate of 0.018 mg/kg, changing the dose by amounts of 20 per cent until the required dose was achieved. Final dose rates varied from 0.012 mg/kg to 0.75 mg/kg. All the cases treated received warfarin daily throughout the trial. Seventeen of the animals became sound and the remaining 3 showed a marked improvement in their gait. The mean of the ages of the horses was 7.5 years, the...
Chronic laminitis in a horse.
The Indian veterinary journal    April 1, 1979   Volume 56, Issue 4 329-330 
Nigam JM, Krishnamurthy D, Shetty BR.No abstract available
Corticosteroid-potentiated vascular responses of the equine digit: a possible pharmacologic basis for laminitis.
American journal of veterinary research    January 1, 1979   Volume 40, Issue 1 135-138 
Eyre P, Elmes PJ, Strickland S.Spirally cut digital arteries and veins were mounted isotonically in organ baths containing oxygenated Krebs' Q-Henseleit solution. Twelve arterial and 12 venous preparations all contracted dose dependently when epinephrine, norepinephrine, serotonin, or histamine were added to the bathing fluid. Addition of hydrocortisone or betamethasone alone did not cause contractions in any of the tissues tested. However, when hydrocortisone or betamethasone was added to vessel strips that were partially contracted (40% to 60% maximal) by epinephrine, norepinephrine, or serotonin, each vessel strip invari...
The use of phenylbutazone in the horse.
The Veterinary record    December 23, 1978   Volume 103, Issue 26-27 571 doi: 10.1136/vr.103.26-27.571
No abstract available
Changes in the caecal flora associated with the onset of laminitis.
Equine veterinary journal    October 1, 1978   Volume 10, Issue 4 249-252 doi: 10.1111/j.2042-3306.1978.tb02273.x
Garner HE, Moore JN, Johnson JH, Clark L, Amend JF, Tritschler LG, Coffmann JR, Sprouse RF, Hutcheson DP, Salem CA.Caecal fluid samples collected 8 and 24 hours after carbohydrate overload were quantitatively compared to control samples in terms of aerobic and anaerobic bacteria. Concomitant increases in lactic acid-producing bacteria and decreases in Gram negative bacteria were substantiated during the onset of acute laminitis. Progressive decreases in caecal fluid pH were also quantitated. Although endotoxin assays of caecal fluid and blood were not done, the caecal flora changes suggest its presence during the onset of acute laminitis.
Surgical approach for medial meniscectomy in the horse.
Journal of the American Veterinary Medical Association    September 15, 1978   Volume 173, Issue 6 766-769 
Valdez H, Adams OR.After preliminary studies on cadavers, a surgical approach was developed for removal of the medial meniscus, offering maximal exposure and minimal trauma to the articular surfaces of the medial tibial plateau and femoral condyle. Medial meniscectomy was then performed on 10 clinically normal horses that had been given general anesthesia. Nine horses were euthanatized at intervals of 1 to 6 months, and the joints were evaluated grossly and histologically. (One horse will be examined 1 year after surgery). Medial meniscus tears were diagnosed in 5 horses; medial meniscectomy was done on 4 horses...
Bucked shin.
Modern veterinary practice    August 1, 1978   Volume 59, Issue 8 633-634 
Rooney JR.No abstract available
Shoulder and hip lameness in horses.
Modern veterinary practice    July 1, 1978   Volume 59, Issue 7 565-568 
No abstract available
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