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Topic:Nervous System

The nervous system in horses is a complex network responsible for transmitting signals between different parts of the body. It consists of the central nervous system (CNS), which includes the brain and spinal cord, and the peripheral nervous system (PNS), which comprises nerves that extend throughout the body. The nervous system regulates various physiological processes, including movement, sensation, and the coordination of bodily functions. It plays a critical role in processing sensory information and initiating appropriate responses. This page compiles peer-reviewed research studies and scholarly articles that explore the structure, function, and clinical implications of the equine nervous system, including its role in behavior, response to stimuli, and neurological disorders.
Spontaneous craniofacial malformations and central nervous system defects in an aborted equine foetus.
Journal of comparative pathology    January 1, 1985   Volume 95, Issue 1 131-135 doi: 10.1016/0021-9975(85)90086-6
Bunton TE.Developmental defects are rarely reported in the horse. Severe craniofacial and central nervous system defects in an equine foetus are described and their possible causation and pathogenesis are suggested.
Coprophagy by foals: effect of age and possible functions.
Equine veterinary journal    January 1, 1985   Volume 17, Issue 1 17-19 doi: 10.1111/j.2042-3306.1985.tb02030.x
Crowell-Davis SL, Houpt KA.In colts and fillies observed from birth to 24 weeks old, coprophagy occurred from Weeks 1 to 19. Its frequency was greatest during the first two months. Coprophagy was rarely observed in mares and stallions. Foals usually ate the faeces of their mother but were observed to eat their own and those of a stallion and another unrelated mare. Urination by the foal occurred before, during or after 26 per cent of the coprophagy incidents. It is hypothesised that foals may consume faeces in response to a maternal pheromone which signals the presence of deoxycholic acid or other acids which the foal m...
Sensory nerve endings in the peroneus tertius muscle of the ass and horse: a functional hypothesis. Palmieri G, Panu R, Asole A, Farina V, Sanna L.The various types of sensory nerve endings found in the peroneus tertius muscle in the ass and in the horse have been studied with Ruffini's gold chloride method. Free nerve endings have been described as well as encapsulated receptors. These corpuscles are classified as Pacini-like, Ruffini's terminations and also Golgi's tendon-organs. The authors have pointed out the morphology, topography and structural characteristics of the above named nervous terminations and have hypothesized that a probable functional relation existed between these nervous corpuscles and the considered tendinous struc...
Distribution of substance P-like immunoreactivity in the enteric neurons of the large colon of normal and amitraz-treated ponies: an immunocytochemical study.
Equine veterinary journal    January 1, 1985   Volume 17, Issue 1 23-29 doi: 10.1111/j.2042-3306.1985.tb02032.x
Cummings JF, Sellers AF, Lowe JE.The distribution of the putative motor excitatory neurotransmitter, substance P, was studied immunocytochemically in the left dorsal colon of four normal control ponies and three ponies with amitraz-induced impaction colic. Substance P-like immunoreactivity in the control ponies was observed in nerve fibres in all layers of the bowel wall and in the nerve cell bodies of the enteric ganglia. The substance P-like immunoreactivity was clearly more intense in the cell bodies of submucosal ganglia than in those of the myenteric ganglia. The internodal nerve strands of the myenteric plexus were very...
[Neurological disorders caused by equine herpesvirus type 1 and cauda equina neuritis in horses].
Tijdschrift voor diergeneeskunde    December 15, 1984   Volume 109, Issue 24 1027-1035 
Sloet van Oldruitenborgh-Oosterbaan MM, Binkhorst GJ.The differences in aetiology, symptomatology, pathomorphology, diagnosis and therapy between the nervous form (paralytic form) of Equine Herpes Virus, type 1, and Neuritis Caudae Equinae are reviewed. The conclusion is that in most cases it is possible to differentiate between these two clinical syndromes.
Equine adenovirus 1 isolated from cauda equina neuritis.
Research in veterinary science    September 1, 1984   Volume 37, Issue 2 252-254 
Edington N, Wright JA, Patel JR, Edwards GB, Griffiths L.Equine adenovirus 1 was recovered after four to six passages from two out of three cases of cauda equina neuritis (CEN) using kidney monolayers. Similar treatment of lumbo-sacral spinal cord from six normal horses did not yield adenovirus. All three cases of CEN had antibodies to the neuritogenic myelin protein P2 while immunofluorescence demonstrated that autologous IgG bound to the myelin of affected nerves. Adenovirus was not detected in neural tissue by immunofluorescence.
Amikacin sulfate in mares: pharmacokinetics and body fluid and endometrial concentrations after repeated intramuscular administration.
American journal of veterinary research    August 1, 1984   Volume 45, Issue 8 1610-1613 
Brown MP, Embertson RM, Gronwall RR, Beal C, Mayhew IG, Curry SH.Six mares were given 5 IM injections (at 12-hour intervals between doses) of amikacin sulfate at a dosage of 7 mg/kg of body weight. Serum amikacin concentrations were measured serially throughout the study; synovial, peritoneal, endometrial, and urine concentrations were determined after the last injection. Amikacin concentrations of the CSF were measured serially in 3 of the 6 mares; 1 of the 3 mares had septic meningitis. Mean serum amikacin concentrations peaked at 1 to 2 hours after IM injection. The highest mean serum concentration was 19.2 micrograms/ml (1.5 hours after the 5th injectio...
Neurological examination of newborn foals.
Equine veterinary journal    July 1, 1984   Volume 16, Issue 4 306-312 doi: 10.1111/j.2042-3306.1984.tb01933.x
Adams R, Mayhew IG.Behaviour and response to neurological testing of apparently healthy newborn foals differed significantly from the adult in several ways. Foals responded to external stimulation with exaggerated movements although they tended to sink into a relaxed state when restrained. They had a more angular head position and assumed a base wide stance. The menace reflex developed during the first two weeks post partum. In general, the foals' gait was choppy or dysmetric. In lateral recumbency the foals had increased extensor tone, hyperreflexive tendon reflexes, crossed extensor reflexes as well as recumbe...
Cauda equina neuritis: a chronic idiopathic polyneuritis in two horses.
The Canadian veterinary journal = La revue veterinaire canadienne    May 1, 1984   Volume 25, Issue 5 214-218 
Rousseaux CG, Futcher KG, Clark EG, Naylor JM.Two cases of cauda equina neuritis are compared and contrasted. Neurological deficits of the tail and perineum were noted and functional deficits were seen in gait, urination, defecation and cranial nerve function. Lesions consisted of nonsuppurative inflammation of the nerve trunks and proliferation of the perineurium of the cauda equina. Cranial nerve involvement in one case supported a diagnosis of polyneuritis equi rather than cauda equina neuritis. The possible etiologies and pathogenesis of this disease are discussed.
Pain perception and alleviation in animals.
Federation proceedings    April 1, 1984   Volume 43, Issue 5 1307-1312 
Erickson HH, Kitchell RL.In the last 2 decades there have been substantial advances in our knowledge of the scientific basis of the mechanisms of pain. Nociceptors or pain receptors are widespread in the skin and tissues of animals; chemical mediation of nociceptor excitation may provide a key for understanding the peripheral phenomena related to pain. The expression of pain in animals involves multiple ascending and descending branches, as well as specialized pain-signaling mechanisms in the spinal cord. The importance of these different pathways varies with species and circumstances. Endogenous neural systems in the...
Neuromuscular arthrogryposis multiplex congenita in a thoroughbred foal.
Veterinary pathology    March 1, 1984   Volume 21, Issue 2 187-192 doi: 10.1177/030098588402100210
Mayhew IG.Arthrogryposis multiplex congenita was studied in a newborn thoroughbred foal. The syndrome affected only the left hind limb allowing the right hind limb to serve as a reference. There was a significant depletion of large motor neurons from the ventral horn of the spinal cord from L3 to S4 on the affected side. Hypoplasia of nerves, muscles, and bones was present in the affected limb. Histologically, hypoplasia and degeneration of myofibers and nerve bundles were seen. No cause of the syndrome, which corresponds to most human cases, was determined. Neuromuscular arthrogryposis was diagnosed be...
[Medial motor nucleus in the lumbosacral segment of the spinal cord of the horse].
Polskie archiwum weterynaryjne    January 1, 1984   Volume 24, Issue 1 133-137 
Sławomirski J, Flieger S, Jastrzebski M, Boratyński Z.The studies carried out on 2 spinal cords of horses showed that cells of the medial motor nucleus (nucleus motorius medialis) are present in all neuromers of the lumbar and sacral segment of the spinal cord. It lies in the medial part of grey matter of the ventral column, neighbouring laterally and ventrally with cells of the lateral motor nucleus, whereas dorsally with cells of the nucleus of the ventral commissural horn. Along the nucleus numerous constrictions and intervals are found, which are connected with various numbers of nerve cells in particular cross-sections.
[Nucleus motorius lateralis in the lumbosacral segment of the spinal cord in horses].
Polskie archiwum weterynaryjne    January 1, 1984   Volume 24, Issue 1 125-131 
Flieger S, Sławomirski J, Boratyński Z, Jastrzebski M.Two medullae oblongatae of horses were cut into 15 microns cross-sections and stained according to the modified method of Nissel. The lateral motor nucleus lies in the lateral and median part of the ventral column of spinal cord grey matter. It adjoins medially nucleus motorius medialis of the spinal ventral column. Cells of this nucleus occur both along the whole lumbar and sacral segment of the spinal cord. In the lateral motor nucleus three cell groups are distinguished-median, basal and lateral. The latter is divided in some segments into subgroups-dorsal and ventral. Along the nucleus qui...
Myelopathy and vitamin E deficiency in six Mongolian wild horses.
Journal of the American Veterinary Medical Association    December 1, 1983   Volume 183, Issue 11 1266-1268 
Liu SK, Dolensek EP, Adams CR, Tappe JP.Degenerative myelopathy was diagnosed in six Mongolian wild horses. Three of the horses had a history of ataxia dating from birth to 3 months of age. The clinical signs were uncoordinated movement of the hindlimbs and an abnormally wide-based gait and stance. The other 3 horses had mild ataxia. There were no gross lesions in the brain, vertebrae, or spinal cord. Histologic examination revealed degeneration of the neural processes in the ventral and lateral funiculi of all 6 horses. Myelin sheaths were dilated and vacuolated, and there were swollen, fragmented, or lysed axons. Neuronal degenera...
Facial and vestibulocochlear nerve disease in six horses.
Journal of the American Veterinary Medical Association    November 15, 1983   Volume 183, Issue 10 1076-1080 
Power HT, Watrous BJ, de Lahunta A.In 6 horses, clinical signs of illness implicated a lesion involving the facial and vestibulocochlear nerves. One horse had signs of otitis externa. Five horses had radiographic changes primarily involving periosteal bony proliferation of the stylohyoid bone at its articulation with the temporal bone. Five horses improved with antibiotic therapy. Otitis media-interna was found at necropsy of one horse.
Cytology of equine cerebrospinal fluid.
Veterinary pathology    September 1, 1983   Volume 20, Issue 5 553-562 doi: 10.1177/030098588302000507
Beech J.The cytology of cerebrospinal fluid samples from horses is described. The samples were obtained from 24 normal horses, 35 horses with axonal degeneration and/or spinal cord compression, 29 horses with encephalomyelitis, 14 horses with other lesions of the nervous system, and eight horses with signs of neurologic dysfunction of undetermined origin. (Three of the latter were suspected botulinum intoxications.) Fluid was aspirated from the atlanto-occipital space following general anesthesia or immediately after a lethal dose of barbiturate. In two horses, fluid also was aspirated from the lumbos...
Clinicopathologic features of brain herniation in animals.
Journal of the American Veterinary Medical Association    May 15, 1983   Volume 182, Issue 10 1111-1116 
Kornegay JN, Oliver JE, Gorgacz EJ.No abstract available
Pineoblastoma, a primitive neuroectodermal tumor in the brain of a horse.
Veterinary pathology    September 1, 1982   Volume 19, Issue 5 567-569 doi: 10.1177/030098588201900516
Holshuh HJ, Howard EB.No abstract available
Teratological effects of western equine encephalitis virus on the fetal nervous system of Macaca mulatta.
Teratology    February 1, 1982   Volume 25, Issue 1 71-79 doi: 10.1002/tera.1420250110
London WT, Levitt NH, Altshuler G, Curfman BL, Kent SG, Palmer AE, Sever JL, Houff SA.Fetal rhesus monkeys were inoculated intracerebrally with an attenuated strain of western equine encephalitis virus. All animals developed microcephaly. Twelve of sixteen monkeys developed ex vacuo hydrocephalus. All virus inoculated fetuses developed WEE virus antibody. Virus could not be recovered at the time of delivery. Monkeys with the highest WEE antibody titers showed the greatest degree of hydrocephalus.
Myocardial necrosis secondary to neural lesions in domestic animals.
Journal of the American Veterinary Medical Association    January 15, 1982   Volume 180, Issue 2 144-148 
King JM, Roth L, Haschek WM.Focal myocardial necrosis secondary to neural lesions was diagnosed in 2 dogs and 1 horse. In each case, the neural lesions were traumatic in origin. Spinal cord injury was evident in 1 dog; brain damage was evident in the other dog and presumably in the horse. Retrospective analysis of necropsy material showed that many species were affected, without apparent age or sex predisposition. Central nervous system injury resulting from trauma, infection or space-occupying lesions was associated with acute myocardial necrosis in all cases. The myocardial necrosis was rarely fatal; however, it did ca...
Chemical composition of the spinal cord in the normal developing fetus and in the premature foal.
Journal of reproduction and fertility. Supplement    January 1, 1982   Volume 32 563-567 
Sweasey D, Patterson DS, Leadon DP.The lipid content of spinal cord, expressed as a percentage of adult values, was considerably higher for newborn foals than for several other species and traces of esterified cholesterol (type A) were only rarely present in horse fetal cord (from 270 days gestational age onwards). This suggested that, at birth, the spinal cord is neurochemically more 'mature' in the horse than in cattle, sheep and pigs. Data for premature foals revealed no lipid abnormality suggestive of myelin immaturity or degeneration.
Observations on vascular accidents in the central nervous system of neonatal foals.
Journal of reproduction and fertility. Supplement    January 1, 1982   Volume 32 569-575 
Mayhew IG.A technique for the subarachnoid perfusion-fixation of the central nervous system was developed to help identify various significant vascular accidents (SVAs) in the central nervous system (CNS) of 24 neonatal foals submitted for necropsy. SVAs, comprising subarachnoid, parenchymal and nerve root haemorrhages, and oedema and necrosis, occurred in 17 foals, more frequently in the spinal cord than the brain. They occurred as frequently in premature foals as in those born at full term, in foals born dead as in foals born alive, and in foals born following dystocia with an assisted delivery as in ...
Circulating antibodies to the neuritogenic myelin protein, P2, in neuritis of the cauda equina of the horse.
Nature    September 24, 1981   Volume 293, Issue 5830 299-300 doi: 10.1038/293299a0
Kadlubowski M, Ingram PL.No abstract available
Infection of the central nervous system of horses with equine herpesvirus serotype 1.
Journal of the South African Veterinary Association    September 1, 1981   Volume 52, Issue 3 239-241 
Thein P.During the last 2 years different equine herpesviruses serotype 1 strains have been isolated from cases of paretic or paralytic disease among horses in the Federal Republic of Germany. In this paper the available information is collated and briefly reviewed. A short description of the symptoms and the possible mechanism of the pathogenesis are given.
Serum thyroxine and triiodothyronine concentrations in neonatal foals and mature horses.
American journal of veterinary research    August 1, 1981   Volume 42, Issue 8 1415-1417 
Chen CL, Riley AM.Serum thyroxine (T-4) and triiodothyronine (T-3) concentrations were assayed in neonatal foals (1.5 to 4 months) and mature horses (2 to 25 years old) by a modified radioimmunoassay procedure. Blood was collected from 52 clinically healthy foals and horses of various breeds (Thoroughbreds, Quarter Horses, American Saddle Horses, and a single cross-bred horse). Neonatal foals had high serum concentrations of T-4 (mean, 4.02 microgram/dl) and T-3 (192.9 ng/dl) as compared with the values in mature horses (T-4, mean of 1.76 microgram/dl; T-3, mean of 98.69 ng/dl). Stallions had slightly higher T-...
Erosion of the internal carotid artery and cranial nerve damage caused by guttural pouch mycosis in a horse.
Australian veterinary journal    July 1, 1981   Volume 57, Issue 7 346-347 doi: 10.1111/j.1751-0813.1981.tb05846.x
Hilbert BJ, Huxtable CR, Brighton AJ.No abstract available
Equine protozoal myeloencephalitis: a report of two cases from Western Canada.
The Canadian veterinary journal = La revue veterinaire canadienne    May 1, 1981   Volume 22, Issue 5 140-144 
Clark EG, Townsend HG, McKenzie NT.Two cases of nonsuppurative myeloencephalitis are reported which clinically and pathologically resemble equine protozoal myeloencephalitis. Lesions in both horses were associated with Toxoplasma-like organisms visible in microscopic sections. Clinical signs and lesions in one case primarily involved the brain and in the other case principally involved the spinal cord and associated meninges. Positive identification of the organisms was not achieved; however, the etiological agent is unlikely to be a species of Toxoplasma. Recently published studies suggest a species of Sarcocystis is involved....
Central nervous system demyelination in Venezuelan equine encephalomyelitis infection.
Journal of the neurological sciences    March 1, 1981   Volume 49, Issue 3 397-418 doi: 10.1016/0022-510x(81)90030-7
Dal Canto MC, Rabinowitz SG.Arboviruses are important pathogens for both animals and humans. Venezuelan equine encephalomyelitis virus (VEEV) is an arbovirus whose pathogenicity for grey matter structures has been previously studied. To our knowledge, the present study is the first to describe extensive inflammation and demyelination in spinal cord white matter of mice infected with VEEV. To probe a possible immunepathogenesis of white matter alterations in this infection, nude mice and heterozygous controls were similarly infected. Whereas controls still showed inflammatory demyelination, nude mice showed no white matte...
Normal and abnormal sleep in mammals.
Journal of the American Veterinary Medical Association    January 15, 1981   Volume 178, Issue 2 121-126 
Hendricks JC, Morrison AR.No abstract available
A survey of post mortem findings in 480 horses 1958 to 1980: (1) causes of death.
Equine veterinary journal    January 1, 1981   Volume 13, Issue 1 43-46 doi: 10.1111/j.2042-3306.1981.tb03448.x
Baker JR, Ellis CE.The causes of death recorded in 480 consecutive post mortem examinations of horses performed at the department of pathology, Veterinary Field Station, University of Liverpool, between February 1958 and February 1980, are reported. The alimentary, locomotor and nervous systems were those most frequently diseased. The most common specific entities were those associated with grass sickness, fractures and endoparasitism.
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