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Topic:Coagulation

Coagulation in horses refers to the complex process by which blood forms clots, a critical function that prevents excessive bleeding following injury. This process involves a series of enzymatic reactions that lead to the transformation of liquid blood components into a stable clot. Key elements in equine coagulation include platelets, clotting factors, and fibrinogen. Abnormalities in the coagulation process can result in disorders such as excessive bleeding or thrombosis. Research in this area focuses on understanding the mechanisms of coagulation, identifying potential genetic or environmental influences, and developing diagnostic and therapeutic strategies for coagulation-related disorders in horses. This page compiles peer-reviewed research studies and scholarly articles that explore the mechanisms, regulation, and clinical implications of coagulation in equine health.
Species differences of clotting factors in ox, dog, horse, and man: thromboplastin and proconvertin.
Acta physiologica Scandinavica    December 31, 1957   Volume 41, Issue 4 301-324 doi: 10.1111/j.1748-1716.1957.tb01530.x
STORMORKEN H.No abstract available
Species differences of clotting factors in ox, dog, horse, and man; thrombin and fibrinogen.
Acta physiologica Scandinavica    October 10, 1957   Volume 40, Issue 2-3 167-181 doi: 10.1111/j.1748-1716.1957.tb01486.x
STORMORKEN H.No abstract available
Comparative coagulation studies on horse and human blood.
Journal of comparative pathology    October 1, 1957   Volume 67, Issue 4 358-368 doi: 10.1016/s0368-1742(57)80036-8
BARKHAN P, TOMLIN SC, ARCHER RK.This research article compares the coagulation properties of horse blood to human blood, aiming to explore several previous findings concerning naturally occurring deficiencies in horse plasma and their relevance to [...]
Species differences of clotting factors in ox, dog, horse, and man: proaccelerin and accelerin.
Acta physiologica Scandinavica    June 8, 1957   Volume 39, Issue 2-3 121-136 doi: 10.1111/j.1748-1716.1957.tb01413.x
STORMORKEN H.No abstract available
Coagulation defect in horse plasma. SJOLIN KE.No abstract available
The coagulation mechanism of the blood of the horse with particular reference to its haemophilioid status.
Journal of comparative pathology    July 1, 1955   Volume 65, Issue 3 255-261 doi: 10.1016/s0368-1742(55)80024-0
BELL WN, TOMLIN SC, ARCHER RK.No abstract available
Coagulation mechanism of the horse.
Nature    April 2, 1955   Volume 175, Issue 4457 596-597 doi: 10.1038/175596a0
BELL WN, ARCHER RK, TOMLIN SC.No abstract available
Demonstration of a heparin-like anticoagulant in normal blood. II. Horse blood.
Acta medica Scandinavica. Supplementum    January 1, 1954   Volume 298 1-16 
NILSSON IM.No abstract available
[Blood thickening in horses and their control].
Deutsche tierarztliche Wochenschrift    January 1, 1947   Volume 54, Issue 1-2 12-14 
MAKSIC D.No abstract available
Horse brain thromboplastin; stabilization of activity of dried brain preparations.
Archives of biochemistry    June 1, 1946   Volume 10 183-193 
KAZAL LA, HIGASHI A.No abstract available
Horse brain thromboplastin; stabilization of activity of suspensions.
Archives of biochemistry    June 1, 1946   Volume 10 173-182 
KAZAL LA, HIGASHI A.No abstract available
The Quantitative Changes in the Proteins in the Blood Plasma of Horses in the Course of Immunization.
The Journal of experimental medicine    May 1, 1910   Volume 12, Issue 3 411-434 doi: 10.1084/jem.12.3.411
Gibson RB, Banzhaf EJ.Gravimetric determinations were recorded for the total and several individual proteins (in the sodium oxalate plasma) fractioned with ammonium sulphate and sodium chloride. At precipitation, the plasma salt mixture had been diluted to a final volume of ten times the amount of plasma employed. Coagulations were on aliquot portions of filtrates, and the individual protein constituents (except serumalbumin) were calculated by difference. The eleven horses had been subjected to simultaneous immunization against diphtheria and tetanus toxins, each horse being subsequently continued on the toxin to ...
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