Analyze Diet

Topic:Disease Etiology

Disease etiology in horses refers to the study of the causes and development of diseases within equine populations. It encompasses various factors, including genetic predisposition, environmental influences, infectious agents, and nutritional imbalances, that contribute to the onset and progression of diseases in horses. Understanding disease etiology is essential for identifying risk factors and implementing preventative measures in equine health management. This topic includes research on pathogen-host interactions, the impact of management practices on disease incidence, and the role of genetic and environmental factors in disease susceptibility. This page compiles peer-reviewed research studies and scholarly articles that explore the mechanisms, contributing factors, and implications of disease etiology in horses.
Hydatidosis in a horse.
Veterinary pathology    September 1, 1986   Volume 23, Issue 5 646-647 doi: 10.1177/030098588602300523
Foley GL, Georgi M.No abstract available
Malignant squamous cell thymoma in a horse.
Veterinary pathology    September 1, 1986   Volume 23, Issue 5 627-629 doi: 10.1177/030098588602300514
Whiteley LO, Leininger JR, Wolf CB, Ames TR.No abstract available
An unusual case of osteomyelitis in a horse.
Equine veterinary journal    September 1, 1986   Volume 18, Issue 5 404-407 doi: 10.1111/j.2042-3306.1986.tb03667.x
Tulamo RM, Alitalo I.No abstract available
Loosely coupled skeletal muscle mitochondria in exertional rhabdomyolysis.
Equine veterinary journal    September 1, 1986   Volume 18, Issue 5 418-421 doi: 10.1111/j.2042-3306.1986.tb03673.x
Van den Hoven R, Breukink HJ, Wensing T, Meijer AE, Tigges AJ.No abstract available
Cardiac failure in a stallion secondary to metastasis of an anaplastic pulmonary carcinoma.
Equine veterinary journal    September 1, 1986   Volume 18, Issue 5 414-417 doi: 10.1111/j.2042-3306.1986.tb03671.x
Dill SG, Moise NS, Meschter CL.No abstract available
Splenic hematoma and abscess as a cause of chronic weight loss in a horse.
Journal of the American Veterinary Medical Association    September 1, 1986   Volume 189, Issue 5 557-559 
Spier S, Carlson GP, Nyland TG, Snyder JR, Fischer PE.An 8-year-old gelding with a 3-month history of anorexia and weight loss was found to have a massive subcapsular splenic hematoma. At flank laparotomy, 36 L of fluid was removed from the hematoma. The horse's condition improved after drainage. Fifteen months later, the horse became depressed and febrile. A splenic abscess containing Bacteroides ruminicola and Clostridium sporogenes was found at necropsy.
Medullary thyroid carcinoma in a horse.
Veterinary pathology    September 1, 1986   Volume 23, Issue 5 622-624 doi: 10.1177/030098588602300512
van der Velden MA, Meulenaar H.No abstract available
Focal and multifocal osteosarcoma in two foals.
Equine veterinary journal    September 1, 1986   Volume 18, Issue 5 407-410 doi: 10.1111/j.2042-3306.1986.tb03668.x
Livesey MA, Wilkie IW.No abstract available
Respiratory disease in foals and the epizootiology of equine herpesvirus type 2 infection.
New Zealand veterinary journal    September 1, 1986   Volume 34, Issue 9 152-155 doi: 10.1080/00480169.1986.35331
Fu ZF, Robinson AJ, Horner GW, Dickinson LG, Grimmett JB, Marshall RB.The epizootiology of equine herpesvirus type 2 (EHV-2) infection was investigated in Thoroughbred foals on a stud farm which in previous years had suffered economic loss due to respiratory disease. Sixteen pairs of foals and their dams were selected for this study and all of the foals became infected with EHV-2 by two to four months of age. These animals responded serologically to the virus infection as detected by an enzyme-linked immunosorbent assay (ELISA). EHV-2 infection persisted in these foals for two to six months with constant or intermittent virus recovery. This persistent infection ...
Coxofemoral luxation in two foals wearing hindlimb casts.
Journal of the American Veterinary Medical Association    September 1, 1986   Volume 189, Issue 5 560-561 
Trotter GW, Auer JA, Arden W, Parks A.Two foals wearing hindlimb casts developed coxofemoral luxations 6 days and 9 days after the casts were applied. One foal had a cast on one hindlimb up to the proximal portion of the tibia and the other foal had tube casts on all 4 limbs. Although the incidents that resulted in the luxations were not observed, they probably occurred during awkward attempts to stand or from being pushed by the mare, with the affected limb being caught under the body in a forward extended and adducted position.
Clostridium perfringens type C enterotoxemia in a newborn foal.
Journal of the American Veterinary Medical Association    September 1, 1986   Volume 189, Issue 5 564-565 
Howard-Martin M, Morton RJ, Qualls CW, MacAllister CG.A 1-day old, full-term foal with a history of colic died 2 hours after admission. Necropsy revealed an extremely flaccid, fluid-filled intestinal tract. Histopathologically, the superficial intestinal mucosa was completely necrotic, with minimal inflammatory response. Numerous large, gram-positive rods covered the villi. Clostridium perfringens was isolated on bacteriologic culturing of the intestinal tract contents and was identified as type C by toxin neutralization tests.
Dourine in Southern Africa 1981-1984: serological findings from the Veterinary Research Institute, Onderstepoort.
Journal of the South African Veterinary Association    September 1, 1986   Volume 57, Issue 3 163-165 
Williamson CC, Herr S.The distribution of positive dourine cases found on the complement fixation test at the Veterinary Research Institute, Onderstepoort from 1981 to 1984, is recorded. Within the Republic of South Africa, foci of infection occurred in the Johannesburg, Pretoria, Potchefstroom, Rustenburg, Upington, Lichtenburg, Kroonstad, Louis Trichardt, Middelburg (Cape) and Mossel Bay state veterinary districts. In Bophuthatswana, Transkei, Lesotho, South West Africa and Swaziland, positive cases were also recorded. Anti-complementary activity of horse sera does not present a problem. In donkey and mule sera, ...
Preliminary characterisation of torovirus-like particles of humans: comparison with Berne virus of horses and Breda virus of calves.
Journal of medical virology    September 1, 1986   Volume 20, Issue 1 67-78 doi: 10.1002/jmv.1890200109
Beards GM, Brown DW, Green J, Flewett TH.Pleomorphic virus-like particles have been observed by electron microscopy in the faeces of children and adults with diarrhoea. Some of these particles were approximately 100 nm in diameter and had a "fringe" of closely applied peplomers approximately 10 nm long; they closely resembled Berne virus of horses and Breda virus of calves, the two representatives of a newly proposed family called the Toroviridae. In one sample a toroidal nucleoprotein-like structure was observed within the particles. For two samples a buoyant density of 1.14 g/ml was determined by centrifugation through a sucrose de...
Demonstration of the carrier state in naturally acquired equine arteritis virus infection in the stallion.
Research in veterinary science    September 1, 1986   Volume 41, Issue 2 279-280 
Timoney PJ, McCollum WH, Roberts AW, Murphy TW.The chronic carrier state was virologically confirmed in 15 thoroughbred stallions naturally infected with equine arteritis virus based on the results of test matings and, or, isolations of the virus from semen. Carrier stallions were shown to shed equine arteritis virus in the semen for at least one to two years. Existence of a short-term or convalescent carrier state was also demonstrated in five additional stallions. The frequency of the long-term carrier state in stallions naturally infected with equine arteritis virus was 35 per cent; it varied considerably between groups of stallions on ...
Elastase-producing microorganisms in horse lungs: their possible role in the pathogenesis of chronic pulmonary disease in the horse.
Equine veterinary journal    September 1, 1986   Volume 18, Issue 5 396-400 doi: 10.1111/j.2042-3306.1986.tb03665.x
Grünig G, Von Fellenberg R, Maier R, Corboz L.Seventeen out of 21 horses had pulmonary microbial organisms which reached considerable numbers in seven cases. Elastase-producing microorganisms from the environment (Streptomyces species and to a lesser extent Bacillus species) constituted 22 per cent to 99 per cent (mean 79 per cent) of the total growth. There was a considerable number of microorganisms with in vitro-produced elastases which were not or only slightly affected by horse serum. There was no correlation between numbers of organisms and pulmonary histopathological findings thus the significance of these microorganisms in the pat...
Epidemiology and control of parasites in warm climates.
The Veterinary clinics of North America. Equine practice    August 1, 1986   Volume 2, Issue 2 357-365 doi: 10.1016/s0749-0739(17)30721-6
Craig TM, Courtney CH.The kind of parasites a horse acquires depends upon its environment. Because patterns of transmission vary greatly with climate and management, no one worming program has universal applications. This article discusses epidemiology and control of equine parasites in the southern United States, where climates vary from warm temperate to subtropical and from humid in the southeast to arid in the southwest.
Other parasites. Recent advances.
The Veterinary clinics of North America. Equine practice    August 1, 1986   Volume 2, Issue 2 329-336 doi: 10.1016/s0749-0739(17)30719-8
Klei TR.This article briefly reviews new information on trichostrongylid stomach worms, spirurid stomach worms, tapeworms, threadworms, pinworms, intestinal protozoa, lungworms, eye worms, and numerous other aberrant parasites as it relates to prevalence, pathogenesis of diseases, and treatments.
Ultrastructural study of the equine cecum during onset of laminitis.
American journal of veterinary research    August 1, 1986   Volume 47, Issue 8 1804-1812 
Krueger AS, Kinden DA, Garner HE, Sprouse RF.The morphologic and pathologic changes which occurred within the cecal mucosa of 4 horses during the onset of laminitis were determined from cecal biopsy materials obtained via a cecal fistula; the laminitis was induced with carbohydrate overload. The cecal epithelial mucosa specimens were obtained at 0 (base line), 24, 32, 48, and 72 hours after horses were given carbohydrate overload, and these were fixed and subsequently photographed. Changes in the cecal epithelium were examined by transmission electron and scanning electron microscopies. These histopathologic changes indicated that the mu...
Parasitology.
The Veterinary clinics of North America. Equine practice    August 1, 1986   Volume 2, Issue 2 263-459 
No abstract available
The effects of natural exposure to high levels of zinc and cadmium in the immature pony as a function of age.
Environmental research    August 1, 1986   Volume 40, Issue 2 285-300 doi: 10.1016/s0013-9351(86)80104-9
Kowalczyk DF, Gunson DE, Shoop CR, Ramberg CF.To study the effects of environmental exposure to zinc and cadmium in immature foals, five pregnant ponies were raised within 2.9 km of the New Jersey Zinc Smelter in Palmerton, Pennsylvania. The mares and their foals were kept outdoors on timothy hay and orchard grass. The foals were examined daily for signs of illness and blood samples were taken monthly for estimation of serum zinc, copper, and ceruloplasmin levels. The foals were sacrificed at 2.5, 4.5, 8.5, 13.5, and 18.5 months of age. Necropsy revealed generalized osteochondrosis in joints of the limbs and cervical vertebrae, lymphoid h...
Parasitic skin diseases.
The Veterinary clinics of North America. Equine practice    August 1, 1986   Volume 2, Issue 2 403-437 doi: 10.1016/s0749-0739(17)30725-3
Foil L, Foil C.In this discussion of parasitic skin diseases of horses, details on life cycles are given to aid the understanding and development of control strategies.
Clinicopathologic study of horses surviving pyrrolizidine alkaloid (Senecio vulgaris) toxicosis.
American journal of veterinary research    August 1, 1986   Volume 47, Issue 8 1776-1780 
Lessard P, Wilson WD, Olander HJ, Rogers QR, Mendel VE.Twenty horses of various ages had inadvertently ingested alfalfa hay contaminated with Senecio vulgaris. Among them, 4 died of liver disease. Blood was collected from affected horses at monthly intervals for 7 months and at the 9th and 14th months. The following serum enzymes and chemical items were assayed: aspartate aminotransferase, lactate dehydrogenase, alkaline phosphatase, gamma-glutamyl transferase, sorbitol dehydrogenase, total bilirubin, BUN, glucose, cholesterol, inorganic phosphate, calcium, total protein, and albumin. Amino acid profiles, conjugated bile acids, sulfobromophthalein...
Protozoal diseases.
The Veterinary clinics of North America. Equine practice    August 1, 1986   Volume 2, Issue 2 439-459 doi: 10.1016/s0749-0739(17)30726-5
Mayhew IG, Greiner EC.The clinical and pathologic findings of and therapy for such protozoal diseases as equine protozoal myeloencephalitis, toxoplasmosis, sarcocystosis, pneumocytosis, cryptosporidiosis, giardiasis, besnoitiosis, and klossiellosis are discussed. Emphasis is placed on disorders that occur with greater frequency in North America and on emerging protozoal diseases affecting horses.
[Echinococcosis in a thoroughbred horse imported from Ireland].
DTW. Deutsche tierarztliche Wochenschrift    July 23, 1986   Volume 93, Issue 7 321-323 
Bauer C, Klein M, Murmann W.No abstract available
More on sudden death in racehorses.
Journal of the American Veterinary Medical Association    July 15, 1986   Volume 189, Issue 2 144 
Vasko KA.No abstract available
Bilateral renal hypoplasia in four young horses.
Journal of the American Veterinary Medical Association    July 15, 1986   Volume 189, Issue 2 209-212 
Andrews FM, Rosol TJ, Kohn CW, Reed SM, DiBartola SP.Three horses less than or equal to 3 years old were evaluated because of stunted growth, weight loss, anorexia, depression, and lethargy of at least 1 month's duration. A neonatal foal was examined after its death. In each case, gross and microscopic renal lesions were compatible with bilateral renal hypoplasia (ie, cortical hypoplasia with severe medullary hypoplasia). In young horses with renal failure, bilateral renal hypoplasia should be considered in the differential diagnosis, and may represent a congenital lesion.
Equine ehrlichial colitis (Potomac horse fever): recognition of the disease in Pennsylvania, New Jersey, New York, Ohio, Idaho, and Connecticut.
Journal of the American Veterinary Medical Association    July 15, 1986   Volume 189, Issue 2 197-199 
Palmer JE, Whitlock RH, Benson CE.Equine ehrlichial colitis (Potomac horse fever), a newly identified colitis of the horse, was first recognized in Maryland. In this report, we document occurrence of the disease in Pennsylvania, New Jersey, New York, Ohio, Idaho, and Connecticut. Enzootic areas were recognized by a characteristic pattern. Frequently there was a seasonal pattern and high prevalence of sporadic colitis in unstressed horses. The attack rate per farm generally was low. Horses on pasture, as well as those stabled, were affected. Clinical signs varied from fever and depression to severe diarrhea and laminitis. Occas...
Gastroenterology.
Equine veterinary journal    July 1, 1986   Volume 18, Issue 4 243-343 
No abstract available
Equine herpesviruses causing respiratory disease.
New Zealand veterinary journal    July 1, 1986   Volume 34, Issue 7 118 doi: 10.1080/00480169.1986.35316
Studdert MJ.No abstract available
Barium swallow as an aid to the diagnosis of grass sickness.
Equine veterinary journal    July 1, 1986   Volume 18, Issue 4 294-297 doi: 10.1111/j.2042-3306.1986.tb03633.x
Greet TR, Whitwell KE.Twenty-five horses, suspected on clinical grounds of having grass sickness, were given a barium swallow. Using image intensification the passage of the barium bolus was followed radiographically from the pharynx to the stomach. All 18 horses in which grass sickness was later confirmed at post mortem examination showed defective oesophageal motility. Although the specificity of the abnormality could not be proven, the technique offers a valuable aid to the ante mortem diagnosis of grass sickness.