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Topic:Equine Diseases

Equine diseases encompass a wide range of health conditions that can affect horses, including infectious diseases, metabolic disorders, and genetic conditions. These diseases can impact the overall health, performance, and well-being of horses. Common equine diseases include equine influenza, equine herpesvirus, laminitis, and equine metabolic syndrome. Diagnosis and management of these diseases often require a combination of clinical evaluation, laboratory testing, and appropriate treatment strategies. This page gathers peer-reviewed research studies and scholarly articles that explore the etiology, pathophysiology, diagnosis, and treatment options for various equine diseases, providing valuable insights for veterinarians and researchers in the field.
Serological response in mares affected by contagious equine metritis 1977.
The Veterinary record    April 1, 1978   Volume 102, Issue 13 277-280 doi: 10.1136/vr.102.13.277
Benson JA, Dawson FL, Durrant DS, Edwards PT, Powell DG.A serum agglutination and antiglobulin test is described for the detection of antibodies to the contagious equine metritis organism. A provisional interpretation of the test is proposed and using this interpretation the results of 66 such tests are discussed.
Efficacy of trivalent inactivated encephalomyelitis virus vaccine in horses.
American journal of veterinary research    April 1, 1978   Volume 39, Issue 4 621-625 
Barber TL, Walton TE, Lewis KJ.Twenty-nine horses were vaccinated with a trivalent (Venezuelan, eastern, and western) inactivated equine encephalomyelitis virus vaccine. The vaccine purchased for this study was the only one licensed and commercially available in May, 1975. Plaque-neutralizing and hemagglutinin-inhibiting antibodies in response to each of the 3 equine encephalomyelitis viruses were determined after vaccination. Horses had rising levels of plaque-neutralizing and hemagglutinin-inhibiting antibodies shortly after injection with the 1st and 2nd doses of the vaccine (given 3 weeks apart) and were refractory to c...
Equine sarcoids.
The Veterinary record    March 18, 1978   Volume 102, Issue 11 248 doi: 10.1136/vr.102.11.248-a
Robinson M, Haywood S.No abstract available
Unusual cause of lameness in a pony.
The Veterinary record    March 18, 1978   Volume 102, Issue 11 247 doi: 10.1136/vr.102.11.247
Aitken DC, Simpson JW.No abstract available
CEM and the foaling mare.
The Veterinary record    March 18, 1978   Volume 102, Issue 11 246 doi: 10.1136/vr.102.11.246
Timoney PJ, Ward J, McArdle JF.No abstract available
[Demonstration of a clinically manifested mixed reovirus serotype III and rhinopneumonitis virus infection in a horse].
Berliner und Munchener tierarztliche Wochenschrift    March 15, 1978   Volume 91, Issue 6 103-106 
Thein P.No abstract available
Lungworm (Dictyocaulus arnfieldi) infection in the horse.
The Veterinary record    March 11, 1978   Volume 102, Issue 10 216-217 doi: 10.1136/vr.102.10.216-a
Nicholls JM, Duncan JL, Greig WA.No abstract available
Equine rhinopneumonitis vaccine: immunogenicity and safety in adult horses, including pregnant mares.
American journal of veterinary research    March 1, 1978   Volume 39, Issue 3 377-383 
Purdy CW, Ford SJ, Porter RC.No abstract available
Equine abortion associated with herpesvirus.
Australian veterinary journal    March 1, 1978   Volume 54, Issue 3 151 doi: 10.1111/j.1751-0813.1978.tb05537.x
Peet RL, Coackley W, Smith VW, Main C.No abstract available
Induction of a cell membrane antigen by equine infectious anemia virus.
American journal of veterinary research    March 1, 1978   Volume 39, Issue 3 385-386 
McGuire TC, Crawford TB.Equine fibroblasts persistently infected with equine infectious anemia virus acquire a new cell membrane antigen demonstrable by indirect radioimmunoassay, using infected horse serum as an antibody source.
Acute laryngeal paralysis of Arabian foals.
Australian veterinary journal    March 1, 1978   Volume 54, Issue 3 154 doi: 10.1111/j.1751-0813.1978.tb05540.x
Rose RJ.No abstract available
Pyrrolizidine alkaloid poisoning of horses.
Australian veterinary journal    March 1, 1978   Volume 54, Issue 3 150 doi: 10.1111/j.1751-0813.1978.tb05536.x
Seaman JT.No abstract available
[History of the discovery of the causative agent of Stachybotrys toxicosis].
Mikrobiolohichnyi zhurnal    March 1, 1978   Volume 40, Issue 2 264-270 
Aĭzenman BE, Kudlaĭ DG.No abstract available
Primary ocular squamous cell carcinoma with metastasis in a horse.
Veterinary medicine, small animal clinician : VM, SAC    March 1, 1978   Volume 73, Issue 3 287-290 
Eversole TG, Lavach JD.No abstract available
Natural intra-uterine infection of foals with equine herpes virus type 1 in Victoria.
Australian veterinary journal    March 1, 1978   Volume 54, Issue 3 147 doi: 10.1111/j.1751-0813.1978.tb05533.x
Westbury HA, Kovesdy L, Barton MD.No abstract available
Isolation of Actinobacillus suis from a colt.
The British veterinary journal    March 1, 1978   Volume 134, Issue 2 122-127 doi: 10.1016/s0007-1935(17)33536-4
Harbourne JF, Mair NS, Keighley SG.No abstract available
A laboratory system for production of flexion rates and forces in the forelimb of the horse.
American journal of veterinary research    March 1, 1978   Volume 39, Issue 3 365-369 
Kingsbury HB, Quddus MA, Rooney JR, Geary JE.The distal portion of the forelimb of the horse is provided with a stay apparatus composed of tendons, ligaments, and fascia. This stay apparatus provides the major resistance to joint flexion during the support phase of the stride. The laboratory test system described was shown to be able to reproduce in vitro limb motions and hoof forces measured with a running horse. These results indicated the stay apparatus operates in a largely passive mode, active muscle contraction apparently serving to provide rigidity only early in the support phase of the stride. The testing system described was des...
Surgical implications of extensibility of the skin of the equine carpus.
American journal of veterinary research    March 1, 1978   Volume 39, Issue 3 387-392 
Cartee RE, Cowles WR.To determine the lines of maximum extensibility of the skin over the equine carpus, round puncture wounds were made 2.0 cm apart over the carpal area of 5 horses (7 carpi). The direction of elongation of the round puncture wound was observed and photographed. Lines of maximum extensibility that occurred over the surfaces of the equine carpus were determined to be proximal to distal, except in a small area over the accessory carpal bone where a state of anisotropism existed. In an immobilized carpus, direction of a surgical incision was not as important as it was in a mobile carpus, in which th...
Study of homologous and heterologous antibody response in California horses vaccinated with attenuated Venezuelan equine encephalomyelitis vaccine (strain TC-83).
American journal of veterinary research    March 1, 1978   Volume 39, Issue 3 371-376 
Ferguson JA, Reeves WC, Milby MM, Hardy JL.Of 359 horses vaccinated with attenuated Venezuelan equine encephalomyelitis (VEE) vaccine (strain TC-83), 87% developed hemagglutination-inhibition (HI) antibodies to VEE virus within 1 month. Blood from a subsample of 101 of the 359 horses was obtained over a 1-year period. Within 1 month after vaccination, 84% of the 101 horses had developed VEE HI antibodies, 87% had developed VEE-neutralizing (Nt) antibodies, and 78% had developed VEE complement-fixing (CF) antibodies. One year after vaccination, 58% of the horses had VEE HI antibodies and 73% had VEE Nt antibodies. The percentage of hors...
[Occurence of an outbreak of horse dermatophytosis caused by the fungus Trichophyton equinum].
Veterinarni medicina    March 1, 1978   Volume 23, Issue 3 175-184 
Stros K, Krivanec K, Komárek J, Malinský B.There is a description of equine dermatophytosis enzootic, caused by the microscopic fungus Trichophyton equinum. The disease affected 32 horses, mostly young, all in the same herd (74.4%). The diseased horses were successfully treated with the preparation Fenoform forte, applied superficially at the concentration of 0.5% of the active substance. The authors made an attempt to determine the criteria for clinical differentiation of fully developed trichophytosis and microsporosis of horses. Trichophytosis (T. equinum) is characterized as typical numerous small and round patches, covered by smal...
Additional recommendations for the control of contagious equine metritis 1977.
The Veterinary record    February 25, 1978   Volume 102, Issue 8 161 doi: 10.1136/vr.102.8.161
David JS, Frank CJ, Powell DG.No abstract available
Equine sarcoids.
The Veterinary record    February 25, 1978   Volume 102, Issue 8 179-180 doi: 10.1136/vr.102.8.179
Baker JR, Neal PA, Wyn-Jones G.No abstract available
Comparison of the bacterial flora of three sites in the genital tract of the mare.
The Veterinary record    February 25, 1978   Volume 102, Issue 8 169-170 doi: 10.1136/vr.102.8.169
Newcombe JR.No abstract available
[Clinical experiences with a prostaglandin F2alpha-analog (Equimate) in the mare].
Berliner und Munchener tierarztliche Wochenschrift    February 19, 1978   Volume 91, Issue 4 61-64 
Leidl W, Stolla R, Rockel P, Mayr B, Färber A.No abstract available
Survival properties of the causal agent of contagious equine metritis 1977.
The Veterinary record    February 18, 1978   Volume 102, Issue 7 152 
Timoney PJ, Harrington A, McArdle J, O'Reilly P.No abstract available
Occurrence and distribution of western equine encephalomyelitis in Florida.
Journal of the American Veterinary Medical Association    February 1, 1978   Volume 172, Issue 3 351-352 
Hoff GL, Bigler WJ, Buff EE, Beck E.Research and surveillance programs relating to the occurrence and distribution of western equine encephalomyelitis virus in Florida, conducted between 1955 and 1976, suggest that the virus is (1) an endemic arbordae, (2) transmitted in a continuous cycle throughout the year by Culiseta melanura mosquitoes, and (3) restricted to fresh water swamps and waterways in central, north, and northwest Florida.
Retrospective study of 350 cases of equine cryptorchidism.
Journal of the American Veterinary Medical Association    February 1, 1978   Volume 172, Issue 3 343-346 
Stickle RL, Fessler JF.Equine cryptorchidism was examined by a review of the literature and a retrospective study of 350 horses over a 14-year period. The incidence of left vs right testis retention was nearly equal. On the left side, 75.2% of the retained testes were retained abdominally and 24.8% inguinally; on the right side, 41.8% of the retained testes were retained abdominally and 58.2% inguinally. Preoperative diagnosis by rectal palpation of the vaginal rings was considered a valuable technique, with 87.9% accuracy in 190 horses. Invasive and nonivasive surgical techniques for abdominal cryptorchidectomy and...
Eastern equine encephalitis in Massachusetts, 1957-1976. A prospective study centered upon analyses of mosquitoes.
American journal of epidemiology    February 1, 1978   Volume 107, Issue 2 170-178 doi: 10.1093/oxfordjournals.aje.a112519
Grady GF, Maxfield HK, Hildreth SW, Timperi RJ, Gilfillan RF, Rosenau BJ, Francy DB, Calisher CH, Marcus LC, Madoff MA.Reappearance of eastern equine encephalitis (EEE) in Massachusetts residents in the 1970's provided an opportunity to assess the predictive value of data on rainfall, EEE in horses, and carriage of EEE virus (EEEV) by mosquitoes, factors which had been studied annually since the last EEE outbreak in 1955-1956. The cycle of multiple cases during 1973-1975 started in a second consecutive year of rainfall that exceeded the annual mean by more than 20 cm, conditions recapitulating the 1955-1956 experience. In 1973, widespread EEE fatalities in horses presaged human cases, another recapitulation of...
Role of intracranial [H+] receptor in physiologic regulation of ventilation in ponies.
Chest    February 1, 1978   Volume 73, Issue 2 Suppl 253-256 doi: 10.1378/chest.73.2_supplement.253
Forster HV, Bisgard GE, Dempsey JA, Orr JA.Numerous studies have demonstrated the existence of an intracranial [H+] chemoreceptor mechanism capable of stimulating ventilation. 1 Supposedly, this chemoreceptor is located 0.2 mm below the surface of the ventrolateral side of the medulla and is responsive to [H+] in the surrounding cerebral extracellular fluid (ECF). During chronic conditions, ECF [H+] is supposedly in equilibrium with CSF [H+]; hence, stimulus level can be established through sampling and analysis of CSF. In this presentation, we summarize data from studies on spontaneously breathing, unanesthetized ponies which suggests...
Veterinary practices of the Plains Indians.
Modern veterinary practice    February 1, 1978   Volume 59, Issue 2 99-102 
Stewart MJ.No abstract available