Analyze Diet

Topic:Toxic plants

Toxic plants are a concern in equine management as they pose health risks to horses when ingested. These plants contain various compounds that can lead to adverse effects ranging from mild discomfort to severe illness or death. Horses may encounter toxic plants in pastures, hay, or during foraging. Common toxic plants include ragwort, yew, and oleander, each containing specific toxins that affect equine physiology differently. Understanding the identification, distribution, and toxicological impact of these plants is important for prevention and management. This page compiles peer-reviewed research studies and scholarly articles that explore the identification, toxic mechanisms, and management strategies of toxic plants in relation to equine health.
Evaluation of an analytical method for the diagnosis of cantharidin toxicosis due to ingestion of blister beetles (Epicauta lemniscata) by horses and sheep.
American journal of veterinary research    June 1, 1980   Volume 41, Issue 6 932-933 
Ray AC, Post LO, Hurst JM, Edwards WC, Reagor JC.An analytical method for cantharidin, using high performance liquid chromatography, was applied to field specimens from horses and sheep with blister beetle (Epicauta lemniscata) poisoning. Stomach content and urine proved to be valuable aids in diagnosis. One incident of cantharidin toxicosis in ruminants (sheep) was confirmed.
Urinary excretion of arsenic from horses injected with an organic arsenical compound.
The British veterinary journal    March 1, 1980   Volume 136, Issue 2 190-192 doi: 10.1016/s0007-1935(17)32343-6
Littlejohn A, Virlas D.Five doses of sodium cacodylate (194 mg) were administered intravenously to two horses at two-day intervals. The arsenic content of daily urine samples was determined for five days after the final dose, by which time urinary arsenic concentrations were close to those of control samples. It was considered that these results reflected a common property of organic arsenicals viz. more rapid excretion by mammals than is the case with inorganic arsenic compounds.
Teratogenicity and toxicity of coniine in cows, ewes, and mares.
The Cornell veterinarian    January 1, 1980   Volume 70, Issue 1 19-26 
Keeler RF, Balls LD, Shupe JL, Crowe MW.Cows, ewes, and mares varied considerably in susceptibility to toxicoses from the oral administration of the piperidine alkaloid, coniine. Cows were most susceptible and ewes least. Only calves had teratogenic effects from maternal administration of coniine during gestation; lambs and foals were apparently resistant. Results suggest that the marked differences between cattle and sheep are probably not due to variation in gut absorption or rumen metabolism.
Phenylbutazone toxicity in ponies.
The Veterinary record    August 18, 1979   Volume 105, Issue 7 150-151 doi: 10.1136/vr.105.7.150
Lees P, Michell AR.The oral administration of phenylbutazone at a dose rate of approximately 10 mg per kg per day for seven to 14 days resulted in the development of signs of toxicity in seven of eight ponies treated. Clinical signs included anorexia, depression and abdominal oedema. Blood biochemical determinations showed a decrease in total plasma protein and calcium concentrations with an increase in urea concentration. These changes were considered indicative of water retention. Three of the ponies died during treatment following the development of shock. Shock was considered to arise from the submucosal oed...
Pathology of blister beetle (Epicauta) poisoning in horses.
Veterinary pathology    January 1, 1979   Volume 16, Issue 1 18-31 doi: 10.1177/030098587901600102
Schoeb TR, Panciera RJ.No abstract available
Crofton weed (Eupatorium adenophorum) toxicity in horses.
Australian veterinary journal    January 1, 1979   Volume 55, Issue 1 19-21 doi: 10.1111/j.1751-0813.1979.tb09538.x
O'Sullivan BM.The clinical signs and pathology of 6 field cases of a respiratory disease of horses which occurs in the coastal hinterland of south-eastern Queensland are described. The condition has occurred for many years and has been thought to have been associated with ingestion of Crofton weed (Eupatorium adenophorum). Coughing, rapid heaving respiration, decreased exercise tolerance and loss of condition were seen in affected horses. In longstanding cases fibrosis, alveolar lining cell proliferation, oedema, neutrophil infiltration and abscessation were seen. In some cases vascular thrombosis and infar...
Nodular goiters of three mares and their foals, induced by feeding excessive amount of seaweed.
Nihon juigaku zasshi. The Japanese journal of veterinary science    December 1, 1978   Volume 40, Issue 6 749-753 doi: 10.1292/jvms1939.40.749
Miyazawa K, Motoyoshi S, Usui K.No abstract available
Ergot toxicosis.
Modern veterinary practice    June 1, 1978   Volume 59, Issue 6 463-464 
Lindley WH.No abstract available
Acute bracken fern poisoning in a 14-month-old horse.
Veterinary medicine, small animal clinician : VM, SAC    March 1, 1978   Volume 73, Issue 3 295-296 
Kelleway RA, Geovjian L.No abstract available
Pyrrolizidine alkaloid poisoning of horses.
Australian veterinary journal    March 1, 1978   Volume 54, Issue 3 150 doi: 10.1111/j.1751-0813.1978.tb05536.x
Seaman JT.No abstract available
[History of the discovery of the causative agent of Stachybotrys toxicosis].
Mikrobiolohichnyi zhurnal    March 1, 1978   Volume 40, Issue 2 264-270 
Aĭzenman BE, Kudlaĭ DG.No abstract available
An outbreak of Swainsona poisoning in horses.
Australian veterinary journal    September 1, 1977   Volume 53, Issue 9 446-447 doi: 10.1111/j.1751-0813.1977.tb05496.x
O'Sullivan BM, Goodwin JA.On 6 properties in south-western Queensland an outbreak of nervous disease occurred horses due to ingestion of Swainsonia (Darling pea). Loss of condition, depression, hyperaesthesia and hyperexcitability were seen in affected horses. At autopsy of 2 horses generalised c ytoplasmic vacuolation was seen in the neurones of the central nervous system and in the liver, adrenal and thyroid. The clinical and pathological features were similar to those described in horses suffering from Swainsona poisoning in Australia and Astragalus and Oxytropis in North America.
Bromide intoxication of horses, goats, and cattle.
Journal of the American Veterinary Medical Association    September 1, 1977   Volume 171, Issue 5 446-448 
Knight HD, Costner GC.During the summer and fall of 1973, a few horses, goats, and cattle in the Napa Valley of California became intoxicated by bromide via the ingestion of volunteer oat hay that had been cut from a field treated with methyl bromide, a soil fumigant. The bromide content of the hay ranged from 6,00 to 8,400 ppm. Signs of intoxication were lethargy, weakness, and ataxia. Animals experimentally fed the contaminated hay developed signs of intoxication between the 7th and 9th days.
Herbicide risk.
The Veterinary record    April 23, 1977   Volume 100, Issue 17 371 doi: 10.1136/vr.100.17.371-b
Cleverly JM.No abstract available
Acorn poisoning.
The Veterinary record    December 4, 1976   Volume 99, Issue 23 465-466 doi: 10.1136/vr.99.23.465
Daniels MG.No abstract available
Acorn poisoning.
The Veterinary record    November 13, 1976   Volume 99, Issue 20 403-404 doi: 10.1136/vr.99.20.403
Broughton JE.No abstract available
Acorn poisoning.
The Veterinary record    October 23, 1976   Volume 99, Issue 17 343 doi: 10.1136/vr.99.17.343-a
Wharmby MJ.No abstract available
[Hematological effects produced on horses and sheeps pasturing upon Brachiaria radicans Napper (Tanner grass) in winter time (author’s transl)].
Arquivos do Instituto Biologico    July 1, 1976   Volume 43, Issue 3-4 99-103 
Rosenfeld G, Reichmann CE, Jaria LJ, Andrade SO.Hematological studies were developed on two mares and twelve adults castrate sheeps of "Ideal" breed. The animals had been on a pasture formed with Brachiaria radicans Napper for one and two months respectively. This experiment was developed in winter time, so the leaves of the graminea had a yellow-green color. Clinical and hematological observations were made weekly. The typical symptoms of intoxication failed to present as observed in the former investigation when the plant was green (3). The hematological exames showed only for ovines a slight anemia, it was also characterized as being mac...
Effects of crude extracts of various plants on infectious bovine rhinotracheitis virus-plaque production.
American journal of veterinary research    February 1, 1976   Volume 37, Issue 2 215-218 
Kelling CL, Schipper IA, Schermeister LJ, Vacik JP.Extracts of 28 plants were tested without demonstable antiviral activity in an agar-overlay plaque-reduction antiviral assay system, using infectious bovine rhinotracheitis virus and bovine endocardial cell cultures. Ethanolic extract of Narcissus tazetta L bulb elicited antiviral activity by inhibition of viral plaque formation. Antiviral activity was demonstrated against infectious bovine rhinotracheitis and equine rhinopneumonitis viruses. Narcissus tazetta L bulb did not directly inactivate the virus extracellularly. The extract exhibited only limited toxicity to rapidly multiplying bovine...
Hypercalcemia and calcinosis in Florida horses: implication of the shrub, Cestrum diurnum, as the causative agent.
The Cornell veterinarian    January 11, 1975   Volume 65, Issue 1 26-56 
Krook L, Wasserman RH, Shively JN, Tashjian AH, Brokken TD, Morton JF.A chronic debilitating disease is described in Florida horses. There is progress weight loss and lameness of increasing severity. Plasma calcium is elevated to moderate or severe degree. Anatomical changes include dystrophic calcinosis of elastic tissues, viz. major arteries, tendons and ligaments. A generalized osteopetrosis is present and may be related to hypoparathyroidsim and hypercalcitoninism. The presence of Cestrum diurnum (day-blooming jessamine, day cestrum, wild jasmin) in areas accessible to affected animals, the observation that leaves of the plant were stripped in these areas, a...
Thiaminases and their effects on animals.
Vitamins and hormones    January 1, 1975   Volume 33 467-504 doi: 10.1016/s0083-6729(08)60970-x
Evans WC.No abstract available
Cyanide poisoning in horses.
Veterinary medicine, small animal clinician : VM, SAC    September 1, 1974   Volume 69, Issue 9 1166-1167 
Prodjoharjono S.No abstract available
Hypercalcaemia and mineralisation of non-osseous tissues in horses due to vitamin-D toxicity.
Zentralblatt fur Veterinarmedizin. Reihe A    August 1, 1974   Volume 21, Issue 8 638-643 doi: 10.1111/j.1439-0442.1974.tb01348.x
Muylle E, Oyaert W, De Roose P, Van Den Hende C.No abstract available
Oral toxicity of azinphosmethyl in horses.
American journal of veterinary research    August 1, 1974   Volume 35, Issue 8 1031-1035 
Giri SN, Peoples SA, Llaguno GV, Mull RL.No abstract available
The interaction of toxic amounts of lead and zinc fed to young growing horses.
The Veterinary record    October 14, 1972   Volume 91, Issue 16 382-383 doi: 10.1136/vr.91.16.382
Willoughby RA, Macdonald E, Mcsherry BJ, Brown G.No abstract available
The effects of chloroform or halothane on horse liver in the early stage of ragwort poisoning.
The Journal of pathology    August 1, 1972   Volume 107, Issue 4 253-263 doi: 10.1002/path.1711070405
Gopinath C, Ford EJ, Jones RS.No abstract available
Case report. An outbreak of locoweed poisoning in horses in Southwestern Alberta.
The Canadian veterinary journal = La revue veterinaire canadienne    June 1, 1972   Volume 13, Issue 6 141-145 
Harries WN, Baker FP, Johnston A.No abstract available
Insecticide spray.
Archives of internal medicine    May 1, 1972   Volume 129, Issue 5 834 
Koloff WJ.No abstract available
Acute hemolytic anemia caused by wild onion poisoning in horses.
Journal of the American Veterinary Medical Association    February 1, 1972   Volume 160, Issue 3 323-327 
Pierce KR, Joyce JR, England RB, Jones LP.No abstract available
Syndromes of locoweed poisoning in livestock.
Clinical toxicology    January 1, 1972   Volume 5, Issue 4 567-573 doi: 10.3109/15563657208991031
James LF.No abstract available.