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Topic:Neurological Diseases

Neurological diseases in horses encompass a range of disorders affecting the central and peripheral nervous systems. These conditions can arise from various etiologies, including infectious agents, genetic predispositions, trauma, or metabolic imbalances. Common neurological diseases in horses include equine protozoal myeloencephalitis (EPM), equine herpesvirus myeloencephalopathy (EHM), and cervical vertebral stenotic myelopathy (CVSM), also known as wobbler syndrome. Clinical signs associated with these diseases may include ataxia, weakness, altered gait, and changes in behavior or mental status. Diagnosis typically involves a combination of clinical evaluation, imaging techniques, and laboratory testing. This page compiles peer-reviewed research studies and scholarly articles that investigate the pathophysiology, diagnostic approaches, and treatment options for neurological diseases in equine populations.
[Neurological disorders caused by equine herpesvirus type 1 and cauda equina neuritis in horses].
Tijdschrift voor diergeneeskunde    December 15, 1984   Volume 109, Issue 24 1027-1035 
Sloet van Oldruitenborgh-Oosterbaan MM, Binkhorst GJ.The differences in aetiology, symptomatology, pathomorphology, diagnosis and therapy between the nervous form (paralytic form) of Equine Herpes Virus, type 1, and Neuritis Caudae Equinae are reviewed. The conclusion is that in most cases it is possible to differentiate between these two clinical syndromes.
Clinical and pathological studies of brain injuries in horse-riding accidents: a description of cases and review with a warning to the unhelmeted.
Clinical neuropathology    November 1, 1984   Volume 3, Issue 6 253-259 
Ilgren EB, Teddy PJ, Vafadis J, Briggs M, Gardiner NG.We present a clinical and pathological study of brain injuries in horse-riding accidents. Among six fatal cases, all experienced riders varying in age and sex, two were not wearing helmets and four had inadequate protective headgear. Among five non-fatal cases, four wore adequate helmets and only one of these suffered permanent neurological damage. The fifth was not wearing a helmet and although he eventually returned to work, a CT scan following the fall showed a deep-seated perithalamic hematoma. These findings are consistent with those of virtually all other studies in that they suggest tha...
Vestibular syndrome associated with temporohyoid joint fusion and temporal bone fracture in three horses.
Journal of the American Veterinary Medical Association    October 1, 1984   Volume 185, Issue 7 775-781 
Blythe LL, Watrous BJ, Schmitz JA, Kaneps AJ.Acute onset of vestibulocochlear and facial nerve dysfunction due to a stress fracture of the petrous part of the temporal bone was diagnosed in 3 horses. The fracture was secondary to chronic inflammatory changes in the petrous part of the temporal bone and the proximal stylohyoid bone, with fusion of the temporohyoid joint. Bacterial meningoencephalitis was a complicating factor. Treatment resulted in reduction of severity of clinical signs in 2 of the 3 cases, but residual compensated vestibular deficits persisted.
Protozoal myeloencephalitis in horses in California.
Journal of the American Veterinary Medical Association    October 1, 1984   Volume 185, Issue 7 801-802 
Dorr TE, Higgins RJ, Dangler CA, Madigan JE, Witham CL.Three cases of equine protozoal myeloencephalitis were diagnosed over a 12-month period in horses that had never left the state of California. These cases suggest that the disease is enzootic in California.
Equine adenovirus 1 isolated from cauda equina neuritis.
Research in veterinary science    September 1, 1984   Volume 37, Issue 2 252-254 
Edington N, Wright JA, Patel JR, Edwards GB, Griffiths L.Equine adenovirus 1 was recovered after four to six passages from two out of three cases of cauda equina neuritis (CEN) using kidney monolayers. Similar treatment of lumbo-sacral spinal cord from six normal horses did not yield adenovirus. All three cases of CEN had antibodies to the neuritogenic myelin protein P2 while immunofluorescence demonstrated that autologous IgG bound to the myelin of affected nerves. Adenovirus was not detected in neural tissue by immunofluorescence.
Ventral approach for stabilization of atlantoaxial subluxation secondary to odontoid fracture in a foal.
Journal of the American Veterinary Medical Association    September 1, 1984   Volume 185, Issue 5 545-549 
McCoy DJ, Shires PK, Beadle R.Atlantoaxial subluxation secondary to odontoid fracture in a 30-day-old foal was corrected by alignment of the atlantoaxial joint and stabilization with 2 ventrally placed dynamic compression plates. At 90 days after surgery, healing of the fracture, with adequate alignment of the atlantoaxial joint, was confirmed radiographically. The foal was only slightly tetraparetic at that time. At 1 year after surgery, the gait was normal. It was concluded that the technique has advantages over the use of Steinmann's pins or external coaptation for stabilization. The ventral approach allows decompressio...
Neuroaxonal dystrophy of the accessory cuneate nucleus in horses.
Veterinary pathology    July 1, 1984   Volume 21, Issue 4 384-393 doi: 10.1177/030098588402100404
Beech J.Data were collected from 37 horses with a neurologic disability and compared to a group of 34 normal horses. Affected horses had neuroaxonal dystrophy, gliosis, vacuoles, and sometimes pigment localized to the accessory cuneate nuclei with minimal or no changes in the spinal cord and no changes in the proximal peripheral nerves. The focal nature of the change and usual absence of significant light microscopic spinal cord or peripheral nerve changes are different than previously described equine neuropathologic conditions.
Neurological examination of newborn foals.
Equine veterinary journal    July 1, 1984   Volume 16, Issue 4 306-312 doi: 10.1111/j.2042-3306.1984.tb01933.x
Adams R, Mayhew IG.Behaviour and response to neurological testing of apparently healthy newborn foals differed significantly from the adult in several ways. Foals responded to external stimulation with exaggerated movements although they tended to sink into a relaxed state when restrained. They had a more angular head position and assumed a base wide stance. The menace reflex developed during the first two weeks post partum. In general, the foals' gait was choppy or dysmetric. In lateral recumbency the foals had increased extensor tone, hyperreflexive tendon reflexes, crossed extensor reflexes as well as recumbe...
Lymphosarcoma as the cause of ataxia in a horse.
Journal of the American Veterinary Medical Association    June 15, 1984   Volume 184, Issue 12 1517-1518 
Shamis LD, Everitt JI, Baker GJ.No abstract available
Spinal cord malacia following general anaesthesia in the horse.
The Veterinary record    June 9, 1984   Volume 114, Issue 23 569-570 doi: 10.1136/vr.114.23.569
Blakemore WF, Jefferies A, White RA, Paton IM, Dobson H.No abstract available
Cauda equina neuritis: a chronic idiopathic polyneuritis in two horses.
The Canadian veterinary journal = La revue veterinaire canadienne    May 1, 1984   Volume 25, Issue 5 214-218 
Rousseaux CG, Futcher KG, Clark EG, Naylor JM.Two cases of cauda equina neuritis are compared and contrasted. Neurological deficits of the tail and perineum were noted and functional deficits were seen in gait, urination, defecation and cranial nerve function. Lesions consisted of nonsuppurative inflammation of the nerve trunks and proliferation of the perineurium of the cauda equina. Cranial nerve involvement in one case supported a diagnosis of polyneuritis equi rather than cauda equina neuritis. The possible etiologies and pathogenesis of this disease are discussed.
Pain perception and alleviation in animals.
Federation proceedings    April 1, 1984   Volume 43, Issue 5 1307-1312 
Erickson HH, Kitchell RL.In the last 2 decades there have been substantial advances in our knowledge of the scientific basis of the mechanisms of pain. Nociceptors or pain receptors are widespread in the skin and tissues of animals; chemical mediation of nociceptor excitation may provide a key for understanding the peripheral phenomena related to pain. The expression of pain in animals involves multiple ascending and descending branches, as well as specialized pain-signaling mechanisms in the spinal cord. The importance of these different pathways varies with species and circumstances. Endogenous neural systems in the...
A vestibular syndrome associated with Cryptococcus neoformans in a horse.
Zentralblatt fur Veterinarmedizin. Reihe A    March 1, 1984   Volume 31, Issue 2 132-139 doi: 10.1111/j.1439-0442.1984.tb01268.x
Teuscher E, Vrins A, Lemaire T.No abstract available
[The neurological form of EVH1 in horses].
Tijdschrift voor diergeneeskunde    February 1, 1984   Volume 109, Issue 3 94 
van Oirschot JT, Binkhorst JT, Breukink HJ.No abstract available
Narcolepsy in a horse.
Journal of the American Veterinary Medical Association    January 15, 1984   Volume 184, Issue 2 131-132 
Dreifuss FE, Flynn DV.No abstract available
[Hypophyseal adenocarcinoma in a horse. A case study].
Tierarztliche Praxis    January 1, 1984   Volume 12, Issue 3 354-358 
Martens J, Rosenbruch M.The paper presents the case-history of a 5-year-old pony suffering from an adenocarcinoma which proceeded from the pars intermedia of the pituitary gland. Neurological and patho-morphological findings are reported.
Verminous (Strongylus vulgaris) myelitis in a donkey.
The Cornell veterinarian    January 1, 1984   Volume 74, Issue 1 30-37 
Mayhew IG, Brewer BD, Reinhard MK, Greiner EC.A fifth stage Strongylus vulgaris migrated through the spinal cord of a 2-year-old, male donkey resulting in progressive paraparesis and then tetraplegia. A profound neutrophilic pleocytosis was detected on analysis of cerebrospinal fluid. The parasite appeared to have entered the mid-lumbar spinal cord, migrated to the cranial thoracic segments, exited, then re-entered the spinal cord a few segments craniad. It then traveled further cranially and was found in the third cervical spinal cord segment. Some parts of the lesion were remarkably free from tissue necrosis, hemorrhage and inflammation...
[Medial motor nucleus in the lumbosacral segment of the spinal cord of the horse].
Polskie archiwum weterynaryjne    January 1, 1984   Volume 24, Issue 1 133-137 
Sławomirski J, Flieger S, Jastrzebski M, Boratyński Z.The studies carried out on 2 spinal cords of horses showed that cells of the medial motor nucleus (nucleus motorius medialis) are present in all neuromers of the lumbar and sacral segment of the spinal cord. It lies in the medial part of grey matter of the ventral column, neighbouring laterally and ventrally with cells of the lateral motor nucleus, whereas dorsally with cells of the nucleus of the ventral commissural horn. Along the nucleus numerous constrictions and intervals are found, which are connected with various numbers of nerve cells in particular cross-sections.
[Nucleus motorius medialis in the lumbosacral segment of the spinal cord in horses].
Polskie archiwum weterynaryjne    January 1, 1984   Volume 24, Issue 1 133-137 
Sławomirski J, Flieger S, Jastrzebski M, Boratyński Z.The studies carried out on 2 spinal cords of horses showed that cells of the medial motor nucleus (nucleus motorius medialis) are present in all neuromers of the lumbar and sacral segment of the spinal cord. It lies in the medial part of grey matter of the ventral column, neighbouring laterally and ventrally with cells of the lateral motor nucleus, whereas dorsally with cells of the nucleus of the ventral commissural horn. Along the nucleus numerous constrictions and intervals are found, which are connected with various numbers of nerve cells in particular cross-sections.
A giant congenital pigmented nevus in a horse.
The American Journal of dermatopathology    January 1, 1984   Volume 6 Suppl 325-330 
Calderwood Mays MB, Mayhew IG, Woodard JC.Pigmented nevi have not been widely recognized in domesticated animals. We describe, for the first time, a giant congenital pigmented nevus in a horse. Because of a prominent neuroid component within the lesion, neurofibromatosis was the major differential diagnosis.
Myelopathy and vitamin E deficiency in six Mongolian wild horses.
Journal of the American Veterinary Medical Association    December 1, 1983   Volume 183, Issue 11 1266-1268 
Liu SK, Dolensek EP, Adams CR, Tappe JP.Degenerative myelopathy was diagnosed in six Mongolian wild horses. Three of the horses had a history of ataxia dating from birth to 3 months of age. The clinical signs were uncoordinated movement of the hindlimbs and an abnormally wide-based gait and stance. The other 3 horses had mild ataxia. There were no gross lesions in the brain, vertebrae, or spinal cord. Histologic examination revealed degeneration of the neural processes in the ventral and lateral funiculi of all 6 horses. Myelin sheaths were dilated and vacuolated, and there were swollen, fragmented, or lysed axons. Neuronal degenera...
Osteochondrotic changes in the vertebrae of four ataxic horses suffering from cervical vertebral malformation.
Nordisk veterinaermedicin    December 1, 1983   Volume 35, Issue 12 468-474 
Alitalo I, Kärkkäinen M.Ataxia caused by a focal compression of the cervical spinal cord was diagnosed in four young standardbred trotting horses. Diagnosis was verified by myelography. Changes in the cervical vertebral column were studied using microradiographic and histologic methods. In the vertebrae involved, there was irregularity of the cartilaginous growth zone, cracks with a loose fragment and disturbance in the enchondral ossification. These changes resemble osteochondrosis. Porous appearance in lateral and ventral funiculi as well as mural calcified plaques in the small vessels of the white matter were foun...
Cytology of equine cerebrospinal fluid.
Veterinary pathology    September 1, 1983   Volume 20, Issue 5 553-562 doi: 10.1177/030098588302000507
Beech J.The cytology of cerebrospinal fluid samples from horses is described. The samples were obtained from 24 normal horses, 35 horses with axonal degeneration and/or spinal cord compression, 29 horses with encephalomyelitis, 14 horses with other lesions of the nervous system, and eight horses with signs of neurologic dysfunction of undetermined origin. (Three of the latter were suspected botulinum intoxications.) Fluid was aspirated from the atlanto-occipital space following general anesthesia or immediately after a lethal dose of barbiturate. In two horses, fluid also was aspirated from the lumbos...
Main Drain virus as a cause of equine encephalomyelitis.
Journal of the American Veterinary Medical Association    September 1, 1983   Volume 183, Issue 5 555-558 
Emmons RW, Woodie JD, Laub RL, Oshiro LS.Main Drain virus, which is thought to be transmitted normally among rabbits and various rodents by its natural vector, Culicoides variipennis, was isolated repeatedly from brain tissue of a sick horse from Sacramento County, California, and was implicated as the causative agent. Signs of illness were incoordination and ataxia, stiff neck, head pressing, inability to swallow, fever, and tachycardia.
Ataxia as the only clinical sign of cerebrospinal meningitis in a horse with equine infectious anemia.
Journal of the American Veterinary Medical Association    August 1, 1983   Volume 183, Issue 3 324-325 
Held JP, McGavin MD, Geiser D.No abstract available
Narcolepsy in a horse.
Journal of the American Veterinary Medical Association    July 1, 1983   Volume 183, Issue 1 126-128 
Sweeney CR, Hendricks JC, Beech J, Morrison AR.No abstract available
Cervical intervertebral disc protrusion in two horses.
The Canadian veterinary journal = La revue veterinaire canadienne    June 1, 1983   Volume 24, Issue 6 188-191 
Foss RR, Genetzky RM, Riedesel EA, Graham C.Two horses with ataxia of all four limbs were found to have cervical intervertebral disc protrusion. Severe pelvic limb ataxia, proprioceptive deficits and spasticity were present in both horses with similar but less severe signs in the thoracic limbs. Cerebrospinal fluid analysis was within normal limits. Metrizamide myelography allowed definitive diagnosis in one case when a compression of the spinal cord was demonstrated at the level of the second intervertebral space. In the second case, an intervertebral disc protrusion between cervical vertebrae 6 and 7 was found at necropsy. Fiber degen...
Clinicopathologic features of brain herniation in animals.
Journal of the American Veterinary Medical Association    May 15, 1983   Volume 182, Issue 10 1111-1116 
Kornegay JN, Oliver JE, Gorgacz EJ.No abstract available
Antibody to neuritogenic myelin protein P2 in equine paresis due to equine herpesvirus 1.
Zentralblatt fur Veterinarmedizin. Reihe B. Journal of veterinary medicine. Series B    March 1, 1983   Volume 30, Issue 2 137-140 doi: 10.1111/j.1439-0450.1983.tb01822.x
Klingeborn B, Dinter Z, Hughes RA.No abstract available
[Progressive course of spinal cord tumors].
Zhurnal nevropatologii i psikhiatrii imeni S.S. Korsakova (Moscow, Russia : 1952)    January 1, 1983   Volume 83, Issue 5 641-646 
Okladnikov GI.The main clinical varieties of spinal cord and equine tail tumors are reviewed. Of 221 cases, the progressive course of the disease was recorded in 76,9%, slow-progressive course was observed in 68,1% and rapid-progressive in 8,8% of cases. It is stressed that in the presence of the progressive course of the disease there may occur different manifestations of the tumorous process of the spinal cord, the examination of which makes it possible to improve the diagnosis, particularly in the early stage of the spinal oncological process.
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