Analyze Diet

Topic:Anti-inflammatory

Anti-inflammatory agents are substances used to reduce inflammation in horses, often employed in the management of various equine conditions. These agents can be categorized into non-steroidal anti-inflammatory drugs (NSAIDs) and corticosteroids, among others. NSAIDs, such as phenylbutazone and flunixin meglumine, work by inhibiting cyclooxygenase enzymes, which play a role in the inflammatory process. Corticosteroids, like dexamethasone, modulate the immune response and reduce inflammation through their effects on gene expression. The use of anti-inflammatory agents is common in equine veterinary practice to alleviate pain and improve the quality of life in horses suffering from musculoskeletal disorders, colic, and other inflammatory conditions. This page compiles peer-reviewed research studies and scholarly articles that explore the pharmacology, efficacy, and safety of anti-inflammatory agents in equine medicine.
Review of pathogenesis and treatment of degenerative joint disease.
Equine veterinary journal. Supplement    September 1, 1988   Issue 6 3-11 doi: 10.1111/j.2042-3306.1988.tb04641.x
McIlwraith CW, Vachon A.Treatment of degenerative joint disease (osteoarthritis) is discussed in relationship to pathogenesis of five clinical entities, defined to facilitate discussion:-1) occurring in high motion joints and associated with synovitis; 2) associated with low motion joints; 3) "non-progressive" articular cartilage erosion; 4) secondary to other identified problems (intra-articular fractures, ligamentous damage, wounds, septic arthritis, osteochondrosis); and 5) chondromalacia of the patella. In addition to direct damage to articular cartilage, synovitis and capsulitis and depletion of matrical glycosa...
A-equi-2 influenza in horses in the Republic of South Africa.
Journal of the South African Veterinary Association    June 1, 1988   Volume 59, Issue 2 123-125 
Rogers AL.In early December 1986 A-equi-2 influenza virus was isolated for the first time in the Republic of South Africa. All horses were susceptible to the highly contagious aerosol-borne orthomyxovirus resulting in widespread outbreaks of equine influenza with typical primary respiratory symptoms. Treatment consisted of rest, anti-inflammatory drugs, antibiotics and good nursing. Future protection can be obtained by vaccination.
Near drowning of a gelding.
Journal of the American Veterinary Medical Association    February 1, 1988   Volume 192, Issue 3 377-378 
Humber KA.After becoming entangled in safety lines and being submerged in a chlorinated swimming pool, a 2-year-old Thoroughbred gelding was evaluated and treated successfully. Treatment included antimicrobial agents, anti-inflammatory drugs, bronchodilators, diuretics, humidified oxygen, and surfactant transplant. The results of follow-up examination of the gelding 120 days after hospitalization were normal.
Serum thromboxane in the horse and its inhibition by aspirin, phenylbutazone and flunixin.
The British veterinary journal    September 1, 1987   Volume 143, Issue 5 462-476 doi: 10.1016/0007-1935(87)90024-8
Lees P, Ewins CP, Taylor JB, Sedgwick AD.No abstract available
Actions of non-steroidal anti-inflammatory drugs on equine leucocyte movement in vitro.
Journal of veterinary pharmacology and therapeutics    June 1, 1987   Volume 10, Issue 2 150-159 doi: 10.1111/j.1365-2885.1987.tb00092.x
Dawson J, Lees P, Sedgwick AD.The direct effects of four non-steroidal anti-inflammatory drugs (NSAIDs) on equine polymorphonuclear (PMN) and mononuclear (MN) leucocyte movement were investigated using two in vitro assay systems. The Boyden chamber microfilter technique measures both chemokinetic and chemotactic locomotion, and the agarose microdroplet assay measures solely chemokinesis. Zymosan-activated plasma (ZAP) and the synthetic peptide N-formyl-methionyl-leucyl-phenylalanine (FMLP) were used as standard chemoattractants for PMN and MN leucocytes, respectively. The actions of six concentrations of each NSAID, indome...
Applications of equine models of acute inflammation. The Ciba-Geigy Prize for Research in Animal Health.
The Veterinary record    May 30, 1987   Volume 120, Issue 22 522-529 doi: 10.1136/vr.120.22.522
Lees P, Higgins AJ, Sedgwick AD, May SA.The development of reproducible models of acute inflammation in which inflammatory heat is easily quantified and from which inflammatory exudate is readily harvested has facilitated studies in the horse of the actions of steroids and non-steroidal anti-inflammatory drugs (NSAIDS). Blockade of the synthesis of eicosanoids and suppression of inflammatory heat by clinical dose rates of NSAIDS suggests a causal link between the two events and provides further evidence for a role of these compounds in acute equine inflammation. The tendency for enolic and carboxylic acids NSAIDS to accumulate in in...
Cellular aspects of inflammation. The Ciba-Geigy Prize for Research in Animal Health.
The Veterinary record    May 30, 1987   Volume 120, Issue 22 529-536 doi: 10.1136/vr.120.22.529
Sedgwick AD, Lees P, Dawson J, May SA.The migration of leucocytes to sites of acute and chronic inflammation is an event of central importance to the maintenance of inflammatory processes; extravascular leucocytes are responsible for generating chemical mediators of inflammation and the phagocytosis of particulate matter. They may also be involved in the conversion of acute to chronic inflammatory lesions. Leucocytes are attracted to sites of tissue injury by a range of chemoattractants. This paper describes the development of a method for separating on Percoll gradients purified populations of equine polymorphonuclear and mononuc...
Complement-induced equine neutrophil adhesiveness and aggregation.
Veterinary pathology    May 1, 1987   Volume 24, Issue 3 239-249 doi: 10.1177/030098588702400308
Slauson DO, Skrabalak DS, Neilsen NR, Zwahlen RD.Equine neutrophils (PMN) were isolated from citrated normal blood by density gradient separation on Ficoll-Hypaque to greater than 96% purity and 98% viability and an average of 3.78 x 10(7) PMN/ml. The agonist C5a des Arg was used in serial dilutions of whole zymosan-activated equine plasma (ZAP) or was partially purified from ZAP by column chromatography. Purified equine PMN exhibited rapid aggregation following incubation with C5a des Arg which was further dependent on the availability of divalent cations, especially Mg++. The microfilament disruptive agent cytochalasin B (5 micrograms/50 m...
Low dose flunixin meglumine: effects on eicosanoid production and clinical signs induced by experimental endotoxaemia in horses.
Equine veterinary journal    May 1, 1987   Volume 19, Issue 3 201-206 doi: 10.1111/j.2042-3306.1987.tb01380.x
Semrad SD, Hardee GE, Hardee MM, Moore JN.The efficacy of low doses of flunixin meglumine in reducing eicosanoid generation and clinical signs in response to experimentally induced endotoxaemia was investigated. Thromboxane B2 and 6-keto-prostaglandin F1 alpha were measured in serum and plasma by radioimmunoassay. Plasma flunixin concentrations were determined by high performance liquid chromatography and pharmacokinetic parameters derived non-compartmentally. In horses administered flunixin meglumine before endotoxin challenge, a significant suppression in plasma thromboxane B2 and 6-keto-prostaglandin F1 alpha generation was observe...
Actions of BW540C in an equine model of acute inflammation: a preliminary study.
The veterinary quarterly    April 1, 1987   Volume 9, Issue 2 103-110 doi: 10.1080/01652176.1987.9694086
Higgins AJ, Lees P, Sedgwick AD.An equine model of acute non-immune inflammation has been developed to facilitate studies of the inflammatory process and the actions of novel anti-inflammatory drugs. Five polyester sponge strips soaked in sterile 2% carrageenin solution were placed in subcutaneous pouches prepared under local anaesthesia in the necks of conscious ponies. Serial removal of the strips and harvesting of the exudate enabled studies to be made of the cellular, biochemical and mediator aspects of the localised, acute inflammation, and the heat generated by the lesion was monitored by infra-red thermometry. Maximal...
Chronic nongranulomatous enteritis in seven horses.
Journal of the American Veterinary Medical Association    March 15, 1987   Volume 190, Issue 6 684-686 
Barclay WP, McCracken RJ, Phillips TN, Foerner JJ.Chronic nongranulomatous intestinal inflammation was found during laparotomy in 7 horses. The clinical signs consisted of recurrent abdominal pain in all horses. Anti-inflammatory agents (corticosteroidal and nonsteroidal agents) appeared to be effective for controlling the signs. Surgical removal of the involved intestine also was effective. Nonocclusive or temporary mesenteric ischemia was proposed as a cause of the intestinal lesions.
Actions of betamethasone in models of acute non-immune inflammation.
The British veterinary journal    March 1, 1987   Volume 143, Issue 2 143-158 doi: 10.1016/0007-1935(87)90006-6
Lees P, Higgins AJ, Sedgwick AD, Daniel MJ.No abstract available
Effect of aspirin on ex vivo generation of thromboxane in healthy horses.
American journal of veterinary research    January 1, 1987   Volume 48, Issue 1 13-16 
Baxter GM, Moore JN.Different dosages of aspirin were administered (by nasogastric tube) to 3 groups of 5 healthy adult horses to determine the minimal effective dosage needed to decrease serum thromboxane B2 (TxB2) concentrations and to determine the duration of this decrease. When compared with their base-line serum TxB2 concentrations, horses in group 1 (given 5 mg/kg) had a 71% decrease in TxB2 concentrations at 24 hours after aspirin was given and a 86% decrease at 48 hours; serum TxB2 concentrations were back to base-line values by 120 hours. Horses in group 2 (given 10 mg/kg) had a 60% decrease in TxB2 con...
Use of a novel non-steroidal anti-inflammatory drug in the horse.
Equine veterinary journal    January 1, 1987   Volume 19, Issue 1 60-66 doi: 10.1111/j.2042-3306.1987.tb02584.x
Higgins AJ, Lees P, Sedgwick AD, Buick AR, Churchus R.In a two-part cross-over experiment in six ponies, an acute inflammatory reaction was generated by injecting carrageenin solution into subcutaneously-implanted tissue-cages lined with fibrovascular granulation tissue. In each part of the cross-over, half of the ponies received a novel phenylpyrazoline anti-inflammatory agent (BW540C) orally and half received a placebo treatment. BW540C inhibited platelet cyclo-oxygenase for 24 h but the reductions in exudate eicosanoid concentrations were less pronounced. A significant suppression in the rise of surface skin temperature in BW540C-treated ponie...
Effects of a phenylbutazone paste in ponies: model of acute nonimmune inflammation.
American journal of veterinary research    November 1, 1986   Volume 47, Issue 11 2359-2363 
Lees P, Higgins AJ.In a 12-day treatment schedule, 5 ponies were given orally a paste formulation of phenylbutazone (PBZ) and 5 matched ponies were given equivalent doses of a placebo paste. On day 12, a mild, nonimmune inflammatory reaction was induced subcutaneously in the neck of each pony by inserting sterile, polyester sponge strips soaked in a 2% carrageenan solution. Exudate was collected at 4, 8, 12, and 24 hours by serial removal of sponges. There were no significant (P less than 0.05) differences in exudate protein concentration and leukocyte numbers between the treatment groups, but the group given PB...
Phenylbutazone and oxyphenbutazone distribution into tissue fluids in the horse.
Journal of veterinary pharmacology and therapeutics    June 1, 1986   Volume 9, Issue 2 204-212 doi: 10.1111/j.1365-2885.1986.tb00031.x
Lees P, Taylor JB, Higgins AJ, Sharma SC.The clinically recommended dose rate of phenylbutazone (4.4 mg/kg) was administered intravenously as a single dose to five Welsh Mountain ponies. Distribution of phenylbutazone and its active metabolite oxyphenbutazone into body fluids was studied by measuring concentrations in plasma, tissue-cage fluid, peritoneal fluid and acute inflammatory exudate harvested from a polyester sponge model of inflammation. The ready penetration of phenylbutazone into inflammatory exudate was demonstrated by the relatively high mean value for Cmax of 12.4 micrograms/ml occurring at a time of 4.6 h and a mean A...
Flunixin meglumine: quantitative determination in and effects on composition of equine inflammatory exudate.
The British veterinary journal    March 1, 1986   Volume 142, Issue 2 163-169 doi: 10.1016/0007-1935(86)90093-X
Higgins AJ, Lees P, Taylor JB, Ewins CP.No abstract available
Effects of flunixin meglumine, phenylbutazone and a selective thromboxane synthetase inhibitor (UK-38,485) on thromboxane and prostacyclin production in healthy horses.
Research in veterinary science    March 1, 1986   Volume 40, Issue 2 152-156 
Hardee MM, Moore JN, Hardee GE.The efficacy of three agents which alter the metabolism of arachidonic acid was investigated in normal, conscious horses. A dose response evaluation was made of flunixin meglumine and phenylbutazone, two cyclo-oxygenase inhibitors, and of a selective thromboxane synthetase inhibitor, UK-38,485. Radioimmunoassay of thromboxane B2 (TxB2) and 6-keto prostaglandin F1 alpha (PGF1 alpha) was used to assess the concentrations of thromboxane A2 (TxA2) and prostacyclin (PGI2) respectively, in serum. Flunixin was the most potent inhibitor of serum TxB2 and 6-keto PGF1 alpha production. UK-38,485 also de...
Phenylbutazone in the horse: a review.
Journal of veterinary pharmacology and therapeutics    March 1, 1986   Volume 9, Issue 1 1-25 doi: 10.1111/j.1365-2885.1986.tb00008.x
Tobin T, Chay S, Kamerling S, Woods WE, Weckman TJ, Blake JW, Lees P.Phenylbutazone is an acidic, lipophilic, non-steroidal anti-inflammatory drug (NSAID). It is extensively metabolized in the horse. The metabolites so far identified, oxyphenbutazone, gamma-hydroxyoxyphenbutazone, account for some 25-30% of administered dose over 24 h. The plasma half-life of phenylbutazone and termination of its pharmacological action are determined primarily by its rate of hepatic metabolism. Phenylbutazone acts by inhibiting the cyclooxygenase enzyme system, which is responsible for synthesis of prostanoids such as PGE2. It appears to act on prostaglandin-H synthase and pros...
Renal toxicity of non-steroidal anti-inflammatory drugs.
Toxicologic pathology    January 1, 1986   Volume 14, Issue 1 83-90 doi: 10.1177/019262338601400110
Black HE.Non-steroidal anti-inflammatory drugs represent the most heavily prescribed and used class of drugs in human medicine. Most are derivatives of either salicylates, propionic acid, indoleacetic acid, anthranilic acid, pyrazolone, or oxicams. They depress the synthesis of prostaglandins from arachidonic acid by reversible inhibition of the enzyme cyclooxygenase. In the kidney, prostaglandins PGE2 and PGI2 modulate the vasoconstrictor effects of angiotensin II, norepinephrine, and vasopressin. In the presence of volume contraction, anesthesia, or disease states associated with high levels of these...
Suspected non-steroidal anti-inflammatory drug toxicity in a horse.
The Veterinary record    November 30, 1985   Volume 117, Issue 22 581-582 doi: 10.1136/vr.117.22.581
Hunt JM, Lees P, Edwards GB.No abstract available
[Clinical use of various non-steroidal anti-inflammatory agents in horses and cattle].
Nordisk veterinaermedicin    November 1, 1985   Volume 37, Issue 6 329-337 
Hesselholt M, Friis C.No abstract available
Template bleeding time and thromboxane generation in the horse: effects of three non-steroidal anti-inflammatory drugs.
Equine veterinary journal    July 1, 1985   Volume 17, Issue 4 322-324 doi: 10.1111/j.2042-3306.1985.tb02509.x
Kopp KJ, Moore JN, Byars TD, Brooks P.No abstract available
Failure of superoxide dismutase to alter equine arachidonic acid-induced platelet aggregation, in vitro or ex vivo.
American journal of veterinary research    May 1, 1985   Volume 46, Issue 5 1104-1106 
Clemmons RM, Lee MR, Bliss EL, Asbury AC, Cook D, Brown V.Superoxide dismutase (SOD), a free radical scavenger with anti-inflammatory activity, was administered IM to horses. Ex vivo platelet aggregation in response to arachidonic acid was monitored to determine whether exogenous SOD altered equine platelet prostaglandin metabolism. Preparations of platelet-rich plasma obtained before SOD administration were incubated with different concentrations of SOD and were aggregated with arachidonic acid. Superoxide dismutase did not exert a demonstrable effect, either ex vivo or in vitro. Aspirin abolished arachidonic acid-induced platelet aggregation in vit...
Sacroiliac lesions as a cause of chronic poor performance in competitive horses.
Equine veterinary journal    March 1, 1985   Volume 17, Issue 2 111-118 doi: 10.1111/j.2042-3306.1985.tb02063.x
Jeffcott LB, Dalin G, Ekman S, Olsson SE.The histories and clinical signs in 11 horses with longstanding poor performance attributed to chronic sacroiliac damage are described. The main clinical feature was a lack of impulsion from one or both hindlimbs causing a restriction in the hind gait or lowgrade lameness. A temporary improvement was often achieved using anti-inflammatory medication, but eventually all the horses were killed because of unsatisfactory progress at exercise. Post mortem examination revealed that changes were confined to the sacroiliac joints. The macroscopic and histological findings varied considerably and in on...
Clinical pharmacology and therapeutic uses of non-steroidal anti-inflammatory drugs in the horse.
Equine veterinary journal    March 1, 1985   Volume 17, Issue 2 83-96 doi: 10.1111/j.2042-3306.1985.tb02056.x
Lees P, Higgins AJ.Weak organic acids possessing anti-inflammatory, analgesic and antipyretic properties--commonly known as aspirin-like drugs--have been used in equine medicine for almost 100 years. These non-steroidal anti-inflammatory drugs (NSAIDs) may be classified chemically into two groups; the enolic acids such as phenylbutazone and carboxylic acids like flunixin, meclofenamate and naproxen. All NSAIDs have similar and possibly identical modes of action accounting for both their therapeutic and their toxic effects. They block some part of the cyclo-oxygenase enzyme pathway and thereby suppress the synthe...
Effects of repeated endotoxin injections on prostanoids, hemodynamics, endothelial cells, and survival in ponies.
Circulatory shock    January 1, 1985   Volume 16, Issue 3 253-264 
Templeton CB, Bottoms GD, Fessler JF, Turek JJ, Boon GD.The objectives of this study were to determine the pathophysiological effects of increasing amounts of endotoxin administered intraperitoneally (IP) for 24 hr at which time an intravenous (IV) injection of endotoxin was given. The ability of flunixin meglumine (FM), a nonsteroidal antiinflammatory drug with antiprostaglandin activity, to provide protective effects was also determined. Eight ponies were divided into two groups of four ponies each; one group (untreated) received endotoxin only and the other group (treated) received endotoxin while being treated with flunixin. Hemodynamic and ser...
Influence of betamethasone on the composition of inflammatory exudate in the horse: a preliminary report.
Equine veterinary journal    November 1, 1984   Volume 16, Issue 6 539-541 doi: 10.1111/j.2042-3306.1984.tb02013.x
Lees P, Higgins AJ.No abstract available
Medical management of esophageal stricture in seven horses.
Journal of the American Veterinary Medical Association    October 1, 1984   Volume 185, Issue 7 784-787 
Todhunter RJ, Stick JA, Trotter GW, Boles C.Esophageal strictures developed in 7 horses that were treated for esophageal obstruction. A soft diet was fed to all horses, with intermittent nasogastric tube feeding in one, and medication included nonsteroidal anti-inflammatory drugs. Maximal reduction in esophageal lumen diameter was evident by 30 days following circumferential esophageal ulceration, after which lumen diameter increased rapidly. Five horses were clinically normal by 60 days after the esophageal injury. Two horses were euthanatized at the owner's request, 16 and 17 days after the original insult.
Successful treatment of pericarditis in a horse.
Journal of the American Veterinary Medical Association    July 1, 1984   Volume 185, Issue 1 94-98 
Reef VB, Gentile DG, Freeman DE.No abstract available