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Topic:Lameness

Lameness in horses is a condition characterized by an abnormal gait or stance due to pain or mechanical dysfunction affecting the limbs or spine. It is a common issue in equine practice and can result from a variety of causes, including injury, infection, or degenerative diseases. Lameness can affect a horse's performance and quality of life, making accurate diagnosis and management important for equine health. Diagnostic techniques often include physical examination, imaging, and gait analysis. This page aggregates peer-reviewed research studies and scholarly articles that explore the etiology, diagnostic methods, and management strategies for lameness in horses.
Lag screw fixation of avulsion fractures of the second phalanx in the horse.
Journal of the American Veterinary Medical Association    August 15, 1975   Volume 167, Issue 4 306-309 
Turner AS, Gabel AA.Avulsion fractures of the posteromedial eminence of the 2nd phalanx were repaired, using an ASIF cortex screw in 3 reining Quarter Horses. Within 9 and 11 months, respectively, 2 horses were sound and back in competition as reining horses. The other horse was sound for pleasure class showing at 7 months after surgery.
Equine laminitis of alimentary origin: an experimental model.
American journal of veterinary research    April 1, 1975   Volume 36, Issue 4 Pt.1 441-444 
Garner HE, Coffman JR, Hahn AW, Hutcheson DP, Tumbleson ME.Acute alimentary form of laminitis was uniformly induced in 11 of 12 horses by administration of a starch and wood flour gruel and could be graded by previously established (Obel) and presently defined criteria. The experimentally induced laminitis was similar to naturally occurring laminitis, as determined on the basis of lameness severity and vital signs. Packed cell volume, leukocyte count, and total protein were significantly increased (P smaller than 0.05) at 24 and 40 hours after administration of gruel. Arterial systolic and diastolic pressures increased, central venous pressure decreas...
Use of the hoof tester in diagnosing lameness in horses.
Veterinary medicine, small animal clinician : VM, SAC    February 1, 1975   Volume 70, Issue 2 205-212 
Szabuniewicz M, Szabuniewicz JM.No abstract available
Hypercalcemia and calcinosis in Florida horses: implication of the shrub, Cestrum diurnum, as the causative agent.
The Cornell veterinarian    January 11, 1975   Volume 65, Issue 1 26-56 
Krook L, Wasserman RH, Shively JN, Tashjian AH, Brokken TD, Morton JF.A chronic debilitating disease is described in Florida horses. There is progress weight loss and lameness of increasing severity. Plasma calcium is elevated to moderate or severe degree. Anatomical changes include dystrophic calcinosis of elastic tissues, viz. major arteries, tendons and ligaments. A generalized osteopetrosis is present and may be related to hypoparathyroidsim and hypercalcitoninism. The presence of Cestrum diurnum (day-blooming jessamine, day cestrum, wild jasmin) in areas accessible to affected animals, the observation that leaves of the plant were stripped in these areas, a...
Sheared heels: diagnosis and treatment.
Journal of the American Veterinary Medical Association    January 1, 1975   Volume 166, Issue 1 53-55 
Moyer W, Anderson JP.No abstract available
Lamenesses caused by improper shoeing.
Journal of the American Veterinary Medical Association    January 1, 1975   Volume 166, Issue 1 47-52 
Moyer W, Anderson JP.No abstract available
Equine laminitis and associated hypertension: a review.
Journal of the American Veterinary Medical Association    January 1, 1975   Volume 166, Issue 1 56-57 
Garner HE, Coffman JR, Hahn AW, Ackerman N, Johnson JH.No abstract available
Castration and other factors affecting the risk of equine laminitis.
The Cornell veterinarian    January 1, 1975   Volume 65, Issue 1 57-64 
Dorn CR, Garner HE, Coffman JR, Hahn AW, Tritschler LG.The characteristics of a series of equine laminitis cases were compared with that of other Equidae examined at the University of Missouri Veterinary Hospital and Clinic during May, 1965 through December, 1971. The model age for ponies with laminitis was 7-9 years and for all other cases the model age was 4-6 years. Also the predominant age for mares was 4-6 years and for males was 7-9 years. After controlling for age and breed differences, there were significantly fewer castrated males among the affected males than expected (P small than 0.02), indicating that hormonal factors may play a role ...
Laminitis therapy.
Journal of the American Veterinary Medical Association    November 1, 1974   Volume 165, Issue 9 776 
Horvath AA.No abstract available
Manipulative procedures in detecting horse lameness.
The Cornell veterinarian    July 1, 1974   Volume 64, Issue 3 443-447 
Delahanty DD.No abstract available
Subchondral cysts of the navicular bone as a cause of equine lameness.
Veterinary medicine, small animal clinician : VM, SAC    July 1, 1974   Volume 69, Issue 7 873 
Merriam JG, Johnson JH.No abstract available
[Prognosis and treatment of podotrochlosis in the horse].
Tierarztliche Praxis    January 1, 1974   Volume 2, Issue 3 281-285 
Wintzer HJ.No abstract available
Springing sole syndrome.
Modern veterinary practice    January 1, 1974   Volume 55, Issue 1 48-51 
Milne FJ, Dudgeon MH, Valli VE.No abstract available
[Prognosis, therapy and pathogenesis of sesamoid-bone lameness].
Tierarztliche Praxis    January 1, 1974   Volume 2, Issue 3 287-298 
Németh F.No abstract available
[Sesamoiditis in the horse].
Tierarztliche Praxis    January 1, 1974   Volume 2, Issue 2 199-206 
Németh F.No abstract available
The navicular syndrome.
Modern veterinary practice    November 1, 1973   Volume 54, Issue 12 69 
Johnson JH.No abstract available
Sesamoiditis in the horse.
Tijdschrift voor diergeneeskunde    October 15, 1973   Volume 98, Issue 20 988-994 
Németh F.No abstract available
Relationship of racetrack surface to lameness in the thoroughbred racehorse.
American journal of veterinary research    October 1, 1973   Volume 34, Issue 10 1285-1289 
Cheney JA, Shen CK, Wheat JD.No abstract available
Obscure lameness in a horse.
Modern veterinary practice    August 1, 1973   Volume 54, Issue 8 51-52 
Cerniga RM, Elliott FA.No abstract available
The primary site of damage in bowed tendon of the horse.
Modern veterinary practice    May 1, 1973   Volume 54, Issue 5 58-59 
Rooney JR.No abstract available
Refractory laminitis.
The Veterinary clinics of North America    May 1, 1973   Volume 3, Issue 2 291-300 doi: 10.1016/s0091-0279(73)50038-8
Coffman JR.No abstract available
Post-anaesthetic forelimb lameness in horses.
Equine veterinary journal    April 1, 1973   Volume 5, Issue 2 71-76 doi: 10.1111/j.2042-3306.1973.tb03197.x
Trim CM, Mason J.No abstract available
Lameness and pathologic changes in the distal joints and the phalanges of the standardbred horse. A correlative study.
Acta veterinaria Scandinavica. Supplementum    January 1, 1973   Volume 44 83-96 
Nilsson G.No abstract available
Some procedures and tools in the diagnostics of distal equine lameness.
Acta veterinaria Scandinavica. Supplementum    January 1, 1973   Volume 44 63-79 
Nilsson G, Fredricson I, Drevemo S.No abstract available
Navicular disease: podotrochlitis chronica aseptica podotrochlosis.
Equine veterinary journal    January 1, 1973   Volume 5, Issue 1 1-7 doi: 10.1111/j.2042-3306.1973.tb03185.x
Numans SR, van der Watering CC.No abstract available
Acute laminitis.
Journal of the American Veterinary Medical Association    December 1, 1972   Volume 161, Issue 11 1280-1283 
Coffman JR, Garner HE.No abstract available
Chronic tendosynovitis of the extensor carpi radialis tendon in the horse.
Australian veterinary journal    November 1, 1972   Volume 48, Issue 11 585-587 doi: 10.1111/j.1751-0813.1972.tb05073.x
Wallace CE.No abstract available
Lead and zinc poisoning and the interaction between Pb and Zn poisoning in the foal.
Canadian journal of comparative medicine : Revue canadienne de medecine comparee    October 1, 1972   Volume 36, Issue 4 348-359 
Willoughby RA, MacDonald E, McSherry BJ, Brown G.Groups of young growing horses were fed toxic amounts of lead only, zinc only and the same amounts of lead and zinc together. Those fed Pb only developed pharyngeal and laryngeal paralysis ("roaring") whereas those fed Zn only and Pb and Zn together developed the same clinical syndrome which included swelling at the epiphyseal region of the long bones, stiffness and lameness. Anemia and decreased weight gains were most pronounced in animals fed Zn for the longest periods. Animals fed Pb only did not become anemic and weight loss did not occur until after there was an interference in swallowing...
Melanosarcoma as a cause of lameness in a horse (a case report).
Veterinary medicine, small animal clinician : VM, SAC    September 1, 1972   Volume 67, Issue 9 995-998 
Grant B, Lincoln S.No abstract available
The surgical treatment of bone spavin. Wamberg’s spavin operation and tenectomy of the medial branch of the tendon of the anterior tibial muscle–a comparison of the results of surgical treatment in trotting horses.
Nordisk veterinaermedicin    July 1, 1972   Volume 24, Issue 7 388-392 
Grande EM.No abstract available